251 related articles for article (PubMed ID: 25117625)
1. Nonhomologous end-joining repair plays a more important role than homologous recombination repair in defining radiosensitivity after exposure to high-LET radiation.
Takahashi A; Kubo M; Ma H; Nakagawa A; Yoshida Y; Isono M; Kanai T; Ohno T; Furusawa Y; Funayama T; Kobayashi Y; Nakano T
Radiat Res; 2014 Sep; 182(3):338-44. PubMed ID: 25117625
[TBL] [Abstract][Full Text] [Related]
2. Regulation of ATM in DNA double strand break repair accounts for the radiosensitivity in human cells exposed to high linear energy transfer ionizing radiation.
Xue L; Yu D; Furusawa Y; Okayasu R; Tong J; Cao J; Fan S
Mutat Res; 2009 Nov; 670(1-2):15-23. PubMed ID: 19583974
[TBL] [Abstract][Full Text] [Related]
3. Single-strand annealing, conservative homologous recombination, nonhomologous DNA end joining, and the cell cycle-dependent repair of DNA double-strand breaks induced by sparsely or densely ionizing radiation.
Frankenberg-Schwager M; Gebauer A; Koppe C; Wolf H; Pralle E; Frankenberg D
Radiat Res; 2009 Mar; 171(3):265-73. PubMed ID: 19267553
[TBL] [Abstract][Full Text] [Related]
4. The role of nonhomologous DNA end joining, conservative homologous recombination, and single-strand annealing in the cell cycle-dependent repair of DNA double-strand breaks induced by H(2)O(2) in mammalian cells.
Frankenberg-Schwager M; Becker M; Garg I; Pralle E; Wolf H; Frankenberg D
Radiat Res; 2008 Dec; 170(6):784-93. PubMed ID: 19138034
[TBL] [Abstract][Full Text] [Related]
5. Most DNA repair defects do not modify the relationship between relative biological effectiveness and linear energy transfer in CRISPR-edited cells.
Guerra Liberal FDC; Parsons JL; McMahon SJ
Med Phys; 2024 Jan; 51(1):591-600. PubMed ID: 37753877
[TBL] [Abstract][Full Text] [Related]
6. DNA double strand breaks induced by the indirect effect of radiation are more efficiently repaired by non-homologous end joining compared to homologous recombination repair.
Bajinskis A; Natarajan AT; Erixon K; Harms-Ringdahl M
Mutat Res; 2013 Aug; 756(1-2):21-9. PubMed ID: 23811167
[TBL] [Abstract][Full Text] [Related]
7. Comparison of the bromodeoxyuridine-mediated sensitization effects between low-LET and high-LET ionizing radiation on DNA double-strand breaks.
Fujii Y; Genet MD; Roybal EJ; Kubota N; Okayasu R; Miyagawa K; Fujimori A; Kato TA
Oncol Rep; 2013 Jun; 29(6):2133-9. PubMed ID: 23525528
[TBL] [Abstract][Full Text] [Related]
8. The repair of environmentally relevant DNA double strand breaks caused by high linear energy transfer irradiation--no simple task.
Moore S; Stanley FK; Goodarzi AA
DNA Repair (Amst); 2014 May; 17():64-73. PubMed ID: 24565812
[TBL] [Abstract][Full Text] [Related]
9. Comparison of cellular lethality in DNA repair-proficient or -deficient cell lines resulting from exposure to 70 MeV/n protons or 290 MeV/n carbon ions.
Genet SC; Maeda J; Fujisawa H; Yurkon CR; Fujii Y; Romero AM; Genik PC; Fujimori A; Kitamura H; Kato TA
Oncol Rep; 2012 Nov; 28(5):1591-6. PubMed ID: 22923057
[TBL] [Abstract][Full Text] [Related]
10. The major DNA repair pathway after both proton and carbon-ion radiation is NHEJ, but the HR pathway is more relevant in carbon ions.
