215 related articles for article (PubMed ID: 24783061)
1. Commonly prescribed β-lactam antibiotics induce C. trachomatis persistence/stress in culture at physiologically relevant concentrations.
Kintner J; Lajoie D; Hall J; Whittimore J; Schoborg RV
Front Cell Infect Microbiol; 2014; 4():44. PubMed ID: 24783061
[TBL] [Abstract][Full Text] [Related]
2. Pre-exposure of infected human endometrial epithelial cells to penicillin in vitro renders Chlamydia trachomatis refractory to azithromycin.
Wyrick PB; Knight ST
J Antimicrob Chemother; 2004 Jul; 54(1):79-85. PubMed ID: 15163653
[TBL] [Abstract][Full Text] [Related]
3. Regulation of the Mitochondrion-Fatty Acid Axis for the Metabolic Reprogramming of Chlamydia trachomatis during Treatment with β-Lactam Antimicrobials.
Shima K; Kaufhold I; Eder T; Käding N; Schmidt N; Ogunsulire IM; Deenen R; Köhrer K; Friedrich D; Isay SE; Grebien F; Klinger M; Richer BC; Günther UL; Deepe GS; Rattei T; Rupp J
mBio; 2021 Mar; 12(2):. PubMed ID: 33785629
[TBL] [Abstract][Full Text] [Related]
4. Effects of sustained antibiotic bactericidal treatment on Chlamydia trachomatis-infected epithelial-like cells (HeLa) and monocyte-like cells (THP-1 and U-937).
Mpiga P; Ravaoarinoro M
Int J Antimicrob Agents; 2006 Apr; 27(4):316-24. PubMed ID: 16527461
[TBL] [Abstract][Full Text] [Related]
5. Metabolic dormancy in
Rockey DD; Wang X; Debrine A; Grieshaber N; Grieshaber SS
Infect Immun; 2024 Feb; 92(2):e0033923. PubMed ID: 38214508
[TBL] [Abstract][Full Text] [Related]
6. The protease inhibitor JO146 demonstrates a critical role for CtHtrA for Chlamydia trachomatis reversion from penicillin persistence.
Ong VA; Marsh JW; Lawrence A; Allan JA; Timms P; Huston WM
Front Cell Infect Microbiol; 2013; 3():100. PubMed ID: 24392355
[TBL] [Abstract][Full Text] [Related]
7. Chlamydia trachomatis persistence in vitro: an overview.
Wyrick PB
J Infect Dis; 2010 Jun; 201 Suppl 2(Suppl 2):S88-95. PubMed ID: 20470046
[TBL] [Abstract][Full Text] [Related]
8. Fosmidomycin, an inhibitor of isoprenoid synthesis, induces persistence in Chlamydia by inhibiting peptidoglycan assembly.
Slade JA; Brockett M; Singh R; Liechti GW; Maurelli AT
PLoS Pathog; 2019 Oct; 15(10):e1008078. PubMed ID: 31622442
[TBL] [Abstract][Full Text] [Related]
9. Chlamydia muridarum enters a viable but non-infectious state in amoxicillin-treated BALB/c mice.
Phillips Campbell R; Kintner J; Whittimore J; Schoborg RV
Microbes Infect; 2012 Nov; 14(13):1177-85. PubMed ID: 22943883
[TBL] [Abstract][Full Text] [Related]
10. Penicillin induced persistence in Chlamydia trachomatis: high quality time lapse video analysis of the developmental cycle.
Skilton RJ; Cutcliffen LT; Barlow D; Wang Y; Salim O; Lambden PR; Clarke IN
PLoS One; 2009 Nov; 4(11):e7723. PubMed ID: 19893744
[TBL] [Abstract][Full Text] [Related]
11. Low-dose doxycycline induces Chlamydia trachomatis persistence in HeLa cells.
Marangoni A; Zalambani C; Marziali G; Salvo M; Fato R; Foschi C; Re MC
Microb Pathog; 2020 Oct; 147():104347. PubMed ID: 32561420
[TBL] [Abstract][Full Text] [Related]
12. Porcine epidemic diarrhea virus (PEDV) co-infection induced chlamydial persistence/stress does not require viral replication.
Schoborg RV; Borel N
Front Cell Infect Microbiol; 2014; 4():20. PubMed ID: 24660163
[TBL] [Abstract][Full Text] [Related]
13. Productive and Penicillin-Stressed
Leonard CA; Schoborg RV; Borel N
Front Cell Infect Microbiol; 2017; 7():180. PubMed ID: 28553623
[TBL] [Abstract][Full Text] [Related]
14. In-vitro and in-vivo activity of parenterally administered beta-lactam antibiotics against Chlamydia trachomatis.
Martin DH; Pastorek JG; Faro S
Sex Transm Dis; 1986; 13(2):81-7. PubMed ID: 3520886
[TBL] [Abstract][Full Text] [Related]
15. An in vitro model of azithromycin-induced persistent Chlamydia trachomatis infection.
Xue Y; Zheng H; Mai Z; Qin X; Chen W; Huang T; Chen D; Zheng L
FEMS Microbiol Lett; 2017 Aug; 364(14):. PubMed ID: 28854672
[TBL] [Abstract][Full Text] [Related]
16. Chlamydia trachomatis enters a viable but non-cultivable (persistent) state within herpes simplex virus type 2 (HSV-2) co-infected host cells.
Deka S; Vanover J; Dessus-Babus S; Whittimore J; Howett MK; Wyrick PB; Schoborg RV
Cell Microbiol; 2006 Jan; 8(1):149-62. PubMed ID: 16367874
[TBL] [Abstract][Full Text] [Related]
17. Chlamydia trachomatis responds to heat shock, penicillin induced persistence, and IFN-gamma persistence by altering levels of the extracytoplasmic stress response protease HtrA.
Huston WM; Theodoropoulos C; Mathews SA; Timms P
BMC Microbiol; 2008 Nov; 8():190. PubMed ID: 18986550
[TBL] [Abstract][Full Text] [Related]
18. Host nectin-1 is required for efficient Chlamydia trachomatis serovar E development.
Hall JV; Sun J; Slade J; Kintner J; Bambino M; Whittimore J; Schoborg RV
Front Cell Infect Microbiol; 2014; 4():158. PubMed ID: 25414835
[TBL] [Abstract][Full Text] [Related]
19. Golgi fragmentation and sphingomyelin transport to Chlamydia trachomatis during penicillin-induced persistence do not depend on the cytosolic presence of the chlamydial protease CPAF.
Dille S; Herbst K; Volceanov L; Nölke T; Kretz O; Häcker G
PLoS One; 2014; 9(7):e103220. PubMed ID: 25068694
[TBL] [Abstract][Full Text] [Related]
20. An early event in the herpes simplex virus type-2 replication cycle is sufficient to induce Chlamydia trachomatis persistence.
Deka S; Vanover J; Sun J; Kintner J; Whittimore J; Schoborg RV
Cell Microbiol; 2007 Mar; 9(3):725-37. PubMed ID: 17140408
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
