324 related articles for article (PubMed ID: 25313866)
1. Early intrinsic hyperexcitability does not contribute to motoneuron degeneration in amyotrophic lateral sclerosis.
Leroy F; Lamotte d'Incamps B; Imhoff-Manuel RD; Zytnicki D
Elife; 2014 Oct; 3():. PubMed ID: 25313866
[TBL] [Abstract][Full Text] [Related]
2. Inhibitory synaptic regulation of motoneurons: a new target of disease mechanisms in amyotrophic lateral sclerosis.
Martin LJ; Chang Q
Mol Neurobiol; 2012 Feb; 45(1):30-42. PubMed ID: 22072396
[TBL] [Abstract][Full Text] [Related]
3. Hypoexcitability precedes denervation in the large fast-contracting motor units in two unrelated mouse models of ALS.
Martínez-Silva ML; Imhoff-Manuel RD; Sharma A; Heckman CJ; Shneider NA; Roselli F; Zytnicki D; Manuel M
Elife; 2018 Mar; 7():. PubMed ID: 29580378
[TBL] [Abstract][Full Text] [Related]
4. Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice.
Kuo JJ; Schonewille M; Siddique T; Schults AN; Fu R; Bär PR; Anelli R; Heckman CJ; Kroese AB
J Neurophysiol; 2004 Jan; 91(1):571-5. PubMed ID: 14523070
[TBL] [Abstract][Full Text] [Related]
5. Hyperexcitability in synaptic and firing activities of spinal motoneurons in an adult mouse model of amyotrophic lateral sclerosis.
Jiang MC; Adimula A; Birch D; Heckman CJ
Neuroscience; 2017 Oct; 362():33-46. PubMed ID: 28844763
[TBL] [Abstract][Full Text] [Related]
6. Homeostatic dysregulation in membrane properties of masticatory motoneurons compared with oculomotor neurons in a mouse model for amyotrophic lateral sclerosis.
Venugopal S; Hsiao CF; Sonoda T; Wiedau-Pazos M; Chandler SH
J Neurosci; 2015 Jan; 35(2):707-20. PubMed ID: 25589764
[TBL] [Abstract][Full Text] [Related]
7. Collapsin response mediator protein 4a (CRMP4a) is upregulated in motoneurons of mutant SOD1 mice and can trigger motoneuron axonal degeneration and cell death.
Duplan L; Bernard N; Casseron W; Dudley K; Thouvenot E; Honnorat J; Rogemond V; De Bovis B; Aebischer P; Marin P; Raoul C; Henderson CE; Pettmann B
J Neurosci; 2010 Jan; 30(2):785-96. PubMed ID: 20071543
[TBL] [Abstract][Full Text] [Related]
8. Depressed excitability and ion currents linked to slow exocytotic fusion pore in chromaffin cells of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
Calvo-Gallardo E; de Pascual R; Fernández-Morales JC; Arranz-Tagarro JA; Maroto M; Nanclares C; Gandía L; de Diego AM; Padín JF; García AG
Am J Physiol Cell Physiol; 2015 Jan; 308(1):C1-19. PubMed ID: 25377090
[TBL] [Abstract][Full Text] [Related]
9. The vulnerability of spinal motoneurons and soma size plasticity in a mouse model of amyotrophic lateral sclerosis.
Dukkipati SS; Garrett TL; Elbasiouny SM
J Physiol; 2018 May; 596(9):1723-1745. PubMed ID: 29502344
[TBL] [Abstract][Full Text] [Related]
10. Dysregulation of astrocyte-motoneuron cross-talk in mutant superoxide dismutase 1-related amyotrophic lateral sclerosis.
Ferraiuolo L; Higginbottom A; Heath PR; Barber S; Greenald D; Kirby J; Shaw PJ
Brain; 2011 Sep; 134(Pt 9):2627-41. PubMed ID: 21908873
[TBL] [Abstract][Full Text] [Related]
11. Axotomy-induced target disconnection promotes an additional death mechanism involved in motoneuron degeneration in amyotrophic lateral sclerosis transgenic mice.
Haulcomb MM; Mesnard NA; Batka RJ; Alexander TD; Sanders VM; Jones KJ
J Comp Neurol; 2014 Jul; 522(10):2349-76. PubMed ID: 24424947
[TBL] [Abstract][Full Text] [Related]
12. Time course of preferential motor unit loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis.
Hegedus J; Putman CT; Gordon T
Neurobiol Dis; 2007 Nov; 28(2):154-64. PubMed ID: 17766128
[TBL] [Abstract][Full Text] [Related]
13. Voltage-gated calcium channels are abnormal in cultured spinal motoneurons in the G93A-SOD1 transgenic mouse model of ALS.
Chang Q; Martin LJ
Neurobiol Dis; 2016 Sep; 93():78-95. PubMed ID: 27151771
[TBL] [Abstract][Full Text] [Related]
14. Altered postnatal maturation of electrical properties in spinal motoneurons in a mouse model of amyotrophic lateral sclerosis.
Quinlan KA; Schuster JE; Fu R; Siddique T; Heckman CJ
J Physiol; 2011 May; 589(Pt 9):2245-60. PubMed ID: 21486770
[TBL] [Abstract][Full Text] [Related]
15. Motoneuron subtypes show specificity in glycine receptor channel abnormalities in a transgenic mouse model of amyotrophic lateral sclerosis.
Chang Q; Martin LJ
Channels (Austin); 2011; 5(4):299-303. PubMed ID: 21558795
[TBL] [Abstract][Full Text] [Related]
16. Mutant SOD1-expressing astrocytes release toxic factors that trigger motoneuron death by inducing hyperexcitability.
Fritz E; Izaurieta P; Weiss A; Mir FR; Rojas P; Gonzalez D; Rojas F; Brown RH; Madrid R; van Zundert B
J Neurophysiol; 2013 Jun; 109(11):2803-14. PubMed ID: 23486205
[TBL] [Abstract][Full Text] [Related]
17. Reduction in the motoneuron inhibitory/excitatory synaptic ratio in an early-symptomatic mouse model of amyotrophic lateral sclerosis.
Sunico CR; Domínguez G; García-Verdugo JM; Osta R; Montero F; Moreno-López B
Brain Pathol; 2011 Jan; 21(1):1-15. PubMed ID: 20653686
[TBL] [Abstract][Full Text] [Related]
18. Early signs of motoneuron vulnerability in a disease model system: Characterization of transverse slice cultures of spinal cord isolated from embryonic ALS mice.
Avossa D; Grandolfo M; Mazzarol F; Zatta M; Ballerini L
Neuroscience; 2006; 138(4):1179-94. PubMed ID: 16442737
[TBL] [Abstract][Full Text] [Related]
19. Altered Functionality, Morphology, and Vesicular Glutamate Transporter Expression of Cortical Motor Neurons from a Presymptomatic Mouse Model of Amyotrophic Lateral Sclerosis.
Saba L; Viscomi MT; Caioli S; Pignataro A; Bisicchia E; Pieri M; Molinari M; Ammassari-Teule M; Zona C
Cereb Cortex; 2016 Apr; 26(4):1512-28. PubMed ID: 25596588
[TBL] [Abstract][Full Text] [Related]
20. Wild-type microglia extend survival in PU.1 knockout mice with familial amyotrophic lateral sclerosis.
Beers DR; Henkel JS; Xiao Q; Zhao W; Wang J; Yen AA; Siklos L; McKercher SR; Appel SH
Proc Natl Acad Sci U S A; 2006 Oct; 103(43):16021-6. PubMed ID: 17043238
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