Gerelchuluun A; Manabe E; Ishikawa T; Sun L; Itoh K; Sakae T; Suzuki K; Hirayama R; Asaithamby A; Chen DJ; Tsuboi K
Radiat Res; 2015 Mar; 183(3):345-56. PubMed ID: 25738894
[TBL] [Abstract][Full Text] [Related]
11. The link between cell-cycle dependent radiosensitivity and repair pathways: a model based on the local, sister-chromatid conformation dependent switch between NHEJ and HR.
Hufnagl A; Herr L; Friedrich T; Durante M; Taucher-Scholz G; Scholz M
DNA Repair (Amst); 2015 Mar; 27():28-39. PubMed ID: 25629437
[TBL] [Abstract][Full Text] [Related]
12. Combining carbon ion irradiation and non-homologous end-joining repair inhibitor NU7026 efficiently kills cancer cells.
Ma H; Takahashi A; Yoshida Y; Adachi A; Kanai T; Ohno T; Nakano T
Radiat Oncol; 2015 Nov; 10():225. PubMed ID: 26553138
[TBL] [Abstract][Full Text] [Related]
13. Inhibition of homologous recombination by hyperthermia shunts early double strand break repair to non-homologous end-joining.
Bergs JW; Krawczyk PM; Borovski T; ten Cate R; Rodermond HM; Stap J; Medema JP; Haveman J; Essers J; van Bree C; Stalpers LJ; Kanaar R; Aten JA; Franken NA
DNA Repair (Amst); 2013 Jan; 12(1):38-45. PubMed ID: 23237939
[TBL] [Abstract][Full Text] [Related]
14. Nonhomologous End Joining Is More Important Than Proton Linear Energy Transfer in Dictating Cell Death.
Bright SJ; Flint DB; Chakraborty S; McFadden CH; Yoon DS; Bronk L; Titt U; Mohan R; Grosshans DR; Sumazin P; Shaitelman SF; Asaithamby A; Sawakuchi GO
Int J Radiat Oncol Biol Phys; 2019 Dec; 105(5):1119-1125. PubMed ID: 31425731
[TBL] [Abstract][Full Text] [Related]
15. Impairment of the non-homologous end joining and homologous recombination pathways of DNA double strand break repair: Impact on spontaneous and radiation-induced mammary and intestinal tumour risk in Apc min/+ mice.
Haines JW; Coster M; Bouffler SD
DNA Repair (Amst); 2015 Nov; 35():19-26. PubMed ID: 26435054
[TBL] [Abstract][Full Text] [Related]
16. The impact of DNA double-strand break repair pathways throughout the carbon ion spread-out Bragg peak beam.
Buglewicz DJ; Buglewicz JKF; Hirakawa H; Kato TA; Liu C; Fang Y; Kusumoto T; Fujimori A; Sai S
Cancer Sci; 2023 Dec; 114(12):4548-4557. PubMed ID: 37786999
[TBL] [Abstract][Full Text] [Related]
17. Investigation of switch from ATM to ATR signaling at the sites of DNA damage induced by low and high LET radiation.
Saha J; Wang M; Cucinotta FA
DNA Repair (Amst); 2013 Dec; 12(12):1143-51. PubMed ID: 24238855
[TBL] [Abstract][Full Text] [Related]
18. S-phase cells are more sensitive to high-linear energy transfer radiation.
Wang H; Liu S; Zhang P; Zhang S; Naidu M; Wang H; Wang Y
Int J Radiat Oncol Biol Phys; 2009 Jul; 74(4):1236-41. PubMed ID: 19545789
[TBL] [Abstract][Full Text] [Related]
19. ATR signaling cooperates with ATM in the mechanism of low dose hypersensitivity induced by carbon ion beam.
Xue L; Furusawa Y; Yu D
DNA Repair (Amst); 2015 Oct; 34():1-8. PubMed ID: 26246317
[TBL] [Abstract][Full Text] [Related]
20. Genistein sensitizes glioblastoma cells to carbon ions via inhibiting DNA-PKcs phosphorylation and subsequently repressing NHEJ and delaying HR repair pathways.
Liu X; Li P; Hirayama R; Niu Y; Liu X; Chen W; Jin X; Zhang P; Ye F; Zhao T; Liu B; Li Q
Radiother Oncol; 2018 Oct; 129(1):84-94. PubMed ID: 29685705
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
