BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

144 related articles for article (PubMed ID: 25108129)

  • 1. Exposure to bisphenol A at physiological concentrations observed in Chinese children promotes primordial follicle growth through the PI3K/Akt pathway in an ovarian culture system.
    Zhao Q; Ma Y; Sun NX; Ye C; Zhang Q; Sun SH; Xu C; Wang F; Li W
    Toxicol In Vitro; 2014 Dec; 28(8):1424-9. PubMed ID: 25108129
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Bisphenol A-Induced Ovotoxicity Involves DNA Damage Induction to Which the Ovary Mounts a Protective Response Indicated by Increased Expression of Proteins Involved in DNA Repair and Xenobiotic Biotransformation.
    Ganesan S; Keating AF
    Toxicol Sci; 2016 Jul; 152(1):169-80. PubMed ID: 27208089
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Bisphenol A Initiates Excessive Premature Activation of Primordial Follicles in Mouse Ovaries via the PTEN Signaling Pathway.
    Hu Y; Yuan DZ; Wu Y; Yu LL; Xu LZ; Yue LM; Liu L; Xu WM; Qiao XY; Zeng RJ; Yang ZL; Yin WY; Ma YX; Nie Y
    Reprod Sci; 2018 Apr; 25(4):609-620. PubMed ID: 28982275
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Di-(2-ethylhexyl) phthalate and bisphenol A exposure impairs mouse primordial follicle assembly in vitro.
    Zhang T; Li L; Qin XS; Zhou Y; Zhang XF; Wang LQ; De Felici M; Chen H; Qin GQ; Shen W
    Environ Mol Mutagen; 2014 May; 55(4):343-53. PubMed ID: 24458533
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Rapamycin Prevents cyclophosphamide-induced Over-activation of Primordial Follicle pool through PI3K/Akt/mTOR Signaling Pathway in vivo.
    Zhou L; Xie Y; Li S; Liang Y; Qiu Q; Lin H; Zhang Q
    J Ovarian Res; 2017 Aug; 10(1):56. PubMed ID: 28814333
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Bisphenol A induce ovarian cancer cell migration via the MAPK and PI3K/Akt signalling pathways.
    Ptak A; Hoffmann M; Gruca I; Barć J
    Toxicol Lett; 2014 Sep; 229(2):357-65. PubMed ID: 24997338
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Quercetin prevents primordial follicle loss via suppression of PI3K/Akt/Foxo3a pathway activation in cyclophosphamide-treated mice.
    Li J; Long H; Cong Y; Gao H; Lyu Q; Yu S; Kuang Y
    Reprod Biol Endocrinol; 2021 Apr; 19(1):63. PubMed ID: 33892727
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Rapamycin-sensitive mTORC1 signaling is involved in physiological primordial follicle activation in mouse ovary.
    Tong Y; Li F; Lu Y; Cao Y; Gao J; Liu J
    Mol Reprod Dev; 2013 Dec; 80(12):1018-34. PubMed ID: 24123525
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Dynamics of PI3K and Hippo signaling pathways during in vitro human follicle activation.
    Grosbois J; Demeestere I
    Hum Reprod; 2018 Sep; 33(9):1705-1714. PubMed ID: 30032281
    [TBL] [Abstract][Full Text] [Related]  

  • 10. The imbalance of Treg/Th17 cells induced by perinatal bisphenol A exposure is associated with activation of the PI3K/Akt/mTOR signaling pathway in male offspring mice.
    Gao L; Dong Y; Lin R; Meng Y; Wu F; Jia L
    Food Chem Toxicol; 2020 Mar; 137():111177. PubMed ID: 32028014
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Maternal exposure to PM2.5 decreases ovarian reserve in neonatal offspring mice through activating PI3K/AKT/FoxO3a pathway and ROS-dependent NF-κB pathway.
    Chen Y; Xi Y; Li M; Wu Y; Yan W; Dai J; Wu M; Ding W; Zhang J; Zhang F; Zhou S; Wang S
    Toxicology; 2022 Nov; 481():153352. PubMed ID: 36243239
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Effects of bisphenol A on ovarian follicular development and female germline stem cells.
    Zhu X; Tian GG; Yu B; Yang Y; Wu J
    Arch Toxicol; 2018 Apr; 92(4):1581-1591. PubMed ID: 29380011
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Staying alive: PI3K pathway promotes primordial follicle activation and survival in response to 3MC-induced ovotoxicity.
    Sobinoff AP; Nixon B; Roman SD; McLaughlin EA
    Toxicol Sci; 2012 Jul; 128(1):258-71. PubMed ID: 22505044
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Understanding the Villain: DMBA-induced preantral ovotoxicity involves selective follicular destruction and primordial follicle activation through PI3K/Akt and mTOR signaling.
    Sobinoff AP; Mahony M; Nixon B; Roman SD; McLaughlin EA
    Toxicol Sci; 2011 Oct; 123(2):563-75. PubMed ID: 21785161
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Neonatal exposure to bisphenol A reduces the pool of primordial follicles in the rat ovary.
    Rodríguez HA; Santambrosio N; Santamaría CG; Muñoz-de-Toro M; Luque EH
    Reprod Toxicol; 2010 Dec; 30(4):550-7. PubMed ID: 20692330
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Insulin-like growth factor-1 (IGF-1) promotes primordial follicle growth and reduces DNA fragmentation through the phosphatidylinositol 3-kinase/protein kinase B (PI3K/AKT) signalling pathway.
    Bezerra MÉS; Barberino RS; Menezes VG; Gouveia BB; Macedo TJS; Santos JMS; Monte APO; Barros VRP; Matos MHT
    Reprod Fertil Dev; 2018 Oct; 30(11):1503-1513. PubMed ID: 29843892
    [TBL] [Abstract][Full Text] [Related]  

  • 17. In utero bisphenol A exposure disturbs germ cell cyst breakdown through the PI3k/Akt signaling pathway and BDNF expression.
    Li C; Qi T; Ma L; Lan YB; Luo J; Chu K; Huang Y; Ruan F; Zhou J
    Ecotoxicol Environ Saf; 2023 Jul; 259():115031. PubMed ID: 37210998
    [TBL] [Abstract][Full Text] [Related]  

  • 18. The proto-oncogene c-src is involved in primordial follicle activation through the PI3K, PKC and MAPK signaling pathways.
    Du XY; Huang J; Xu LQ; Tang DF; Wu L; Zhang LX; Pan XL; Chen WY; Zheng LP; Zheng YH
    Reprod Biol Endocrinol; 2012 Aug; 10():58. PubMed ID: 22905678
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Bisphenol A accelerates meiotic progression in embryonic chickens via the estrogen receptor β signaling pathway.
    Yu M; Xu Y; Li M; Li D; Lu Y; Yu D; Du W
    Gen Comp Endocrinol; 2018 Apr; 259():66-75. PubMed ID: 29113915
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Bisphenol A induces leptin receptor expression, creating more binding sites for leptin, and activates the JAK/Stat, MAPK/ERK and PI3K/Akt signalling pathways in human ovarian cancer cell.
    Ptak A; Gregoraszczuk EL
    Toxicol Lett; 2012 May; 210(3):332-7. PubMed ID: 22343039
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 8.