BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

114 related articles for article (PubMed ID: 23891849)

  • 21. Enhanced histone H3 acetylation of the PD-L1 promoter via the COP1/c-Jun/HDAC3 axis is required for PD-L1 expression in drug-resistant cancer cells.
    Wang H; Fu C; Du J; Wang H; He R; Yin X; Li H; Li X; Wang H; Li K; Zheng L; Liu Z; Qiu Y
    J Exp Clin Cancer Res; 2020 Feb; 39(1):29. PubMed ID: 32024543
    [TBL] [Abstract][Full Text] [Related]  

  • 22. NF-kappaB and activator protein 1 response elements and the role of histone modifications in IL-1beta-induced TGF-beta1 gene transcription.
    Lee KY; Ito K; Hayashi R; Jazrawi EP; Barnes PJ; Adcock IM
    J Immunol; 2006 Jan; 176(1):603-15. PubMed ID: 16365456
    [TBL] [Abstract][Full Text] [Related]  

  • 23. COP9 signalosome-directed c-Jun activation/stabilization is independent of JNK.
    Naumann M; Bech-Otschir D; Huang X; Ferrell K; Dubiel W
    J Biol Chem; 1999 Dec; 274(50):35297-300. PubMed ID: 10585392
    [TBL] [Abstract][Full Text] [Related]  

  • 24. Heterodimerization with Fra-1 cooperates with the ERK pathway to stabilize c-Jun in response to the RAS oncoprotein.
    Talotta F; Mega T; Bossis G; Casalino L; Basbous J; Jariel-Encontre I; Piechaczyk M; Verde P
    Oncogene; 2010 Aug; 29(33):4732-40. PubMed ID: 20543861
    [TBL] [Abstract][Full Text] [Related]  

  • 25. Adenovirus type 12 early region 1A expresses a 52R protein repressing the trans-activating activity of transcription factor c-Jun/AP-1.
    Brockmann D; Feng L; Kröner G; Tries B; Esche H
    Virology; 1994 Feb; 198(2):717-23. PubMed ID: 8291251
    [TBL] [Abstract][Full Text] [Related]  

  • 26. Toll-like receptor-3 mediates HIV-1 transactivation via NFκB and JNK pathways and histone acetylation, but prolonged activation suppresses Tat and HIV-1 replication.
    Bhargavan B; Woollard SM; Kanmogne GD
    Cell Signal; 2016 Feb; 28(2):7-22. PubMed ID: 26569339
    [TBL] [Abstract][Full Text] [Related]  

  • 27. Disruption of the c-JUN-JNK complex by a cell-permeable peptide containing the c-JUN delta domain induces apoptosis and affects a distinct set of interleukin-1-induced inflammatory genes.
    Holzberg D; Knight CG; Dittrich-Breiholz O; Schneider H; Dörrie A; Hoffmann E; Resch K; Kracht M
    J Biol Chem; 2003 Oct; 278(41):40213-23. PubMed ID: 12832416
    [TBL] [Abstract][Full Text] [Related]  

  • 28. The tyrosine kinase c-Abl protects c-Jun from ubiquitination-mediated degradation in T cells.
    Gao B; Lee SM; Fang D
    J Biol Chem; 2006 Oct; 281(40):29711-8. PubMed ID: 16901904
    [TBL] [Abstract][Full Text] [Related]  

  • 29. Roles of HIPK1 and HIPK2 in AML1- and p300-dependent transcription, hematopoiesis and blood vessel formation.
    Aikawa Y; Nguyen LA; Isono K; Takakura N; Tagata Y; Schmitz ML; Koseki H; Kitabayashi I
    EMBO J; 2006 Sep; 25(17):3955-65. PubMed ID: 16917507
    [TBL] [Abstract][Full Text] [Related]  

