657 related articles for article (PubMed ID: 24522183)
1. Dysregulated interactions between lamin A and SUN1 induce abnormalities in the nuclear envelope and endoplasmic reticulum in progeric laminopathies.
Chen ZJ; Wang WP; Chen YC; Wang JY; Lin WH; Tai LA; Liou GG; Yang CS; Chi YH
J Cell Sci; 2014 Apr; 127(Pt 8):1792-804. PubMed ID: 24522183
[TBL] [Abstract][Full Text] [Related]
2. Incomplete processing of mutant lamin A in Hutchinson-Gilford progeria leads to nuclear abnormalities, which are reversed by farnesyltransferase inhibition.
Glynn MW; Glover TW
Hum Mol Genet; 2005 Oct; 14(20):2959-69. PubMed ID: 16126733
[TBL] [Abstract][Full Text] [Related]
3. Reversal of laminopathies: the curious case of SUN1.
Chi YH; Chen CY; Jeang KT
Nucleus; 2012; 3(5):418-21. PubMed ID: 22895095
[TBL] [Abstract][Full Text] [Related]
4. Nuclear localization signal deletion mutants of lamin A and progerin reveal insights into lamin A processing and emerin targeting.
Wu D; Flannery AR; Cai H; Ko E; Cao K
Nucleus; 2014; 5(1):66-74. PubMed ID: 24637396
[TBL] [Abstract][Full Text] [Related]
5. Inhibiting farnesylation reverses the nuclear morphology defect in a HeLa cell model for Hutchinson-Gilford progeria syndrome.
Mallampalli MP; Huyer G; Bendale P; Gelb MH; Michaelis S
Proc Natl Acad Sci U S A; 2005 Oct; 102(40):14416-21. PubMed ID: 16186497
[TBL] [Abstract][Full Text] [Related]
6. Accumulation of the inner nuclear envelope protein Sun1 is pathogenic in progeric and dystrophic laminopathies.
Chen CY; Chi YH; Mutalif RA; Starost MF; Myers TG; Anderson SA; Stewart CL; Jeang KT
Cell; 2012 Apr; 149(3):565-77. PubMed ID: 22541428
[TBL] [Abstract][Full Text] [Related]
7. Mammalian SUN protein interaction networks at the inner nuclear membrane and their role in laminopathy disease processes.
Haque F; Mazzeo D; Patel JT; Smallwood DT; Ellis JA; Shanahan CM; Shackleton S
J Biol Chem; 2010 Jan; 285(5):3487-98. PubMed ID: 19933576
[TBL] [Abstract][Full Text] [Related]
8. Imbalanced nucleocytoskeletal connections create common polarity defects in progeria and physiological aging.
Chang W; Wang Y; Luxton GWG; Östlund C; Worman HJ; Gundersen GG
Proc Natl Acad Sci U S A; 2019 Feb; 116(9):3578-3583. PubMed ID: 30808750
[TBL] [Abstract][Full Text] [Related]
9. Blocking farnesylation of the prelamin A variant in Hutchinson-Gilford progeria syndrome alters the distribution of A-type lamins.
Wang Y; Ostlund C; Choi JC; Swayne TC; Gundersen GG; Worman HJ
Nucleus; 2012; 3(5):452-62. PubMed ID: 22895092
[TBL] [Abstract][Full Text] [Related]
10. Progerin impairs chromosome maintenance by depleting CENP-F from metaphase kinetochores in Hutchinson-Gilford progeria fibroblasts.
Eisch V; Lu X; Gabriel D; Djabali K
Oncotarget; 2016 Apr; 7(17):24700-18. PubMed ID: 27015553
[TBL] [Abstract][Full Text] [Related]
11. A lamin A protein isoform overexpressed in Hutchinson-Gilford progeria syndrome interferes with mitosis in progeria and normal cells.
Cao K; Capell BC; Erdos MR; Djabali K; Collins FS
Proc Natl Acad Sci U S A; 2007 Mar; 104(12):4949-54. PubMed ID: 17360355
[TBL] [Abstract][Full Text] [Related]
12. Blocking protein farnesyltransferase improves nuclear blebbing in mouse fibroblasts with a targeted Hutchinson-Gilford progeria syndrome mutation.
Yang SH; Bergo MO; Toth JI; Qiao X; Hu Y; Sandoval S; Meta M; Bendale P; Gelb MH; Young SG; Fong LG
Proc Natl Acad Sci U S A; 2005 Jul; 102(29):10291-6. PubMed ID: 16014412
[TBL] [Abstract][Full Text] [Related]
13. Hutchinson-Gilford progeria syndrome through the lens of transcription.
Prokocimer M; Barkan R; Gruenbaum Y
Aging Cell; 2013 Aug; 12(4):533-43. PubMed ID: 23496208
[TBL] [Abstract][Full Text] [Related]
14. Inhibiting farnesylation of progerin prevents the characteristic nuclear blebbing of Hutchinson-Gilford progeria syndrome.
Capell BC; Erdos MR; Madigan JP; Fiordalisi JJ; Varga R; Conneely KN; Gordon LB; Der CJ; Cox AD; Collins FS
Proc Natl Acad Sci U S A; 2005 Sep; 102(36):12879-84. PubMed ID: 16129833
[TBL] [Abstract][Full Text] [Related]
15. Lamin A, farnesylation and aging.
Reddy S; Comai L
Exp Cell Res; 2012 Jan; 318(1):1-7. PubMed ID: 21871450
[TBL] [Abstract][Full Text] [Related]
16. The truncated prelamin A in Hutchinson-Gilford progeria syndrome alters segregation of A-type and B-type lamin homopolymers.
Delbarre E; Tramier M; Coppey-Moisan M; Gaillard C; Courvalin JC; Buendia B
Hum Mol Genet; 2006 Apr; 15(7):1113-22. PubMed ID: 16481358
[TBL] [Abstract][Full Text] [Related]
17. Dermal fibroblasts in Hutchinson-Gilford progeria syndrome with the lamin A G608G mutation have dysmorphic nuclei and are hypersensitive to heat stress.
Paradisi M; McClintock D; Boguslavsky RL; Pedicelli C; Worman HJ; Djabali K
BMC Cell Biol; 2005 Jun; 6():27. PubMed ID: 15982412
[TBL] [Abstract][Full Text] [Related]
18. Absence of progeria-like disease phenotypes in knock-in mice expressing a non-farnesylated version of progerin.
Yang SH; Chang SY; Ren S; Wang Y; Andres DA; Spielmann HP; Fong LG; Young SG
Hum Mol Genet; 2011 Feb; 20(3):436-44. PubMed ID: 21088111
[TBL] [Abstract][Full Text] [Related]
19. Epigenetic involvement in Hutchinson-Gilford progeria syndrome: a mini-review.
Arancio W; Pizzolanti G; Genovese SI; Pitrone M; Giordano C
Gerontology; 2014; 60(3):197-203. PubMed ID: 24603298
[TBL] [Abstract][Full Text] [Related]
20. Lamin A-linked progerias: is farnesylation the be all and end all?
Smallwood DT; Shackleton S
Biochem Soc Trans; 2010 Feb; 38(Pt 1):281-6. PubMed ID: 20074075
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]