244 related articles for article (PubMed ID: 24781191)
1. Silencing VDAC1 Expression by siRNA Inhibits Cancer Cell Proliferation and Tumor Growth In Vivo.
Arif T; Vasilkovsky L; Refaely Y; Konson A; Shoshan-Barmatz V
Mol Ther Nucleic Acids; 2014 Apr; 3(4):e159. PubMed ID: 24781191
[TBL] [Abstract][Full Text] [Related]
2. VDAC1 is a molecular target in glioblastoma, with its depletion leading to reprogrammed metabolism and reversed oncogenic properties.
Arif T; Krelin Y; Nakdimon I; Benharroch D; Paul A; Dadon-Klein D; Shoshan-Barmatz V
Neuro Oncol; 2017 Jul; 19(7):951-964. PubMed ID: 28339833
[TBL] [Abstract][Full Text] [Related]
3. The mitochondrial voltage-dependent anion channel 1 in tumor cells.
Shoshan-Barmatz V; Ben-Hail D; Admoni L; Krelin Y; Tripathi SS
Biochim Biophys Acta; 2015 Oct; 1848(10 Pt B):2547-75. PubMed ID: 25448878
[TBL] [Abstract][Full Text] [Related]
4. Silencing the Mitochondrial Gatekeeper VDAC1 as a Potential Treatment for Bladder Cancer.
Alhozeel B; Pandey SK; Shteinfer-Kuzmine A; Santhanam M; Shoshan-Barmatz V
Cells; 2024 Apr; 13(7):. PubMed ID: 38607066
[TBL] [Abstract][Full Text] [Related]
5. Downregulation of voltage-dependent anion channel-1 expression by RNA interference prevents cancer cell growth in vivo.
Koren I; Raviv Z; Shoshan-Barmatz V
Cancer Biol Ther; 2010 Jun; 9(12):1046-52. PubMed ID: 20404552
[TBL] [Abstract][Full Text] [Related]
6. Metabolic Reprograming Via Silencing of Mitochondrial VDAC1 Expression Encourages Differentiation of Cancer Cells.
Arif T; Amsalem Z; Shoshan-Barmatz V
Mol Ther Nucleic Acids; 2019 Sep; 17():24-37. PubMed ID: 31195298
[TBL] [Abstract][Full Text] [Related]
7. Silencing VDAC1 to Treat Mesothelioma Cancer: Tumor Reprograming and Altering Tumor Hallmarks.
Pandey SK; Machlof-Cohen R; Santhanam M; Shteinfer-Kuzmine A; Shoshan-Barmatz V
Biomolecules; 2022 Jun; 12(7):. PubMed ID: 35883451
[TBL] [Abstract][Full Text] [Related]
8. Mitochondrial VDAC1 Silencing Leads to Metabolic Rewiring and the Reprogramming of Tumour Cells into Advanced Differentiated States.
Arif T; Paul A; Krelin Y; Shteinfer-Kuzmine A; Shoshan-Barmatz V
Cancers (Basel); 2018 Dec; 10(12):. PubMed ID: 30544833
[TBL] [Abstract][Full Text] [Related]
9. Reducing VDAC1 expression induces a non-apoptotic role for pro-apoptotic proteins in cancer cell differentiation.
Arif T; Krelin Y; Shoshan-Barmatz V
Biochim Biophys Acta; 2016 Aug; 1857(8):1228-1242. PubMed ID: 27080741
[TBL] [Abstract][Full Text] [Related]
10. Voltage-Dependent Anion Channel 1 As an Emerging Drug Target for Novel Anti-Cancer Therapeutics.
Shoshan-Barmatz V; Krelin Y; Shteinfer-Kuzmine A; Arif T
Front Oncol; 2017; 7():154. PubMed ID: 28824871
[TBL] [Abstract][Full Text] [Related]
11. An N-terminal nucleotide-binding site in VDAC1: involvement in regulating mitochondrial function.
Yehezkel G; Abu-Hamad S; Shoshan-Barmatz V
J Cell Physiol; 2007 Aug; 212(2):551-61. PubMed ID: 17503466
[TBL] [Abstract][Full Text] [Related]
12. The Voltage-dependent Anion Channel 1 Mediates Amyloid β Toxicity and Represents a Potential Target for Alzheimer Disease Therapy.
Smilansky A; Dangoor L; Nakdimon I; Ben-Hail D; Mizrachi D; Shoshan-Barmatz V
J Biol Chem; 2015 Dec; 290(52):30670-83. PubMed ID: 26542804
[TBL] [Abstract][Full Text] [Related]
13. VDAC1 Silencing in Cancer Cells Leads to Metabolic Reprogramming That Modulates Tumor Microenvironment.
Zerbib E; Arif T; Shteinfer-Kuzmine A; Chalifa-Caspi V; Shoshan-Barmatz V
Cancers (Basel); 2021 Jun; 13(11):. PubMed ID: 34200480
[TBL] [Abstract][Full Text] [Related]
14. Selective induction of cancer cell death by VDAC1-based peptides and their potential use in cancer therapy.
Shteinfer-Kuzmine A; Amsalem Z; Arif T; Zooravlov A; Shoshan-Barmatz V
Mol Oncol; 2018 Jun; 12(7):1077-1103. PubMed ID: 29698587
[TBL] [Abstract][Full Text] [Related]
15. Rewiring of Cancer Cell Metabolism by Mitochondrial VDAC1 Depletion Results in Time-Dependent Tumor Reprogramming: Glioblastoma as a Proof of Concept.
Arif T; Stern O; Pittala S; Chalifa-Caspi V; Shoshan-Barmatz V
Cells; 2019 Oct; 8(11):. PubMed ID: 31661894
[TBL] [Abstract][Full Text] [Related]
16. The expression level of the voltage-dependent anion channel controls life and death of the cell.
Abu-Hamad S; Sivan S; Shoshan-Barmatz V
Proc Natl Acad Sci U S A; 2006 Apr; 103(15):5787-92. PubMed ID: 16585511
[TBL] [Abstract][Full Text] [Related]
17. The Mitochondrial Protein VDAC1 at the Crossroads of Cancer Cell Metabolism: The Epigenetic Link.
Amsalem Z; Arif T; Shteinfer-Kuzmine A; Chalifa-Caspi V; Shoshan-Barmatz V
Cancers (Basel); 2020 Apr; 12(4):. PubMed ID: 32331482
[TBL] [Abstract][Full Text] [Related]
18. Clinical implication of voltage-dependent anion channel 1 in uterine cervical cancer and its action on cervical cancer cells.
Wu CH; Lin YW; Wu TF; Ko JL; Wang PH
Oncotarget; 2016 Jan; 7(4):4210-25. PubMed ID: 26716410
[TBL] [Abstract][Full Text] [Related]
19. Vdac1 Downregulation Causes Mitochondrial Disintegration Leading to Hippocampal Neurodegeneration in Scopolamine-Induced Amnesic Mice.
Baghel MS; Thakur MK
Mol Neurobiol; 2019 Mar; 56(3):1707-1718. PubMed ID: 29916145
[TBL] [Abstract][Full Text] [Related]
20. Mitochondrial VDAC1: A Key Gatekeeper as Potential Therapeutic Target.
Camara AKS; Zhou Y; Wen PC; Tajkhorshid E; Kwok WM
Front Physiol; 2017; 8():460. PubMed ID: 28713289
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]