297 related articles for article (PubMed ID: 25362149)
1. Roles and post-translational regulation of cardiac class IIa histone deacetylase isoforms.
Weeks KL; Avkiran M
J Physiol; 2015 Apr; 593(8):1785-97. PubMed ID: 25362149
[TBL] [Abstract][Full Text] [Related]
2. Regulation of acetylation of histone deacetylase 2 by p300/CBP-associated factor/histone deacetylase 5 in the development of cardiac hypertrophy.
Eom GH; Nam YS; Oh JG; Choe N; Min HK; Yoo EK; Kang G; Nguyen VH; Min JJ; Kim JK; Lee IK; Bassel-Duby R; Olson EN; Park WJ; Kook H
Circ Res; 2014 Mar; 114(7):1133-43. PubMed ID: 24526703
[TBL] [Abstract][Full Text] [Related]
3. Converse role of class I and class IIa HDACs in the progression of atrial fibrillation.
Zhang D; Hu X; Li J; Hoogstra-Berends F; Zhuang Q; Esteban MA; de Groot N; Henning RH; Brundel BJJM
J Mol Cell Cardiol; 2018 Dec; 125():39-49. PubMed ID: 30321539
[TBL] [Abstract][Full Text] [Related]
4. The role of post-translational modifications in cardiac hypertrophy.
Yan K; Wang K; Li P
J Cell Mol Med; 2019 Jun; 23(6):3795-3807. PubMed ID: 30950211
[TBL] [Abstract][Full Text] [Related]
5. Post-translational modifications talk and crosstalk to class IIa histone deacetylases.
Guttzeit S; Backs J
J Mol Cell Cardiol; 2022 Jan; 162():53-61. PubMed ID: 34416247
[TBL] [Abstract][Full Text] [Related]
6. The transcriptional coactivator CAMTA2 stimulates cardiac growth by opposing class II histone deacetylases.
Song K; Backs J; McAnally J; Qi X; Gerard RD; Richardson JA; Hill JA; Bassel-Duby R; Olson EN
Cell; 2006 May; 125(3):453-66. PubMed ID: 16678093
[TBL] [Abstract][Full Text] [Related]
7. Class II histone deacetylases act as signal-responsive repressors of cardiac hypertrophy.
Zhang CL; McKinsey TA; Chang S; Antos CL; Hill JA; Olson EN
Cell; 2002 Aug; 110(4):479-88. PubMed ID: 12202037
[TBL] [Abstract][Full Text] [Related]
8. Neurohormonal regulation of cardiac histone deacetylase 5 nuclear localization by phosphorylation-dependent and phosphorylation-independent mechanisms.
Haworth RS; Stathopoulou K; Candasamy AJ; Avkiran M
Circ Res; 2012 Jun; 110(12):1585-95. PubMed ID: 22581927
[TBL] [Abstract][Full Text] [Related]
9. Nuclear calcium/calmodulin-dependent protein kinase IIdelta preferentially transmits signals to histone deacetylase 4 in cardiac cells.
Little GH; Bai Y; Williams T; Poizat C
J Biol Chem; 2007 Mar; 282(10):7219-31. PubMed ID: 17179159
[TBL] [Abstract][Full Text] [Related]
10. Chromatin modifications remodel cardiac gene expression.
Mathiyalagan P; Keating ST; Du XJ; El-Osta A
Cardiovasc Res; 2014 Jul; 103(1):7-16. PubMed ID: 24812277
[TBL] [Abstract][Full Text] [Related]
11. Roles and targets of class I and IIa histone deacetylases in cardiac hypertrophy.
Kee HJ; Kook H
J Biomed Biotechnol; 2011; 2011():928326. PubMed ID: 21151616
[TBL] [Abstract][Full Text] [Related]
12. Posttranslational modifications of histone deacetylases: implications for cardiovascular diseases.
Eom GH; Kook H
Pharmacol Ther; 2014 Aug; 143(2):168-80. PubMed ID: 24594235
[TBL] [Abstract][Full Text] [Related]
13. The black sheep of class IIa: HDAC7 SIKens the heart.
Travers JG; Hu T; McKinsey TA
J Clin Invest; 2020 Jun; 130(6):2811-2813. PubMed ID: 32364532
[TBL] [Abstract][Full Text] [Related]
14. CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy.
Backs J; Song K; Bezprozvannaya S; Chang S; Olson EN
J Clin Invest; 2006 Jul; 116(7):1853-64. PubMed ID: 16767219
[TBL] [Abstract][Full Text] [Related]
15. Signal-dependent repression of DUSP5 by class I HDACs controls nuclear ERK activity and cardiomyocyte hypertrophy.
Ferguson BS; Harrison BC; Jeong MY; Reid BG; Wempe MF; Wagner FF; Holson EB; McKinsey TA
Proc Natl Acad Sci U S A; 2013 Jun; 110(24):9806-11. PubMed ID: 23720316
[TBL] [Abstract][Full Text] [Related]
16. CaMKII exacerbates heart failure progression by activating class I HDACs.
Zhang M; Yang X; Zimmerman RJ; Wang Q; Ross MA; Granger JM; Luczak ED; Bedja D; Jiang H; Feng N
J Mol Cell Cardiol; 2020 Dec; 149():73-81. PubMed ID: 32971072
[TBL] [Abstract][Full Text] [Related]
17. The CRM1 nuclear export receptor controls pathological cardiac gene expression.
Harrison BC; Roberts CR; Hood DB; Sweeney M; Gould JM; Bush EW; McKinsey TA
Mol Cell Biol; 2004 Dec; 24(24):10636-49. PubMed ID: 15572669
[TBL] [Abstract][Full Text] [Related]
18. Class II HDACs mediate CaMK-dependent signaling to NRSF in ventricular myocytes.
Nakagawa Y; Kuwahara K; Harada M; Takahashi N; Yasuno S; Adachi Y; Kawakami R; Nakanishi M; Tanimoto K; Usami S; Kinoshita H; Saito Y; Nakao K
J Mol Cell Cardiol; 2006 Dec; 41(6):1010-22. PubMed ID: 17011572
[TBL] [Abstract][Full Text] [Related]
19. A novel kinase inhibitor establishes a predominant role for protein kinase D as a cardiac class IIa histone deacetylase kinase.
Monovich L; Vega RB; Meredith E; Miranda K; Rao C; Capparelli M; Lemon DD; Phan D; Koch KA; Chapo JA; Hood DB; McKinsey TA
FEBS Lett; 2010 Feb; 584(3):631-7. PubMed ID: 20018189
[TBL] [Abstract][Full Text] [Related]
20. Suppression of HDAC nuclear export and cardiomyocyte hypertrophy by novel irreversible inhibitors of CRM1.
Monovich L; Koch KA; Burgis R; Osimboni E; Mann T; Wall D; Gao J; Feng Y; Vega RB; Turner BA; Hood DB; Law A; Papst PJ; Koditek D; Chapo JA; Reid BG; Melvin LS; Pagratis NC; McKinsey TA
Biochim Biophys Acta; 2009 May; 1789(5):422-31. PubMed ID: 19414071
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
