254 related articles for article (PubMed ID: 25030372)
1. Hypoxia provokes base excision repair changes and a repair-deficient, mutator phenotype in colorectal cancer cells.
Chan N; Ali M; McCallum GP; Kumareswaran R; Koritzinsky M; Wouters BG; Wells PG; Gallinger S; Bristow RG
Mol Cancer Res; 2014 Oct; 12(10):1407-15. PubMed ID: 25030372
[TBL] [Abstract][Full Text] [Related]
2. Expression of human oxoguanine glycosylase 1 or formamidopyrimidine glycosylase in human embryonic kidney 293 cells exacerbates methylmercury toxicity in vitro.
Ondovcik SL; Preston TJ; McCallum GP; Wells PG
Toxicol Appl Pharmacol; 2013 Aug; 271(1):41-8. PubMed ID: 23607987
[TBL] [Abstract][Full Text] [Related]
3. An association selected polymorphisms of XRCC1, OGG1 and MUTYH gene and the level of efficiency oxidative DNA damage repair with a risk of colorectal cancer.
Przybylowska K; Kabzinski J; Sygut A; Dziki L; Dziki A; Majsterek I
Mutat Res; 2013; 745-746():6-15. PubMed ID: 23618615
[TBL] [Abstract][Full Text] [Related]
4. Activity of OGG1 variants in the repair of pro-oxidant-induced 8-oxo-2'-deoxyguanosine.
Smart DJ; Chipman JK; Hodges NJ
DNA Repair (Amst); 2006 Nov; 5(11):1337-45. PubMed ID: 16861056
[TBL] [Abstract][Full Text] [Related]
5. Cells with pathogenic biallelic mutations in the human MUTYH gene are defective in DNA damage binding and repair.
Parker AR; Sieber OM; Shi C; Hua L; Takao M; Tomlinson IP; Eshleman JR
Carcinogenesis; 2005 Nov; 26(11):2010-8. PubMed ID: 15987719
[TBL] [Abstract][Full Text] [Related]
6. Different organization of base excision repair of uracil in DNA in nuclei and mitochondria and selective upregulation of mitochondrial uracil-DNA glycosylase after oxidative stress.
Akbari M; Otterlei M; Peña-Diaz J; Krokan HE
Neuroscience; 2007 Apr; 145(4):1201-12. PubMed ID: 17101234
[TBL] [Abstract][Full Text] [Related]
7. In vivo measurements of interindividual differences in DNA glycosylases and APE1 activities.
Chaim IA; Nagel ZD; Jordan JJ; Mazzucato P; Ngo LP; Samson LD
Proc Natl Acad Sci U S A; 2017 Nov; 114(48):E10379-E10388. PubMed ID: 29122935
[TBL] [Abstract][Full Text] [Related]
8. First evidence for digenic inheritance in hereditary colorectal cancer by mutations in the base excision repair genes.
Morak M; Massdorf T; Sykora H; Kerscher M; Holinski-Feder E
Eur J Cancer; 2011 May; 47(7):1046-55. PubMed ID: 21195604
[TBL] [Abstract][Full Text] [Related]
9. Butin decreases oxidative stress-induced 8-hydroxy-2'-deoxyguanosine levels via activation of oxoguanine glycosylase 1.
Kang KA; Lee JH; Chae S; Zhang R; Piao MJ; Kim HS; You HJ; Hyun JW
Chem Biol Interact; 2009 Oct; 181(3):338-42. PubMed ID: 19631197
[TBL] [Abstract][Full Text] [Related]
10. A combinatorial role for MutY and Fpg DNA glycosylases in mutation avoidance in Mycobacterium smegmatis.
Hassim F; Papadopoulos AO; Kana BD; Gordhan BG
Mutat Res; 2015 Sep; 779():24-32. PubMed ID: 26125998
[TBL] [Abstract][Full Text] [Related]
11. Germline mutations but not somatic changes at the MYH locus contribute to the pathogenesis of unselected colorectal cancers.
Halford SE; Rowan AJ; Lipton L; Sieber OM; Pack K; Thomas HJ; Hodgson SV; Bodmer WF; Tomlinson IP
Am J Pathol; 2003 May; 162(5):1545-8. PubMed ID: 12707038
[TBL] [Abstract][Full Text] [Related]
12. Loss of MUTYH function in human cells leads to accumulation of oxidative damage and genetic instability.
Ruggieri V; Pin E; Russo MT; Barone F; Degan P; Sanchez M; Quaia M; Minoprio A; Turco E; Mazzei F; Viel A; Bignami M
Oncogene; 2013 Sep; 32(38):4500-8. PubMed ID: 23108399
[TBL] [Abstract][Full Text] [Related]
13. Ischemia-induced K-ras mutations in human colorectal cancer cells: role of microenvironmental regulation of MSH2 expression.
Shahrzad S; Quayle L; Stone C; Plumb C; Shirasawa S; Rak JW; Coomber BL
Cancer Res; 2005 Sep; 65(18):8134-41. PubMed ID: 16166287
[TBL] [Abstract][Full Text] [Related]
14. Distinct functional consequences of MUTYH variants associated with colorectal cancer: Damaged DNA affinity, glycosylase activity and interaction with PCNA and Hus1.
Brinkmeyer MK; David SS
DNA Repair (Amst); 2015 Oct; 34():39-51. PubMed ID: 26377631
[TBL] [Abstract][Full Text] [Related]
15. Alterations of the base excision repair gene MUTYH in sporadic colorectal cancer.
Kuno T; Matsubara N; Tsuda S; Kobayashi M; Hamanaka M; Yamagishi D; Tsukamoto K; Yamano T; Noda M; Ikeuchi H; Kim S; Tamura K; Tomita N
Oncol Rep; 2012 Aug; 28(2):473-80. PubMed ID: 22641385
[TBL] [Abstract][Full Text] [Related]
16. In vivo treatment with aflatoxin B1 increases DNA oxidation, base excision repair activity and 8-oxoguanine DNA glycosylase 1 levels in mouse lung.
Guindon-Kezis KA; Mulder JE; Massey TE
Toxicology; 2014 Jul; 321():21-6. PubMed ID: 24675474
[TBL] [Abstract][Full Text] [Related]
17. Oxidative Damage in Sporadic Colorectal Cancer: Molecular Mapping of Base Excision Repair Glycosylases MUTYH and hOGG1 in Colorectal Cancer Patients.
Kavec MJ; Urbanova M; Makovicky P; Opattová A; Tomasova K; Kroupa M; Kostovcikova K; Siskova A; Navvabi N; Schneiderova M; Vymetalkova V; Vodickova L; Vodicka P
Int J Mol Sci; 2022 May; 23(10):. PubMed ID: 35628513
[TBL] [Abstract][Full Text] [Related]
18. Differential DNA damage response to UV and hydrogen peroxide depending of differentiation stage in a neuroblastoma model.
Ramos-Espinosa P; Rojas E; Valverde M
Neurotoxicology; 2012 Oct; 33(5):1086-95. PubMed ID: 22687812
[TBL] [Abstract][Full Text] [Related]
19. Base excision repair modulation as a risk factor for human cancers.
Tudek B
Mol Aspects Med; 2007; 28(3-4):258-75. PubMed ID: 17628657
[TBL] [Abstract][Full Text] [Related]
20. Repair of 8-oxoG:A mismatches by the MUTYH glycosylase: Mechanism, metals and medicine.
Banda DM; Nuñez NN; Burnside MA; Bradshaw KM; David SS
Free Radic Biol Med; 2017 Jun; 107():202-215. PubMed ID: 28087410
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]