BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

300 related articles for article (PubMed ID: 24399619)

  • 1. Inhibition of the leptin-induced activation of the p38 MAPK pathway contributes to the protective effects of naringin against high glucose-induced injury in H9c2 cardiac cells.
    Chen J; Mo H; Guo R; You Q; Huang R; Wu K
    Int J Mol Med; 2014 Mar; 33(3):605-12. PubMed ID: 24399619
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Exogenous hydrogen sulfide protects H9c2 cardiac cells against high glucose-induced injury by inhibiting the activities of the p38 MAPK and ERK1/2 pathways.
    Xu W; Wu W; Chen J; Guo R; Lin J; Liao X; Feng J
    Int J Mol Med; 2013 Oct; 32(4):917-25. PubMed ID: 23912965
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Naringin inhibits ROS-activated MAPK pathway in high glucose-induced injuries in H9c2 cardiac cells.
    Chen J; Guo R; Yan H; Tian L; You Q; Li S; Huang R; Wu K
    Basic Clin Pharmacol Toxicol; 2014 Apr; 114(4):293-304. PubMed ID: 24118820
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Exogenous H2S protects H9c2 cardiac cells against high glucose-induced injury and inflammation by inhibiting the activation of the NF-κB and IL-1β pathways.
    Xu W; Chen J; Lin J; Liu D; Mo L; Pan W; Feng J; Wu W; Zheng D
    Int J Mol Med; 2015 Jan; 35(1):177-86. PubMed ID: 25412187
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Exogenous hydrogen sulfide alleviates high glucose-induced cardiotoxicity via inhibition of leptin signaling in H9c2 cells.
    Zhuang XD; Hu X; Long M; Dong XB; Liu DH; Liao XX
    Mol Cell Biochem; 2014 Jun; 391(1-2):147-55. PubMed ID: 24687304
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Hydrogen sulfide attenuates doxorubicin-induced cardiotoxicity by inhibition of the p38 MAPK pathway in H9c2 cells.
    Guo R; Lin J; Xu W; Shen N; Mo L; Zhang C; Feng J
    Int J Mol Med; 2013 Mar; 31(3):644-50. PubMed ID: 23338126
    [TBL] [Abstract][Full Text] [Related]  

  • 7. The Protective Effect of INH2BP, a Novel PARP Inhibitor 5-Iodo-6-Amino-1,2-Benzopyrone, Against Hydrogen Peroxide-Induced Apoptosis Through ERK and p38 MAPK in H9c2 Cells.
    Back OC; Jang HS; Lee JS; Kim DE; Lee Y; Park ES; Kim IS
    Pharmacology; 2015; 96(5-6):259-70. PubMed ID: 26550832
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Angiotensin-(1-7) protects cardiomyocytes against high glucose-induced injuries through inhibiting reactive oxygen species-activated leptin-p38 mitogen-activated protein kinase/extracellular signal-regulated protein kinase 1/2 pathways, but not the leptin-c-Jun N-terminal kinase pathway in vitro.
    Lei Y; Xu Q; Zeng B; Zhang W; Zhen Y; Zhai Y; Cheng F; Mei W; Zheng D; Feng J; Lan J; Chen J
    J Diabetes Investig; 2017 Jul; 8(4):434-445. PubMed ID: 27896943
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Protective effect of angiotensin-(1-7) against hyperglycaemia-induced injury in H9c2 cardiomyoblast cells via the PI3K̸Akt signaling pathway.
    Yang YY; Sun XT; Li ZX; Chen WY; Wang X; Liang ML; Shi H; Yang ZS; Zeng WT
    Int J Mol Med; 2018 Mar; 41(3):1283-1292. PubMed ID: 29286068
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Exogenous hydrogen sulfide protects against doxorubicin-induced inflammation and cytotoxicity by inhibiting p38MAPK/NFκB pathway in H9c2 cardiac cells.
    Guo R; Wu K; Chen J; Mo L; Hua X; Zheng D; Chen P; Chen G; Xu W; Feng J
    Cell Physiol Biochem; 2013; 32(6):1668-80. PubMed ID: 24356372
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Naringin inhibits high glucose-induced cardiomyocyte apoptosis by attenuating mitochondrial dysfunction and modulating the activation of the p38 signaling pathway.
    Huang H; Wu K; You Q; Huang R; Li S; Wu K
    Int J Mol Med; 2013 Aug; 32(2):396-402. PubMed ID: 23732220
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Naringin attenuates high glucose-induced injuries and inflammation by modulating the leptin-JAK2/STAT3 pathway in H9c2 cardiac cells.
    Luo C; Ke X; Xiong S; Sun Y; Xu Q; Zhang W; Lei Y; Ding Y; Zhen Y; Feng J; Cheng F; Chen J
    Arch Med Sci; 2021; 17(5):1145-1157. PubMed ID: 34522243
    [TBL] [Abstract][Full Text] [Related]  

