BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

843 related articles for article (PubMed ID: 24359187)

  • 1. Hypoxia causes autophagic stress and derangement of metabolic adaptation in a cell model of amyotrophic lateral sclerosis.
    Cimini S; Rizzardini M; Biella G; Cantoni L
    J Neurochem; 2014 May; 129(3):413-25. PubMed ID: 24359187
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Mitochondrial dysfunction in a cell culture model of familial amyotrophic lateral sclerosis.
    Menzies FM; Cookson MR; Taylor RW; Turnbull DM; Chrzanowska-Lightowlers ZM; Dong L; Figlewicz DA; Shaw PJ
    Brain; 2002 Jul; 125(Pt 7):1522-33. PubMed ID: 12077002
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Transcriptomic indices of fast and slow disease progression in two mouse models of amyotrophic lateral sclerosis.
    Nardo G; Iennaco R; Fusi N; Heath PR; Marino M; Trolese MC; Ferraiuolo L; Lawrence N; Shaw PJ; Bendotti C
    Brain; 2013 Nov; 136(Pt 11):3305-32. PubMed ID: 24065725
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Cu/Zn superoxide dismutase (SOD1) mutations associated with familial amyotrophic lateral sclerosis (ALS) affect cellular free radical release in the presence of oxidative stress.
    Cookson MR; Menzies FM; Manning P; Eggett CJ; Figlewicz DA; McNeil CJ; Shaw PJ
    Amyotroph Lateral Scler Other Motor Neuron Disord; 2002 Jun; 3(2):75-85. PubMed ID: 12215229
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Autophagy activation and neuroprotection by progesterone in the G93A-SOD1 transgenic mouse model of amyotrophic lateral sclerosis.
    Kim J; Kim TY; Cho KS; Kim HN; Koh JY
    Neurobiol Dis; 2013 Nov; 59():80-5. PubMed ID: 23891729
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Zinc pre-treatment enhances NMDAR-mediated excitotoxicity in cultured cortical neurons from SOD1(G93A) mouse, a model of amyotrophic lateral sclerosis.
    Nutini M; Frazzini V; Marini C; Spalloni A; Sensi SL; Longone P
    Neuropharmacology; 2011 Jun; 60(7-8):1200-8. PubMed ID: 21056589
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Neurodegeneration induced by complex I inhibition in a cellular model of familial amyotrophic lateral sclerosis.
    Rizzardini M; Lupi M; Mangolini A; Babetto E; Ubezio P; Cantoni L
    Brain Res Bull; 2006 Apr; 69(4):465-74. PubMed ID: 16624679
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Metabolomic Analysis Reveals Increased Aerobic Glycolysis and Amino Acid Deficit in a Cellular Model of Amyotrophic Lateral Sclerosis.
    Valbuena GN; Rizzardini M; Cimini S; Siskos AP; Bendotti C; Cantoni L; Keun HC
    Mol Neurobiol; 2016 May; 53(4):2222-40. PubMed ID: 25963727
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Trehalose decreases mutant SOD1 expression and alleviates motor deficiency in early but not end-stage amyotrophic lateral sclerosis in a SOD1-G93A mouse model.
    Li Y; Guo Y; Wang X; Yu X; Duan W; Hong K; Wang J; Han H; Li C
    Neuroscience; 2015 Jul; 298():12-25. PubMed ID: 25841320
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M; Giribaldi F; Melone M; Bonifacino T; Musante I; Carminati E; Rossi PI; Vergani L; Voci A; Conti F; Puliti A; Bonanno G
    Neurobiol Dis; 2014 Apr; 64():48-59. PubMed ID: 24361555
    [TBL] [Abstract][Full Text] [Related]  

  • 12. HoxB2 binds mutant SOD1 and is altered in transgenic model of ALS.
    Zhai J; Lin H; Canete-Soler R; Schlaepfer WW
    Hum Mol Genet; 2005 Sep; 14(18):2629-40. PubMed ID: 16079151
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Selective loss of neurofilament expression in Cu/Zn superoxide dismutase (SOD1) linked amyotrophic lateral sclerosis.
    Menzies FM; Grierson AJ; Cookson MR; Heath PR; Tomkins J; Figlewicz DA; Ince PG; Shaw PJ
    J Neurochem; 2002 Sep; 82(5):1118-28. PubMed ID: 12358759
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Cell culture models to investigate the selective vulnerability of motoneuronal mitochondria to familial ALS-linked G93ASOD1.
    Raimondi A; Mangolini A; Rizzardini M; Tartari S; Massari S; Bendotti C; Francolini M; Borgese N; Cantoni L; Pietrini G
    Eur J Neurosci; 2006 Jul; 24(2):387-99. PubMed ID: 16903849
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Increased mitochondrial antioxidative activity or decreased oxygen free radical propagation prevent mutant SOD1-mediated motor neuron cell death and increase amyotrophic lateral sclerosis-like transgenic mouse survival.
    Liu R; Li B; Flanagan SW; Oberley LW; Gozal D; Qiu M
    J Neurochem; 2002 Feb; 80(3):488-500. PubMed ID: 11905995
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
    Beqollari D; Romberg CF; Dobrowolny G; Martini M; Voss AA; MusarĂ² A; Bannister RA
    Skelet Muscle; 2016; 6():24. PubMed ID: 27340545
    [TBL] [Abstract][Full Text] [Related]  

  • 17. The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).
    Crippa V; Sau D; Rusmini P; Boncoraglio A; Onesto E; Bolzoni E; Galbiati M; Fontana E; Marino M; Carra S; Bendotti C; De Biasi S; Poletti A
    Hum Mol Genet; 2010 Sep; 19(17):3440-56. PubMed ID: 20570967
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Derlin-1 overexpression ameliorates mutant SOD1-induced endoplasmic reticulum stress by reducing mutant SOD1 accumulation.
    Mori A; Yamashita S; Uchino K; Suga T; Ikeda T; Takamatsu K; Ishizaki M; Koide T; Kimura E; Mita S; Maeda Y; Hirano T; Uchino M
    Neurochem Int; 2011 Feb; 58(3):344-53. PubMed ID: 21185345
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Calcium dysregulation, mitochondrial pathology and protein aggregation in a culture model of amyotrophic lateral sclerosis: mechanistic relationship and differential sensitivity to intervention.
    Tradewell ML; Cooper LA; Minotti S; Durham HD
    Neurobiol Dis; 2011 Jun; 42(3):265-75. PubMed ID: 21296666
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Stabilization of mutant Cu/Zn superoxide dismutase (SOD1) protein by coexpressed wild SOD1 protein accelerates the disease progression in familial amyotrophic lateral sclerosis mice.
    Fukada K; Nagano S; Satoh M; Tohyama C; Nakanishi T; Shimizu A; Yanagihara T; Sakoda S
    Eur J Neurosci; 2001 Dec; 14(12):2032-6. PubMed ID: 11860498
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 43.