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2. Mitochondrial DNA somatic mutations (point mutations and large deletions) and mitochondrial DNA variants in human thyroid pathology: a study with emphasis on Hürthle cell tumors. Máximo V; Soares P; Lima J; Cameselle-Teijeiro J; Sobrinho-Simões M Am J Pathol; 2002 May; 160(5):1857-65. PubMed ID: 12000737 [TBL] [Abstract][Full Text] [Related]
3. The RET/PTC oncogene is frequently activated in oncocytic thyroid tumors (Hurthle cell adenomas and carcinomas), but not in oncocytic hyperplastic lesions. Chiappetta G; Toti P; Cetta F; Giuliano A; Pentimalli F; Amendola I; Lazzi S; Monaco M; Mazzuchelli L; Tosi P; Santoro M; Fusco A J Clin Endocrinol Metab; 2002 Jan; 87(1):364-9. PubMed ID: 11788677 [TBL] [Abstract][Full Text] [Related]
4. Frequent chromosomal DNA unbalance in thyroid oncocytic (Hürthle cell) neoplasms detected by comparative genomic hybridization. Tallini G; Hsueh A; Liu S; Garcia-Rostan G; Speicher MR; Ward DC Lab Invest; 1999 May; 79(5):547-55. PubMed ID: 10334566 [TBL] [Abstract][Full Text] [Related]
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6. Genomic dissection of Hurthle cell carcinoma reveals a unique class of thyroid malignancy. Ganly I; Ricarte Filho J; Eng S; Ghossein R; Morris LG; Liang Y; Socci N; Kannan K; Mo Q; Fagin JA; Chan TA J Clin Endocrinol Metab; 2013 May; 98(5):E962-72. PubMed ID: 23543667 [TBL] [Abstract][Full Text] [Related]
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