BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

135 related articles for article (PubMed ID: 2531841)

  • 1. Factor C3f is a spasmogenic fragment released from C3b by factors I and H: the heptadeca-peptide C3f was synthesized and characterized.
    Ganu VS; Müller-Eberhard HJ; Hugli TE
    Mol Immunol; 1989 Oct; 26(10):939-48. PubMed ID: 2531841
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Physiologic inactivation of fluid phase C3b: isolation and structural analysis of C3c, C3d,g (alpha 2D), and C3g.
    Davis AE; Harrison RA; Lachmann PJ
    J Immunol; 1984 Apr; 132(4):1960-6. PubMed ID: 6607952
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Structure of C3f, a small peptide specifically released during inactivation of the third component of complement.
    Harrison RA; Farries TC; Northrop FD; Lachmann PJ; Davis AE
    Complement; 1988; 5(1):27-32. PubMed ID: 3338271
    [TBL] [Abstract][Full Text] [Related]  

  • 4. The active site of human C4a anaphylatoxin.
    Hugli TE; Kawahara MS; Unson CG; Molinar-Rode R; Erickson BW
    Mol Immunol; 1983 Jun; 20(6):637-45. PubMed ID: 6603573
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Preparation of complement fragments C3b and C3a from purified rat complement component C3 by activated cobra venom factor.
    Usami M; Ohno Y
    J Pharmacol Toxicol Methods; 2005; 52(2):260-3. PubMed ID: 16125624
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Synthetic peptides with the biological activities and specificity of human C3a anaphylatoxin.
    Hugli TE; Erickson BW
    Proc Natl Acad Sci U S A; 1977 May; 74(5):1826-30. PubMed ID: 266705
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Functional consequences of the genetic polymorphism of the third component of complement.
    Welch TR; Beischel L; Kleesattel A
    J Pediatr; 1990 May; 116(5):S92-7. PubMed ID: 2139466
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Regulatory system of guinea-pig complement C3b: tests for compatibility of guinea-pig factors H and I with human factors.
    Seya T; Okada M; Hazeki K; Nagasawa S
    Mol Immunol; 1991; 28(4-5):375-82. PubMed ID: 1829503
    [TBL] [Abstract][Full Text] [Related]  

  • 9. The active site of C3a anaphylatoxin.
    Caporale LH; Tippett PS; Erickson BW; Hugli TE
    J Biol Chem; 1980 Nov; 255(22):10758-63. PubMed ID: 6968751
    [TBL] [Abstract][Full Text] [Related]  

  • 10. A revised mechanism for the activation of complement C3 to C3b: a molecular explanation of a disease-associated polymorphism.
    Rodriguez E; Nan R; Li K; Gor J; Perkins SJ
    J Biol Chem; 2015 Jan; 290(4):2334-50. PubMed ID: 25488663
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Inhibition of factor I by diisopropylfluorophosphate. Evidence of conformational changes in factor I induced by C3b and additional studies on the specificity of factor I.
    Ekdahl KN; Nilsson UR; Nilsson B
    J Immunol; 1990 Jun; 144(11):4269-74. PubMed ID: 2140392
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Amino acid sequence of guinea pig C3a anaphylatoxin.
    Gerard NP; Lively MO; Gerard C
    Protein Seq Data Anal; 1988; 1(6):473-8. PubMed ID: 3064079
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Characterization of C3 in C3 glomerulopathy.
    Sethi S; Vrana JA; Fervenza FC; Theis JD; Sethi A; Kurtin PJ; Zhang Y; Smith RJH
    Nephrol Dial Transplant; 2017 Mar; 32(3):459-465. PubMed ID: 27507892
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Identification of classical anaphylatoxin as the des-Arg form of the C5a molecule: evidence of a modulator role for the oligosaccharide unit in human des-Arg74-C5a.
    Gerard C; Hugli TE
    Proc Natl Acad Sci U S A; 1981 Mar; 78(3):1833-7. PubMed ID: 6940191
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Investigation of mechanism-based inhibitors of complement targeting the activated thioester of human C3.
    Sahu A; Pangburn MK
    Biochem Pharmacol; 1996 Mar; 51(6):797-804. PubMed ID: 8602875
    [TBL] [Abstract][Full Text] [Related]  

  • 16. A synthetic nonapeptide corresponding to the NH2-terminal sequence of C3d-K causes leukocytosis in rabbits.
    Hoeprich PD; Dahinden CA; Lachmann PJ; Davis AE; Hugli TE
    J Biol Chem; 1985 Mar; 260(5):2597-600. PubMed ID: 3871772
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Interaction of desialated guinea pig erythrocytes with the classical and alternative pathways of guinea pig complement in vivo and in vitro.
    Brown EJ; Joiner KA; Frank MM
    J Clin Invest; 1983 Jun; 71(6):1710-9. PubMed ID: 6863540
    [TBL] [Abstract][Full Text] [Related]  

  • 18. A discontinuous factor H binding site in the third component of complement as delineated by synthetic peptides.
    Lambris JD; Avila D; Becherer JD; Müller-Eberhard HJ
    J Biol Chem; 1988 Aug; 263(24):12147-50. PubMed ID: 2969896
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Comparison of the binding of C3S and C3F to complement receptors types 1, 2, and 3.
    Bartók I; Walport MJ
    J Immunol; 1995 May; 154(10):5367-75. PubMed ID: 7730637
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Paroxysmal nocturnal hemoglobinuria: deficiency in factor H-like functions of the abnormal erythrocytes.
    Pangburn MK; Schreiber RD; Trombold JS; Müller-Eberhard HJ
    J Exp Med; 1983 Jun; 157(6):1971-80. PubMed ID: 6222136
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 7.