497 related articles for article (PubMed ID: 25429069)
1. MHC-mismatched mixed chimerism mediates thymic deletion of cross-reactive autoreactive T cells and prevents insulitis in nonobese diabetic mice.
Racine JJ; Zhang M; Wang M; Morales W; Shen C; Zeng D
J Immunol; 2015 Jan; 194(1):407-17. PubMed ID: 25429069
[TBL] [Abstract][Full Text] [Related]
2. MHC-mismatched mixed chimerism restores peripheral tolerance of noncross-reactive autoreactive T cells in NOD mice.
Zhang M; Racine JJ; Lin Q; Liu Y; Tang S; Qin Q; Qi T; Riggs AD; Zeng D
Proc Natl Acad Sci U S A; 2018 Mar; 115(10):E2329-E2337. PubMed ID: 29463744
[TBL] [Abstract][Full Text] [Related]
3. MHC-mismatched chimerism is required for induction of transplantation tolerance in autoimmune nonobese diabetic recipients.
Wang M; Racine J; Zhang M; Wu T; Deng R; Johnston H; Shen C; Siswanto K; Zeng D
J Immunol; 2014 Aug; 193(4):2005-15. PubMed ID: 25000982
[TBL] [Abstract][Full Text] [Related]
4. Induction of mixed chimerism with MHC-mismatched but not matched bone marrow transplants results in thymic deletion of host-type autoreactive T-cells in NOD mice.
Racine J; Wang M; Zhang C; Lin CL; Liu H; Todorov I; Atkinson M; Zeng D
Diabetes; 2011 Feb; 60(2):555-64. PubMed ID: 21270266
[TBL] [Abstract][Full Text] [Related]
5. Transplantation of MHC-mismatched mouse embryonic stem cell-derived thymic epithelial progenitors and MHC-matched bone marrow prevents autoimmune diabetes.
Su M; Lin Y; He Z; Lai L
Stem Cell Res Ther; 2019 Aug; 10(1):239. PubMed ID: 31387620
[TBL] [Abstract][Full Text] [Related]
6. Autoantigen-independent deletion of diabetogenic CD4+ thymocytes by protective MHC class II molecules.
Schmidt D; Amrani A; Verdaguer J; Bou S; Santamaria P
J Immunol; 1999 Apr; 162(8):4627-36. PubMed ID: 10202002
[TBL] [Abstract][Full Text] [Related]
7. Induction of mixed chimerism depletes pre-existing and de novo-developed autoreactive B cells in autoimmune NOD mice.
Racine JJ; Wang M; Zhang M; Zeng D
Diabetes; 2014 Jun; 63(6):2051-62. PubMed ID: 24458357
[TBL] [Abstract][Full Text] [Related]
8. Bone marrow-derived cells are essential for intrathymic deletion of self-reactive T cells in both the host- and donor-derived thymocytes of fully allogeneic bone marrow chimeras.
Yoshikai Y; Ogimoto M; Matsuzaki G; Nomoto K
J Immunol; 1990 Jul; 145(2):505-9. PubMed ID: 2114441
[TBL] [Abstract][Full Text] [Related]
9. MHC-mismatched mixed chimerism augments thymic regulatory T-cell production and prevents relapse of EAE in mice.
Wu L; Li N; Zhang M; Xue SL; Cassady K; Lin Q; Riggs AD; Zeng D
Proc Natl Acad Sci U S A; 2015 Dec; 112(52):15994-9. PubMed ID: 26647186
[TBL] [Abstract][Full Text] [Related]
10. Reconstitution ratio is critical for alloreactive T cell deletion and skin graft survival in mixed bone marrow chimeras.
Taniguchi H; Abe M; Shirai T; Fukao K; Nakauchi H
J Immunol; 1995 Dec; 155(12):5631-6. PubMed ID: 7499847
[TBL] [Abstract][Full Text] [Related]
11. Thymic positive selection and peripheral activation of islet antigen-specific T cells: separation of two diabetogenic steps by an I-A(g7) class II MHC beta-chain mutant.
Kanagawa O; Vaupel BA; Xu G; Unanue ER; Katz JD
J Immunol; 1998 Nov; 161(9):4489-92. PubMed ID: 9794372
[TBL] [Abstract][Full Text] [Related]
12. Development of diabetogenic T cells from NOD/Lt marrow is blocked when an allo-H-2 haplotype is expressed on cells of hemopoietic origin, but not on thymic epithelium.
Serreze DV; Leiter EH
J Immunol; 1991 Aug; 147(4):1222-9. PubMed ID: 1869820
[TBL] [Abstract][Full Text] [Related]
13. Selection of aberrant class II restricted CD8+ T cells in NOD mice expressing a glutamic acid decarboxylase (GAD)65-specific T cell receptor transgene.
Ranheim EA; Tarbell KV; Krogsgaard M; Mallet-Designe V; Teyton L; McDevitt HO; Weissman IL
Autoimmunity; 2004 Dec; 37(8):555-67. PubMed ID: 15763918
[TBL] [Abstract][Full Text] [Related]
14. MHC-mismatched islet allografts are vulnerable to autoimmune recognition in vivo.
Kupfer TM; Crawford ML; Pham K; Gill RG
J Immunol; 2005 Aug; 175(4):2309-16. PubMed ID: 16081800
[TBL] [Abstract][Full Text] [Related]
15. B6.g7 mice reconstituted with BDC2·5 non-obese diabetic (BDC2·5NOD) stem cells do not develop autoimmune diabetes.
Rajasekaran N; Wang N; Hang Y; Macaubas C; Rinderknecht C; Beilhack GF; Shizuru JA; Mellins ED
Clin Exp Immunol; 2013 Oct; 174(1):27-37. PubMed ID: 23795893
[TBL] [Abstract][Full Text] [Related]
16. A mechanism for the major histocompatibility complex-linked resistance to autoimmunity.
Schmidt D; Verdaguer J; Averill N; Santamaria P
J Exp Med; 1997 Oct; 186(7):1059-75. PubMed ID: 9314555
[TBL] [Abstract][Full Text] [Related]
17. Through regulation of TCR expression levels, an Idd7 region gene(s) interactively contributes to the impaired thymic deletion of autoreactive diabetogenic CD8+ T cells in nonobese diabetic mice.
Serreze DV; Choisy-Rossi CM; Grier AE; Holl TM; Chapman HD; Gahagan JR; Osborne MA; Zhang W; King BL; Brown A; Roopenian D; Marron MP
J Immunol; 2008 Mar; 180(5):3250-9. PubMed ID: 18292549
[TBL] [Abstract][Full Text] [Related]
18. E expression is needed on both bone marrow derived cells and thymic epithelium to increase IL-4 production and achieve protection in NOD bone marrow chimeras.
Brenden N; Rietz C; Böhme J
Cytokine; 1999 Oct; 11(10):766-72. PubMed ID: 10525315
[TBL] [Abstract][Full Text] [Related]
19. Differential MHC expression requirements for positive selection of separate TCR Vb families.
Brenden N; Böhme J
Immunogenetics; 1999 Jan; 49(1):1-6. PubMed ID: 9811963
[TBL] [Abstract][Full Text] [Related]
20. Defective thymic T cell activation by concanavalin A and anti-CD3 in autoimmune nonobese diabetic mice. Evidence for thymic T cell anergy that correlates with the onset of insulitis.
Zipris D; Lazarus AH; Crow AR; Hadzija M; Delovitch TL
J Immunol; 1991 Jun; 146(11):3763-71. PubMed ID: 1827815
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