  • 13. ATP-sensitive K⁺ channels contribute to the protective effects of exogenous hydrogen sulfide against high glucose-induced injury in H9c2 cardiac cells.
    Liang W; Chen J; Mo L; Ke X; Zhang W; Zheng D; Pan W; Wu S; Feng J; Song M; Liao X
    Int J Mol Med; 2016 Mar; 37(3):763-72. PubMed ID: 26820501
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Curcumin induces the apoptotic intrinsic pathway via upregulation of reactive oxygen species and JNKs in H9c2 cardiac myoblasts.
    Zikaki K; Aggeli IK; Gaitanaki C; Beis I
    Apoptosis; 2014 Jun; 19(6):958-74. PubMed ID: 24668280
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Chlorogenic acid analogues from Gynura nepalensis protect H9c2 cardiomyoblasts against H
    Yu BW; Li JL; Guo BB; Fan HM; Zhao WM; Wang HY
    Acta Pharmacol Sin; 2016 Nov; 37(11):1413-1422. PubMed ID: 27593219
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Acetylcholine prevents angiotensin II-induced oxidative stress and apoptosis in H9c2 cells.
    Liu JJ; Li DL; Zhou J; Sun L; Zhao M; Kong SS; Wang YH; Yu XJ; Zhou J; Zang WJ
    Apoptosis; 2011 Jan; 16(1):94-103. PubMed ID: 20963497
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Exogenous hydrogen sulfide ameliorates high glucose-induced myocardial injury & inflammation via the CIRP-MAPK signaling pathway in H9c2 cardiac cells.
    Zhao HL; Wu BQ; Luo Y; Zhang WY; Hao YL; Liang JJ; Fang F; Liu W; Chen XH
    Life Sci; 2018 Sep; 208():315-324. PubMed ID: 29857073
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Activation of the p38 MAPK/NF-κB pathway contributes to doxorubicin-induced inflammation and cytotoxicity in H9c2 cardiac cells.
    Guo RM; Xu WM; Lin JC; Mo LQ; Hua XX; Chen PX; Wu K; Zheng DD; Feng JQ
    Mol Med Rep; 2013 Aug; 8(2):603-8. PubMed ID: 23807148
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Diallyl trisufide (DATS) suppresses high glucose-induced cardiomyocyte apoptosis by inhibiting JNK/NFκB signaling via attenuating ROS generation.
    Kuo WW; Wang WJ; Tsai CY; Way CL; Hsu HH; Chen LM
    Int J Cardiol; 2013 Sep; 168(1):270-80. PubMed ID: 23158927
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Taurine prevents arsenic-induced cardiac oxidative stress and apoptotic damage: role of NF-kappa B, p38 and JNK MAPK pathway.
    Ghosh J; Das J; Manna P; Sil PC
    Toxicol Appl Pharmacol; 2009 Oct; 240(1):73-87. PubMed ID: 19616567
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 15.