361 related articles for article (PubMed ID: 25384392)
1. Tau deletion impairs intracellular β-amyloid-42 clearance and leads to more extracellular plaque deposition in gene transfer models.
Lonskaya I; Hebron M; Chen W; Schachter J; Moussa C
Mol Neurodegener; 2014 Nov; 9():46. PubMed ID: 25384392
[TBL] [Abstract][Full Text] [Related]
2. Diminished parkin solubility and co-localization with intraneuronal amyloid-β are associated with autophagic defects in Alzheimer's disease.
Lonskaya I; Shekoyan AR; Hebron ML; Desforges N; Algarzae NK; Moussa CE
J Alzheimers Dis; 2013; 33(1):231-47. PubMed ID: 22954671
[TBL] [Abstract][Full Text] [Related]
3. NDP52 associates with phosphorylated tau in brains of an Alzheimer disease mouse model.
Kim S; Lee D; Song JC; Cho SJ; Yun SM; Koh YH; Song J; Johnson GV; Jo C
Biochem Biophys Res Commun; 2014 Nov; 454(1):196-201. PubMed ID: 25450380
[TBL] [Abstract][Full Text] [Related]
4. Genetically augmenting tau levels does not modulate the onset or progression of Abeta pathology in transgenic mice.
Oddo S; Caccamo A; Cheng D; Jouleh B; Torp R; LaFerla FM
J Neurochem; 2007 Aug; 102(4):1053-63. PubMed ID: 17472708
[TBL] [Abstract][Full Text] [Related]
5. Effects of the superoxide dismutase/catalase mimetic EUK-207 in a mouse model of Alzheimer's disease: protection against and interruption of progression of amyloid and tau pathology and cognitive decline.
Clausen A; Xu X; Bi X; Baudry M
J Alzheimers Dis; 2012; 30(1):183-208. PubMed ID: 22406441
[TBL] [Abstract][Full Text] [Related]
6. Aβ secretion and plaque formation depend on autophagy.
Nilsson P; Loganathan K; Sekiguchi M; Matsuba Y; Hui K; Tsubuki S; Tanaka M; Iwata N; Saito T; Saido TC
Cell Rep; 2013 Oct; 5(1):61-9. PubMed ID: 24095740
[TBL] [Abstract][Full Text] [Related]
7. Pyroglutamation of amyloid-βx-42 (Aβx-42) followed by Aβ1-40 deposition underlies plaque polymorphism in progressing Alzheimer's disease pathology.
Michno W; Nyström S; Wehrli P; Lashley T; Brinkmalm G; Guerard L; Syvänen S; Sehlin D; Kaya I; Brinet D; Nilsson KPR; Hammarström P; Blennow K; Zetterberg H; Hanrieder J
J Biol Chem; 2019 Apr; 294(17):6719-6732. PubMed ID: 30814252
[TBL] [Abstract][Full Text] [Related]
8. Tau pathogenesis is promoted by Aβ1-42 but not Aβ1-40.
Hu X; Li X; Zhao M; Gottesdiener A; Luo W; Paul S
Mol Neurodegener; 2014 Nov; 9():52. PubMed ID: 25417177
[TBL] [Abstract][Full Text] [Related]
9. Beta-amyloid 1-42 monomers, but not oligomers, produce PHF-like conformation of Tau protein.
Manassero G; Guglielmotto M; Zamfir R; Borghi R; Colombo L; Salmona M; Perry G; Odetti P; Arancio O; Tamagno E; Tabaton M
Aging Cell; 2016 Oct; 15(5):914-23. PubMed ID: 27406053
[TBL] [Abstract][Full Text] [Related]
10. Inhibitor Kappa B Kinase β, Modulated by DJ-1/p-VHL, Reduces Phosphorylated Tau (p-Tau) Accumulation via Autophagy in Alzheimer's Disease Model.
