BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

587 related articles for article (PubMed ID: 24667415)

  • 1. Treatment with an antibody directed against Nogo-A delays disease progression in the SOD1G93A mouse model of Amyotrophic lateral sclerosis.
    Bros-Facer V; Krull D; Taylor A; Dick JR; Bates SA; Cleveland MS; Prinjha RK; Greensmith L
    Hum Mol Genet; 2014 Aug; 23(16):4187-200. PubMed ID: 24667415
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Delayed disease onset and extended survival in the SOD1G93A rat model of amyotrophic lateral sclerosis after suppression of mutant SOD1 in the motor cortex.
    Thomsen GM; Gowing G; Latter J; Chen M; Vit JP; Staggenborg K; Avalos P; Alkaslasi M; Ferraiuolo L; Likhite S; Kaspar BK; Svendsen CN
    J Neurosci; 2014 Nov; 34(47):15587-600. PubMed ID: 25411487
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Amyloid precursor protein (APP) contributes to pathology in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Bryson JB; Hobbs C; Parsons MJ; Bosch KD; Pandraud A; Walsh FS; Doherty P; Greensmith L
    Hum Mol Genet; 2012 Sep; 21(17):3871-82. PubMed ID: 22678056
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
    Beqollari D; Romberg CF; Dobrowolny G; Martini M; Voss AA; Musarò A; Bannister RA
    Skelet Muscle; 2016; 6():24. PubMed ID: 27340545
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Microglia and motor neurons during disease progression in the SOD1G93A mouse model of amyotrophic lateral sclerosis: changes in arginase1 and inducible nitric oxide synthase.
    Lewis KE; Rasmussen AL; Bennett W; King A; West AK; Chung RS; Chuah MI
    J Neuroinflammation; 2014 Mar; 11():55. PubMed ID: 24655927
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Functional over-load saves motor units in the SOD1-G93A transgenic mouse model of amyotrophic lateral sclerosis.
    Gordon T; Tyreman N; Li S; Putman CT; Hegedus J
    Neurobiol Dis; 2010 Feb; 37(2):412-22. PubMed ID: 19879358
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Phenotype of transgenic mice carrying a very low copy number of the mutant human G93A superoxide dismutase-1 gene associated with amyotrophic lateral sclerosis.
    Deitch JS; Alexander GM; Bensinger A; Yang S; Jiang JT; Heiman-Patterson TD
    PLoS One; 2014; 9(6):e99879. PubMed ID: 24945277
    [TBL] [Abstract][Full Text] [Related]  

  • 8. c-Abl inhibition delays motor neuron degeneration in the G93A mouse, an animal model of amyotrophic lateral sclerosis.
    Katsumata R; Ishigaki S; Katsuno M; Kawai K; Sone J; Huang Z; Adachi H; Tanaka F; Urano F; Sobue G
    PLoS One; 2012; 7(9):e46185. PubMed ID: 23049975
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M; Giribaldi F; Melone M; Bonifacino T; Musante I; Carminati E; Rossi PI; Vergani L; Voci A; Conti F; Puliti A; Bonanno G
    Neurobiol Dis; 2014 Apr; 64():48-59. PubMed ID: 24361555
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Toll-Like Receptor-4 Inhibitor TAK-242 Attenuates Motor Dysfunction and Spinal Cord Pathology in an Amyotrophic Lateral Sclerosis Mouse Model.
    Fellner A; Barhum Y; Angel A; Perets N; Steiner I; Offen D; Lev N
    Int J Mol Sci; 2017 Aug; 18(8):. PubMed ID: 28763002
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Axonal degeneration, distal collateral branching and neuromuscular junction architecture alterations occur prior to symptom onset in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Clark JA; Southam KA; Blizzard CA; King AE; Dickson TC
    J Chem Neuroanat; 2016 Oct; 76(Pt A):35-47. PubMed ID: 27038603
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Aberrant enteric neuromuscular system and dysbiosis in amyotrophic lateral sclerosis.
    Zhang Y; Ogbu D; Garrett S; Xia Y; Sun J
    Gut Microbes; 2021; 13(1):1996848. PubMed ID: 34812107
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Trehalose decreases mutant SOD1 expression and alleviates motor deficiency in early but not end-stage amyotrophic lateral sclerosis in a SOD1-G93A mouse model.
    Li Y; Guo Y; Wang X; Yu X; Duan W; Hong K; Wang J; Han H; Li C
    Neuroscience; 2015 Jul; 298():12-25. PubMed ID: 25841320
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Macrophage-mediated inflammation and glial response in the skeletal muscle of a rat model of familial amyotrophic lateral sclerosis (ALS).
    Van Dyke JM; Smit-Oistad IM; Macrander C; Krakora D; Meyer MG; Suzuki M
    Exp Neurol; 2016 Mar; 277():275-282. PubMed ID: 26775178
    [TBL] [Abstract][Full Text] [Related]  

  • 16. PACAP signaling exerts opposing effects on neuroprotection and neuroinflammation during disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Ringer C; Büning LS; Schäfer MK; Eiden LE; Weihe E; Schütz B
    Neurobiol Dis; 2013 Jun; 54():32-42. PubMed ID: 23466699
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Molecular chaperone mediated late-stage neuroprotection in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Novoselov SS; Mustill WJ; Gray AL; Dick JR; Kanuga N; Kalmar B; Greensmith L; Cheetham ME
    PLoS One; 2013; 8(8):e73944. PubMed ID: 24023695
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Different human copper-zinc superoxide dismutase mutants, SOD1G93A and SOD1H46R, exert distinct harmful effects on gross phenotype in mice.
    Pan L; Yoshii Y; Otomo A; Ogawa H; Iwasaki Y; Shang HF; Hadano S
    PLoS One; 2012; 7(3):e33409. PubMed ID: 22438926
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Preferential motor unit loss in the SOD1 G93A transgenic mouse model of amyotrophic lateral sclerosis.
    Hegedus J; Putman CT; Tyreman N; Gordon T
    J Physiol; 2008 Jul; 586(14):3337-51. PubMed ID: 18467368
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Intact single muscle fibres from SOD1
    Cheng AJ; Allodi I; Chaillou T; Schlittler M; Ivarsson N; Lanner JT; Thams S; Hedlund E; Andersson DC
    J Physiol; 2019 Jun; 597(12):3133-3146. PubMed ID: 31074054
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 30.