  • 30. Drosophila activating transcription factor-2 is involved in stress response via activation by p38, but not c-Jun NH(2)-terminal kinase.
    Sano Y; Akimaru H; Okamura T; Nagao T; Okada M; Ishii S
    Mol Biol Cell; 2005 Jun; 16(6):2934-46. PubMed ID: 15788564
    [TBL] [Abstract][Full Text] [Related]  

  • 31. c-Jun phosphorylation in signal transduction and gene regulation.
    Papavassiliou AG
    Anticancer Res; 1993; 13(6A):2213-20. PubMed ID: 8297136
    [TBL] [Abstract][Full Text] [Related]  

  • 32. Identification of amino acid residues in the ETS transcription factor Erg that mediate Erg-Jun/Fos-DNA ternary complex formation.
    Verger A; Buisine E; Carrère S; Wintjens R; Flourens A; Coll J; Stéhelin D; Duterque-Coquillaud M
    J Biol Chem; 2001 May; 276(20):17181-9. PubMed ID: 11278640
    [TBL] [Abstract][Full Text] [Related]  

  • 33. JNK phosphorylation relieves HDAC3-dependent suppression of the transcriptional activity of c-Jun.
    Weiss C; Schneider S; Wagner EF; Zhang X; Seto E; Bohmann D
    EMBO J; 2003 Jul; 22(14):3686-95. PubMed ID: 12853483
    [TBL] [Abstract][Full Text] [Related]  

  • 34. Phosphorylation of c-jun mediated by MAP kinases.
    Pulverer BJ; Kyriakis JM; Avruch J; Nikolakaki E; Woodgett JR
    Nature; 1991 Oct; 353(6345):670-4. PubMed ID: 1922387
    [TBL] [Abstract][Full Text] [Related]  

  • 35. Serum-independent phosphorylation of c-Jun and alterations in AP-1 components by transformation with various oncogenes.
    Kamada S; Toyoshima K; Akiyama T
    J Biol Chem; 1994 Feb; 269(6):4565-70. PubMed ID: 8308027
    [TBL] [Abstract][Full Text] [Related]  

  • 36. The HDAC1/c-JUN complex is essential in the promotion of nerve injury-induced neuropathic pain through JNK signaling.
    Sanna MD; Galeotti N
    Eur J Pharmacol; 2018 Apr; 825():99-106. PubMed ID: 29477655
    [TBL] [Abstract][Full Text] [Related]  

  • 37. Long-range electrostatic interactions influence the orientation of Fos-Jun binding at AP-1 sites.
    Ramirez-Carrozzi VR; Kerppola TK
    J Mol Biol; 2001 Jan; 305(3):411-27. PubMed ID: 11152600
    [TBL] [Abstract][Full Text] [Related]  

  • 38. Tyrosine 170 is dispensable for c-Jun turnover.
    Xie M; Sabapathy K
    Cell Signal; 2010 Feb; 22(2):330-7. PubMed ID: 19818398
    [TBL] [Abstract][Full Text] [Related]  

  • 39. Ret oncogene signal transduction via a IRS-2/PI 3-kinase/PKB and a SHC/Grb-2 dependent pathway: possible implication for transforming activity in NIH3T3 cells.
    Hennige AM; Lammers R; Arlt D; Höppner W; Strack V; Niederfellner G; Seif FJ; Häring HU; Kellerer M
    Mol Cell Endocrinol; 2000 Sep; 167(1-2):69-76. PubMed ID: 11000521
    [TBL] [Abstract][Full Text] [Related]  

  • 40. p75-Ras-GRF1 is a c-Jun/AP-1 target protein: its up regulation results in increased Ras activity and is necessary for c-Jun-induced nonadherent growth of Rat1a cells.
    Leaner VD; Donninger H; Ellis CA; Clark GJ; Birrer MJ
    Mol Cell Biol; 2005 Apr; 25(8):3324-37. PubMed ID: 15798216
    [TBL] [Abstract][Full Text] [Related]  

    [Previous]   [Next]    [New Search]
    of 6.