231 related articles for article (PubMed ID: 24205359)
1. Inhibition of cdk9 during herpes simplex virus 1 infection impedes viral transcription.
Ou M; Sandri-Goldin RM
PLoS One; 2013; 8(10):e79007. PubMed ID: 24205359
[TBL] [Abstract][Full Text] [Related]
2. ICP27 interacts with the C-terminal domain of RNA polymerase II and facilitates its recruitment to herpes simplex virus 1 transcription sites, where it undergoes proteasomal degradation during infection.
Dai-Ju JQ; Li L; Johnson LA; Sandri-Goldin RM
J Virol; 2006 Apr; 80(7):3567-81. PubMed ID: 16537625
[TBL] [Abstract][Full Text] [Related]
3. Role of cdk9 in the optimization of expression of the genes regulated by ICP22 of herpes simplex virus 1.
Durand LO; Roizman B
J Virol; 2008 Nov; 82(21):10591-9. PubMed ID: 18753202
[TBL] [Abstract][Full Text] [Related]
4. Tat modifies the activity of CDK9 to phosphorylate serine 5 of the RNA polymerase II carboxyl-terminal domain during human immunodeficiency virus type 1 transcription.
Zhou M; Halanski MA; Radonovich MF; Kashanchi F; Peng J; Price DH; Brady JN
Mol Cell Biol; 2000 Jul; 20(14):5077-86. PubMed ID: 10866664
[TBL] [Abstract][Full Text] [Related]
5. Herpes simplex virus type 1 infection leads to loss of serine-2 phosphorylation on the carboxyl-terminal domain of RNA polymerase II.
Fraser KA; Rice SA
J Virol; 2005 Sep; 79(17):11323-34. PubMed ID: 16103184
[TBL] [Abstract][Full Text] [Related]
6. Phosphorylation of the RNA polymerase II carboxyl-terminal domain by CDK9 is directly responsible for human immunodeficiency virus type 1 Tat-activated transcriptional elongation.
Kim YK; Bourgeois CF; Isel C; Churcher MJ; Karn J
Mol Cell Biol; 2002 Jul; 22(13):4622-37. PubMed ID: 12052871
[TBL] [Abstract][Full Text] [Related]
7. The carboxyl-terminal domain of RNA polymerase II is phosphorylated by a complex containing cdk9 and infected-cell protein 22 of herpes simplex virus 1.
Durand LO; Advani SJ; Poon AP; Roizman B
J Virol; 2005 Jun; 79(11):6757-62. PubMed ID: 15890914
[TBL] [Abstract][Full Text] [Related]
8. Herpes simplex virus immediate-early protein ICP22 triggers loss of serine 2-phosphorylated RNA polymerase II.
Fraser KA; Rice SA
J Virol; 2007 May; 81(10):5091-101. PubMed ID: 17344289
[TBL] [Abstract][Full Text] [Related]
9. Hsc70 focus formation at the periphery of HSV-1 transcription sites requires ICP27.
Li L; Johnson LA; Dai-Ju JQ; Sandri-Goldin RM
PLoS One; 2008 Jan; 3(1):e1491. PubMed ID: 18231578
[TBL] [Abstract][Full Text] [Related]
10. Herpes Simplex Virus 1 (HSV-1) ICP22 protein directly interacts with cyclin-dependent kinase (CDK)9 to inhibit RNA polymerase II transcription elongation.
Zaborowska J; Baumli S; Laitem C; O'Reilly D; Thomas PH; O'Hare P; Murphy S
PLoS One; 2014; 9(9):e107654. PubMed ID: 25233083
[TBL] [Abstract][Full Text] [Related]
11. T-loop phosphorylated Cdk9 localizes to nuclear speckle domains which may serve as sites of active P-TEFb function and exchange between the Brd4 and 7SK/HEXIM1 regulatory complexes.
Dow EC; Liu H; Rice AP
J Cell Physiol; 2010 Jul; 224(1):84-93. PubMed ID: 20201073
[TBL] [Abstract][Full Text] [Related]
12. CDK9 and SPT5 proteins are specifically required for expression of herpes simplex virus 1 replication-dependent late genes.
Zhao Z; Tang KW; Muylaert I; Samuelsson T; Elias P
J Biol Chem; 2017 Sep; 292(37):15489-15500. PubMed ID: 28743741
[TBL] [Abstract][Full Text] [Related]
13. RNA polymerase II holoenzyme modifications accompany transcription reprogramming in herpes simplex virus type 1-infected cells.
Jenkins HL; Spencer CA
J Virol; 2001 Oct; 75(20):9872-84. PubMed ID: 11559820
[TBL] [Abstract][Full Text] [Related]
14. Herpes simplex virus immediate-early protein ICP22 is required for viral modification of host RNA polymerase II and establishment of the normal viral transcription program.
Rice SA; Long MC; Lam V; Schaffer PA; Spencer CA
J Virol; 1995 Sep; 69(9):5550-9. PubMed ID: 7637000
[TBL] [Abstract][Full Text] [Related]
15. Inhibition of HIV-1 replication by P-TEFb inhibitors DRB, seliciclib and flavopiridol correlates with release of free P-TEFb from the large, inactive form of the complex.
Biglione S; Byers SA; Price JP; Nguyen VT; Bensaude O; Price DH; Maury W
Retrovirology; 2007 Jul; 4():47. PubMed ID: 17625008
[TBL] [Abstract][Full Text] [Related]
16. CDK9 inhibition strategy defines distinct sets of target genes.
Garriga J; Graña X
BMC Res Notes; 2014 May; 7():301. PubMed ID: 24886624
[TBL] [Abstract][Full Text] [Related]
17. Human cytomegalovirus infection induces specific hyperphosphorylation of the carboxyl-terminal domain of the large subunit of RNA polymerase II that is associated with changes in the abundance, activity, and localization of cdk9 and cdk7.
Tamrakar S; Kapasi AJ; Spector DH
J Virol; 2005 Dec; 79(24):15477-93. PubMed ID: 16306619
[TBL] [Abstract][Full Text] [Related]
18. Bromodomain protein Brd4 regulates human immunodeficiency virus transcription through phosphorylation of CDK9 at threonine 29.
Zhou M; Huang K; Jung KJ; Cho WK; Klase Z; Kashanchi F; Pise-Masison CA; Brady JN
J Virol; 2009 Jan; 83(2):1036-44. PubMed ID: 18971272
[TBL] [Abstract][Full Text] [Related]
19. CDK9 regulates AR promoter selectivity and cell growth through serine 81 phosphorylation.
Gordon V; Bhadel S; Wunderlich W; Zhang J; Ficarro SB; Mollah SA; Shabanowitz J; Hunt DF; Xenarios I; Hahn WC; Conaway M; Carey MF; Gioeli D
Mol Endocrinol; 2010 Dec; 24(12):2267-80. PubMed ID: 20980437
[TBL] [Abstract][Full Text] [Related]
20. ICP22 and the UL13 protein kinase are both required for herpes simplex virus-induced modification of the large subunit of RNA polymerase II.
Long MC; Leong V; Schaffer PA; Spencer CA; Rice SA
J Virol; 1999 Jul; 73(7):5593-604. PubMed ID: 10364308
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
