118 related articles for article (PubMed ID: 2562156)
1. Development of autoimmune insulitis is prevented in E alpha d but not in A beta k NOD transgenic mice.
Uehira M; Uno M; Kürner T; Kikutani H; Mori K; Inomoto T; Uede T; Miyazaki J; Nishimoto H; Kishimoto T
Int Immunol; 1989; 1(2):209-13. PubMed ID: 2562156
[TBL] [Abstract][Full Text] [Related]
2. Prevention of autoimmune insulitis by expression of I-E molecules in NOD mice.
Nishimoto H; Kikutani H; Yamamura K; Kishimoto T
Nature; 1987 Jul 30-Aug 5; 328(6129):432-4. PubMed ID: 3302721
[TBL] [Abstract][Full Text] [Related]
3. Prevention of insulin-dependent diabetes mellitus in non-obese diabetic mice by transgenes encoding modified I-A beta-chain or normal I-E alpha-chain.
Lund T; O'Reilly L; Hutchings P; Kanagawa O; Simpson E; Gravely R; Chandler P; Dyson J; Picard JK; Edwards A
Nature; 1990 Jun; 345(6277):727-9. PubMed ID: 2163026
[TBL] [Abstract][Full Text] [Related]
4. Immunogenetic analysis of beta-cell autoimmunity in NOD mice. Relationship of insulitis to T-lymphocyte-receptor beta nod and A beta nod genes.
Winter WE; Shimpo K; Obata M; Yamada K; Luchetta R
Diabetes; 1990 Aug; 39(8):975-82. PubMed ID: 1973674
[TBL] [Abstract][Full Text] [Related]
5. Prevention of diabetes in non-obese diabetic I-Ak transgenic mice.
Slattery RM; Kjer-Nielsen L; Allison J; Charlton B; Mandel TE; Miller JF
Nature; 1990 Jun; 345(6277):724-6. PubMed ID: 1972779
[TBL] [Abstract][Full Text] [Related]
6. A single recessive non-MHC diabetogenic gene determines the development of insulitis in the presence of an MHC-linked diabetogenic gene in NOD mice.
Hattori M; Fukuda M; Ichikawa T; Baumgartl HJ; Katoh H; Makino S
J Autoimmun; 1990 Feb; 3(1):1-10. PubMed ID: 2184821
[TBL] [Abstract][Full Text] [Related]
7. Genetic control of diabetes and insulitis in the nonobese diabetic (NOD) mouse.
Wicker LS; Miller BJ; Coker LZ; McNally SE; Scott S; Mullen Y; Appel MC
J Exp Med; 1987 Jun; 165(6):1639-54. PubMed ID: 3585250
[TBL] [Abstract][Full Text] [Related]
8. Expression of genetically determined diabetes and insulitis in the nonobese diabetic (NOD) mouse at the level of bone marrow-derived cells. Transfer of diabetes and insulitis to nondiabetic (NOD X B10) F1 mice with bone marrow cells from NOD mice.
Wicker LS; Miller BJ; Chai A; Terada M; Mullen Y
J Exp Med; 1988 Jun; 167(6):1801-10. PubMed ID: 3290380
[TBL] [Abstract][Full Text] [Related]
9. Direct evidence for the contribution of the unique I-ANOD to the development of insulitis in non-obese diabetic mice.
Miyazaki T; Uno M; Uehira M; Kikutani H; Kishimoto T; Kimoto M; Nishimoto H; Miyazaki J; Yamamura K
Nature; 1990 Jun; 345(6277):722-4. PubMed ID: 2113614
[TBL] [Abstract][Full Text] [Related]
10. Genetic analysis for insulitis in NOD mice.
Makino S; Muraoka Y; Kishimoto Y; Hayashi Y
Jikken Dobutsu; 1985 Oct; 34(4):425-31. PubMed ID: 3910444
[TBL] [Abstract][Full Text] [Related]
11. Complete prevention of diabetes in transgenic NOD mice expressing I-E molecules.
Uno M; Miyazaki T; Uehira M; Nishimoto H; Kimoto M; Miyazaki J; Yamamura K
Immunol Lett; 1992 Jan; 31(1):47-52. PubMed ID: 1548033
[TBL] [Abstract][Full Text] [Related]
12. Genetic control of diabetes and insulitis in the nonobese diabetic mouse. Pedigree analysis of a diabetic H-2nod/b heterozygote.
Wicker LS; Miller BJ; Fischer PA; Pressey A; Peterson LB
J Immunol; 1989 Feb; 142(3):781-4. PubMed ID: 2643662
[TBL] [Abstract][Full Text] [Related]
13. An explanation for the protective effect of the MHC class II I-E molecule in murine diabetes.
Reich EP; Sherwin RS; Kanagawa O; Janeway CA
Nature; 1989 Sep; 341(6240):326-8. PubMed ID: 2507922
[TBL] [Abstract][Full Text] [Related]
14. The NOD mouse as a model of type I diabetes.
Tochino Y
Crit Rev Immunol; 1987; 8(1):49-81. PubMed ID: 2961513
[TBL] [Abstract][Full Text] [Related]
15. Genetic analysis of diabetes in the nonobese diabetic mouse. I. MHC and T cell receptor beta gene expression.
Livingstone A; Edwards CT; Shizuru JA; Fathman CG
J Immunol; 1991 Jan; 146(2):529-34. PubMed ID: 1824775
[TBL] [Abstract][Full Text] [Related]
16. Transgenic tumor necrosis factor (TNF)-alpha production in pancreatic islets leads to insulitis, not diabetes. Distinct patterns of inflammation in TNF-alpha and TNF-beta transgenic mice.
Picarella DE; Kratz A; Li CB; Ruddle NH; Flavell RA
J Immunol; 1993 May; 150(9):4136-50. PubMed ID: 7682590
[TBL] [Abstract][Full Text] [Related]
17. IL-10 is necessary and sufficient for autoimmune diabetes in conjunction with NOD MHC homozygosity.
Lee MS; Mueller R; Wicker LS; Peterson LB; Sarvetnick N
J Exp Med; 1996 Jun; 183(6):2663-8. PubMed ID: 8676087
[TBL] [Abstract][Full Text] [Related]
18. Role of the first external domain of I-A beta chain in immune responses and diabetes in non-obese diabetic (NOD) mice.
Singh B; Dillon T; Fraga E; Lauzon J
J Autoimmun; 1990 Oct; 3(5):507-21. PubMed ID: 1701300
[TBL] [Abstract][Full Text] [Related]
19. I-E+ nonobese diabetic mice develop insulitis and diabetes.
Podolin PL; Pressey A; DeLarato NH; Fischer PA; Peterson LB; Wicker LS
J Exp Med; 1993 Sep; 178(3):793-803. PubMed ID: 8350054
[TBL] [Abstract][Full Text] [Related]
20. High and low diabetes incidence nonobese diabetic (NOD) mice: origins and characterisation.
Baxter AG; Koulmanda M; Mandel TE
Autoimmunity; 1991; 9(1):61-7. PubMed ID: 1669848
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]