985 related articles for article (PubMed ID: 23970536)
1. Muscle immobilization and remobilization downregulates PGC-1α signaling and the mitochondrial biogenesis pathway.
Kang C; Ji LL
J Appl Physiol (1985); 2013 Dec; 115(11):1618-25. PubMed ID: 23970536
[TBL] [Abstract][Full Text] [Related]
2. PGC-1α overexpression via local transfection attenuates mitophagy pathway in muscle disuse atrophy.
Kang C; Ji LL
Free Radic Biol Med; 2016 Apr; 93():32-40. PubMed ID: 26746585
[TBL] [Abstract][Full Text] [Related]
3. PGC-1α overexpression by in vivo transfection attenuates mitochondrial deterioration of skeletal muscle caused by immobilization.
Kang C; Goodman CA; Hornberger TA; Ji LL
FASEB J; 2015 Oct; 29(10):4092-106. PubMed ID: 26178167
[TBL] [Abstract][Full Text] [Related]
4. Muscle immobilization activates mitophagy and disrupts mitochondrial dynamics in mice.
Kang C; Yeo D; Ji LL
Acta Physiol (Oxf); 2016 Nov; 218(3):188-197. PubMed ID: 27083499
[TBL] [Abstract][Full Text] [Related]
5. Exercise activation of muscle peroxisome proliferator-activated receptor-gamma coactivator-1alpha signaling is redox sensitive.
Kang C; O'Moore KM; Dickman JR; Ji LL
Free Radic Biol Med; 2009 Nov; 47(10):1394-400. PubMed ID: 19686839
[TBL] [Abstract][Full Text] [Related]
6. A novel hindlimb immobilization procedure for studying skeletal muscle atrophy and recovery in mouse.
Caron AZ; Drouin G; Desrosiers J; Trensz F; Grenier G
J Appl Physiol (1985); 2009 Jun; 106(6):2049-59. PubMed ID: 19342435
[TBL] [Abstract][Full Text] [Related]
7. Activation of alternative NF-κB signaling during recovery of disuse-induced loss of muscle oxidative phenotype.
Remels AH; Pansters NA; Gosker HR; Schols AM; Langen RC
Am J Physiol Endocrinol Metab; 2014 Mar; 306(6):E615-26. PubMed ID: 24425759
[TBL] [Abstract][Full Text] [Related]
8. PGC1-α over-expression prevents metabolic alterations and soleus muscle atrophy in hindlimb unloaded mice.
Cannavino J; Brocca L; Sandri M; Bottinelli R; Pellegrino MA
J Physiol; 2014 Oct; 592(20):4575-89. PubMed ID: 25128574
[TBL] [Abstract][Full Text] [Related]
9. Exercise training attenuates aging-associated mitochondrial dysfunction in rat skeletal muscle: role of PGC-1α.
Kang C; Chung E; Diffee G; Ji LL
Exp Gerontol; 2013 Nov; 48(11):1343-50. PubMed ID: 23994518
[TBL] [Abstract][Full Text] [Related]
10. PGC-1α buffers ROS-mediated removal of mitochondria during myogenesis.
Baldelli S; Aquilano K; Ciriolo MR
Cell Death Dis; 2014 Nov; 5(11):e1515. PubMed ID: 25375380
[TBL] [Abstract][Full Text] [Related]
11. Immobilization-induced activation of key proteolytic systems in skeletal muscles is prevented by a mitochondria-targeted antioxidant.
Talbert EE; Smuder AJ; Min K; Kwon OS; Szeto HH; Powers SK
J Appl Physiol (1985); 2013 Aug; 115(4):529-38. PubMed ID: 23766499
[TBL] [Abstract][Full Text] [Related]
12. The role of PGC-1alpha on mitochondrial function and apoptotic susceptibility in muscle.
Adhihetty PJ; Uguccioni G; Leick L; Hidalgo J; Pilegaard H; Hood DA
Am J Physiol Cell Physiol; 2009 Jul; 297(1):C217-25. PubMed ID: 19439529
[TBL] [Abstract][Full Text] [Related]
13. Training-induced mitochondrial adaptation: role of peroxisome proliferator-activated receptor γ coactivator-1α, nuclear factor-κB and β-blockade.
Feng H; Kang C; Dickman JR; Koenig R; Awoyinka I; Zhang Y; Ji LL
Exp Physiol; 2013 Mar; 98(3):784-95. PubMed ID: 23104933
[TBL] [Abstract][Full Text] [Related]
14. Intensified mitophagy in skeletal muscle with aging is downregulated by PGC-1alpha overexpression in vivo.
Yeo D; Kang C; Gomez-Cabrera MC; Vina J; Ji LL
Free Radic Biol Med; 2019 Jan; 130():361-368. PubMed ID: 30395971
[TBL] [Abstract][Full Text] [Related]
15. TNF-alpha impairs regulation of muscle oxidative phenotype: implications for cachexia?
Remels AH; Gosker HR; Schrauwen P; Hommelberg PP; Sliwinski P; Polkey M; Galdiz J; Wouters EF; Langen RC; Schols AM
FASEB J; 2010 Dec; 24(12):5052-62. PubMed ID: 20807714
[TBL] [Abstract][Full Text] [Related]
16. Overexpression of human selenoprotein H in neuronal cells enhances mitochondrial biogenesis and function through activation of protein kinase A, protein kinase B, and cyclic adenosine monophosphate response element-binding protein pathway.
Mehta SL; Mendelev N; Kumari S; Andy Li P
Int J Biochem Cell Biol; 2013 Mar; 45(3):604-11. PubMed ID: 23220172
[TBL] [Abstract][Full Text] [Related]
17. Classical NF-κB activation impairs skeletal muscle oxidative phenotype by reducing IKK-α expression.
Remels AH; Gosker HR; Langen RC; Polkey M; Sliwinski P; Galdiz J; van den Borst B; Pansters NA; Schols AM
Biochim Biophys Acta; 2014 Feb; 1842(2):175-85. PubMed ID: 24215713
[TBL] [Abstract][Full Text] [Related]
18. PGC-1α over-expression suppresses the skeletal muscle atrophy and myofiber-type composition during hindlimb unloading.
Wang J; Wang F; Zhang P; Liu H; He J; Zhang C; Fan M; Chen X
Biosci Biotechnol Biochem; 2017 Mar; 81(3):500-513. PubMed ID: 27869526
[TBL] [Abstract][Full Text] [Related]
19. Mechanisms involved in 3',5'-cyclic adenosine monophosphate-mediated inhibition of the ubiquitin-proteasome system in skeletal muscle.
Gonçalves DA; Lira EC; Baviera AM; Cao P; Zanon NM; Arany Z; Bedard N; Tanksale P; Wing SS; Lecker SH; Kettelhut IC; Navegantes LC
Endocrinology; 2009 Dec; 150(12):5395-404. PubMed ID: 19837877
[TBL] [Abstract][Full Text] [Related]
20. Propofol induces impairment of mitochondrial biogenesis through inhibiting the expression of peroxisome proliferator-activated receptor-γ coactivator-1α.
Qin J; Li Y; Wang K
J Cell Biochem; 2019 Oct; 120(10):18288-18297. PubMed ID: 31190345
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