Chen WP; Zhang G; Cheng ZJ; Gu XH; Li M; Liu X
Neuroscience; 2021 Jan; 452():1-12. PubMed ID: 33069779
[TBL] [Abstract][Full Text] [Related]
11. Selective cytotoxicity of intracellular amyloid beta peptide1-42 through p53 and Bax in cultured primary human neurons.
Zhang Y; McLaughlin R; Goodyer C; LeBlanc A
J Cell Biol; 2002 Feb; 156(3):519-29. PubMed ID: 11815632
[TBL] [Abstract][Full Text] [Related]
12. Forebrain microglia from wild-type but not adult 5xFAD mice prevent amyloid-β plaque formation in organotypic hippocampal slice cultures.
Hellwig S; Masuch A; Nestel S; Katzmarski N; Meyer-Luehmann M; Biber K
Sci Rep; 2015 Sep; 5():14624. PubMed ID: 26416689
[TBL] [Abstract][Full Text] [Related]
13. Parkin null cortical neuronal/glial cultures are resistant to amyloid-β1-42 toxicity: a role for autophagy?
Solano RM; Casarejos MJ; Gómez A; Perucho J; de Yébenes JG; Mena MA
J Alzheimers Dis; 2012; 32(1):57-76. PubMed ID: 22785397
[TBL] [Abstract][Full Text] [Related]
14. Amyloid precursor protein cytoplasmic domain with phospho-Thr668 accumulates in Alzheimer's disease and its transgenic models: a role to mediate interaction of Abeta and tau.
Shin RW; Ogino K; Shimabuku A; Taki T; Nakashima H; Ishihara T; Kitamoto T
Acta Neuropathol; 2007 Jun; 113(6):627-36. PubMed ID: 17431643
[TBL] [Abstract][Full Text] [Related]
15. Fractalkine signaling and Tau hyper-phosphorylation are associated with autophagic alterations in lentiviral Tau and Aβ1-42 gene transfer models.
Hebron ML; Algarzae NK; Lonskaya I; Moussa C
Exp Neurol; 2014 Jan; 251():127-38. PubMed ID: 23333589
[TBL] [Abstract][Full Text] [Related]
16. Neuropep-1 ameliorates learning and memory deficits in an Alzheimer's disease mouse model, increases brain-derived neurotrophic factor expression in the brain, and causes reduction of amyloid beta plaques.
Shin MK; Kim HG; Baek SH; Jung WR; Park DI; Park JS; Jo DG; Kim KL
Neurobiol Aging; 2014 May; 35(5):990-1001. PubMed ID: 24268884
[TBL] [Abstract][Full Text] [Related]
17. Accumulation of amyloid-like Aβ1-42 in AEL (autophagy-endosomal-lysosomal) vesicles: potential implications for plaque biogenesis.
Ling D; Magallanes M; Salvaterra PM
ASN Neuro; 2014 Mar; 6(2):. PubMed ID: 24521233
[TBL] [Abstract][Full Text] [Related]
18. Normal cognition in transgenic BRI2-Aβ mice.
Kim J; Chakrabarty P; Hanna A; March A; Dickson DW; Borchelt DR; Golde T; Janus C
Mol Neurodegener; 2013 May; 8():15. PubMed ID: 23663320
[TBL] [Abstract][Full Text] [Related]
19. Tau reduction prevents Aβ-induced axonal transport deficits by blocking activation of GSK3β.
Vossel KA; Xu JC; Fomenko V; Miyamoto T; Suberbielle E; Knox JA; Ho K; Kim DH; Yu GQ; Mucke L
J Cell Biol; 2015 May; 209(3):419-33. PubMed ID: 25963821
[TBL] [Abstract][Full Text] [Related]
20. Abeta immunotherapy leads to clearance of early, but not late, hyperphosphorylated tau aggregates via the proteasome.
Oddo S; Billings L; Kesslak JP; Cribbs DH; LaFerla FM
Neuron; 2004 Aug; 43(3):321-32. PubMed ID: 15294141
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
