BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

259 related articles for article (PubMed ID: 24573678)

  • 1. Insight into the roles of helicase motif Ia by characterizing Fanconi anemia group J protein (FANCJ) patient mutations.
    Guo M; Vidhyasagar V; Ding H; Wu Y
    J Biol Chem; 2014 Apr; 289(15):10551-10565. PubMed ID: 24573678
    [TBL] [Abstract][Full Text] [Related]  

  • 2. The Q motif of Fanconi anemia group J protein (FANCJ) DNA helicase regulates its dimerization, DNA binding, and DNA repair function.
    Wu Y; Sommers JA; Loiland JA; Kitao H; Kuper J; Kisker C; Brosh RM
    J Biol Chem; 2012 Jun; 287(26):21699-716. PubMed ID: 22582397
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Fanconi anemia group J mutation abolishes its DNA repair function by uncoupling DNA translocation from helicase activity or disruption of protein-DNA complexes.
    Wu Y; Sommers JA; Suhasini AN; Leonard T; Deakyne JS; Mazin AV; Shin-Ya K; Kitao H; Brosh RM
    Blood; 2010 Nov; 116(19):3780-91. PubMed ID: 20639400
    [TBL] [Abstract][Full Text] [Related]  

  • 4. A minimal threshold of FANCJ helicase activity is required for its response to replication stress or double-strand break repair.
    Bharti SK; Sommers JA; Awate S; Bellani MA; Khan I; Bradley L; King GA; Seol Y; Vidhyasagar V; Wu Y; Abe T; Kobayashi K; Shin-Ya K; Kitao H; Wold MS; Branzei D; Neuman KC; Brosh RM
    Nucleic Acids Res; 2018 Jul; 46(12):6238-6256. PubMed ID: 29788478
    [TBL] [Abstract][Full Text] [Related]  

  • 5. FANCJ helicase uniquely senses oxidative base damage in either strand of duplex DNA and is stimulated by replication protein A to unwind the damaged DNA substrate in a strand-specific manner.
    Suhasini AN; Sommers JA; Mason AC; Voloshin ON; Camerini-Otero RD; Wold MS; Brosh RM
    J Biol Chem; 2009 Jul; 284(27):18458-70. PubMed ID: 19419957
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Mutational analysis of FANCJ helicase.
    Guo M; Vidhyasagar V; Talwar T; Kariem A; Wu Y
    Methods; 2016 Oct; 108():118-29. PubMed ID: 27107905
    [TBL] [Abstract][Full Text] [Related]  

  • 7. FANCJ (BACH1) helicase forms DNA damage inducible foci with replication protein A and interacts physically and functionally with the single-stranded DNA-binding protein.
    Gupta R; Sharma S; Sommers JA; Kenny MK; Cantor SB; Brosh RM
    Blood; 2007 Oct; 110(7):2390-8. PubMed ID: 17596542
    [TBL] [Abstract][Full Text] [Related]  

  • 8. FANCJ helicase defective in Fanconia anemia and breast cancer unwinds G-quadruplex DNA to defend genomic stability.
    Wu Y; Shin-ya K; Brosh RM
    Mol Cell Biol; 2008 Jun; 28(12):4116-28. PubMed ID: 18426915
    [TBL] [Abstract][Full Text] [Related]  

  • 9. FANCJ uses its motor ATPase to destabilize protein-DNA complexes, unwind triplexes, and inhibit RAD51 strand exchange.
    Sommers JA; Rawtani N; Gupta R; Bugreev DV; Mazin AV; Cantor SB; Brosh RM
    J Biol Chem; 2009 Mar; 284(12):7505-17. PubMed ID: 19150983
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Specialization among iron-sulfur cluster helicases to resolve G-quadruplex DNA structures that threaten genomic stability.
    Bharti SK; Sommers JA; George F; Kuper J; Hamon F; Shin-ya K; Teulade-Fichou MP; Kisker C; Brosh RM
    J Biol Chem; 2013 Sep; 288(39):28217-29. PubMed ID: 23935105
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome.
    Suhasini AN; Rawtani NA; Wu Y; Sommers JA; Sharma S; Mosedale G; North PS; Cantor SB; Hickson ID; Brosh RM
    EMBO J; 2011 Feb; 30(4):692-705. PubMed ID: 21240188
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Novel function of the Fanconi anemia group J or RECQ1 helicase to disrupt protein-DNA complexes in a replication protein A-stimulated manner.
    Sommers JA; Banerjee T; Hinds T; Wan B; Wold MS; Lei M; Brosh RM
    J Biol Chem; 2014 Jul; 289(29):19928-41. PubMed ID: 24895130
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Fanconi anemia group J helicase and MRE11 nuclease interact to facilitate the DNA damage response.
    Suhasini AN; Sommers JA; Muniandy PA; Coulombe Y; Cantor SB; Masson JY; Seidman MM; Brosh RM
    Mol Cell Biol; 2013 Jun; 33(11):2212-27. PubMed ID: 23530059
    [TBL] [Abstract][Full Text] [Related]  

  • 14. The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells.
    Peng M; Litman R; Xie J; Sharma S; Brosh RM; Cantor SB
    EMBO J; 2007 Jul; 26(13):3238-49. PubMed ID: 17581638
    [TBL] [Abstract][Full Text] [Related]  

  • 15. FANCJ helicase operates in the Fanconi Anemia DNA repair pathway and the response to replicational stress.
    Wu Y; Brosh RM
    Curr Mol Med; 2009 May; 9(4):470-82. PubMed ID: 19519404
    [TBL] [Abstract][Full Text] [Related]  

  • 16. The Fanconi anemia proteins FANCD2 and FANCJ interact and regulate each other's chromatin localization.
    Chen X; Wilson JB; McChesney P; Williams SA; Kwon Y; Longerich S; Marriott AS; Sung P; Jones NJ; Kupfer GM
    J Biol Chem; 2014 Sep; 289(37):25774-82. PubMed ID: 25070891
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Purification and biochemical characterization of the G4 resolvase and DNA helicase FANCJ.
    Kulikowicz T; Sommers JA; Fuchs KF; Wu Y; Brosh RM
    Methods Enzymol; 2024; 695():1-27. PubMed ID: 38521581
    [TBL] [Abstract][Full Text] [Related]  

  • 18. FANCJ protein is important for the stability of FANCD2/FANCI proteins and protects them from proteasome and caspase-3 dependent degradation.
    Clark DW; Tripathi K; Dorsman JC; Palle K
    Oncotarget; 2015 Oct; 6(30):28816-32. PubMed ID: 26336824
    [TBL] [Abstract][Full Text] [Related]  

  • 19. FancJ (Brip1) loss-of-function allele results in spermatogonial cell depletion during embryogenesis and altered processing of crossover sites during meiotic prophase I in mice.
    Sun X; Brieño-Enríquez MA; Cornelius A; Modzelewski AJ; Maley TT; Campbell-Peterson KM; Holloway JK; Cohen PE
    Chromosoma; 2016 Jun; 125(2):237-52. PubMed ID: 26490168
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Inhibition of BACH1 (FANCJ) helicase by backbone discontinuity is overcome by increased motor ATPase or length of loading strand.
    Gupta R; Sharma S; Doherty KM; Sommers JA; Cantor SB; Brosh RM
    Nucleic Acids Res; 2006; 34(22):6673-83. PubMed ID: 17145708
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 13.