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2. Mouse-hamster chimeric prion protein (PrP) devoid of N-terminal residues 23-88 restores susceptibility to 22L prions, but not to RML prions in PrP-knockout mice. Uchiyama K; Miyata H; Yano M; Yamaguchi Y; Imamura M; Muramatsu N; Das NR; Chida J; Hara H; Sakaguchi S PLoS One; 2014; 9(10):e109737. PubMed ID: 25330286 [TBL] [Abstract][Full Text] [Related]
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8. Prion Protein Devoid of the Octapeptide Repeat Region Delays Bovine Spongiform Encephalopathy Pathogenesis in Mice. Hara H; Miyata H; Das NR; Chida J; Yoshimochi T; Uchiyama K; Watanabe H; Kondoh G; Yokoyama T; Sakaguchi S J Virol; 2018 Jan; 92(1):. PubMed ID: 29046443 [TBL] [Abstract][Full Text] [Related]
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10. The Ninth Datta Lecture. Molecular biology of transmissible spongiform encephalopathies. Weissmann C FEBS Lett; 1996 Jun; 389(1):3-11. PubMed ID: 8682199 [TBL] [Abstract][Full Text] [Related]
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14. Cholesterol transporter ATP-binding cassette A1 (ABCA1) is elevated in prion disease and affects PrPC and PrPSc concentrations in cultured cells. Kumar R; McClain D; Young R; Carlson GA J Gen Virol; 2008 Jun; 89(Pt 6):1525-1532. PubMed ID: 18474570 [TBL] [Abstract][Full Text] [Related]
15. Inhibition of PrPSc formation by lentiviral gene transfer of PrP containing dominant negative mutations. Crozet C; Lin YL; Mettling C; Mourton-Gilles C; Corbeau P; Lehmann S; Perrier V J Cell Sci; 2004 Nov; 117(Pt 23):5591-7. PubMed ID: 15494372 [TBL] [Abstract][Full Text] [Related]
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