These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
324 related articles for article (PubMed ID: 25666897)
21. Cysteine residues in Cu,Zn-superoxide dismutase are essential to toxicity in Caenorhabditis elegans model of amyotrophic lateral sclerosis. Ogawa M; Shidara H; Oka K; Kurosawa M; Nukina N; Furukawa Y Biochem Biophys Res Commun; 2015 Aug; 463(4):1196-202. PubMed ID: 26086102 [TBL] [Abstract][Full Text] [Related]
22. Lipid molecules induce the cytotoxic aggregation of Cu/Zn superoxide dismutase with structurally disordered regions. Choi I; Yang YI; Song HD; Lee JS; Kang T; Sung JJ; Yi J Biochim Biophys Acta; 2011 Jan; 1812(1):41-8. PubMed ID: 20837142 [TBL] [Abstract][Full Text] [Related]
23. Superoxide dismutase 1 mutants related to amyotrophic lateral sclerosis induce endoplasmic stress in neuro2a cells. Oh YK; Shin KS; Yuan J; Kang SJ J Neurochem; 2008 Feb; 104(4):993-1005. PubMed ID: 18233996 [TBL] [Abstract][Full Text] [Related]
24. Histological evidence of protein aggregation in mutant SOD1 transgenic mice and in amyotrophic lateral sclerosis neural tissues. Watanabe M; Dykes-Hoberg M; Culotta VC; Price DL; Wong PC; Rothstein JD Neurobiol Dis; 2001 Dec; 8(6):933-41. PubMed ID: 11741389 [TBL] [Abstract][Full Text] [Related]
25. Amyloid-like filaments and water-filled nanotubes formed by SOD1 mutant proteins linked to familial ALS. Elam JS; Taylor AB; Strange R; Antonyuk S; Doucette PA; Rodriguez JA; Hasnain SS; Hayward LJ; Valentine JS; Yeates TO; Hart PJ Nat Struct Biol; 2003 Jun; 10(6):461-7. PubMed ID: 12754496 [TBL] [Abstract][Full Text] [Related]
26. Solid-state NMR studies of metal-free SOD1 fibrillar structures. Banci L; Blaževitš O; Cantini F; Danielsson J; Lang L; Luchinat C; Mao J; Oliveberg M; Ravera E J Biol Inorg Chem; 2014 Jun; 19(4-5):659-66. PubMed ID: 24719206 [TBL] [Abstract][Full Text] [Related]
27. Cu/Zn-superoxide dismutase forms fibrillar hydrogels in a pH-dependent manner via a water-rich extended intermediate state. Fujiwara N; Wagatsuma M; Oba N; Yoshihara D; Tokuda E; Sakiyama H; Eguchi H; Ichihashi M; Furukawa Y; Inoue T; Suzuki K PLoS One; 2018; 13(10):e0205090. PubMed ID: 30289953 [TBL] [Abstract][Full Text] [Related]
28. A double point mutation of SOD1 targeting net charge promotes aggregation under destabilizing conditions: Correlation of charge distribution and ALS-provoking mutation. Mavadat E; Seyedalipour B; Hosseinkhani S Biochim Biophys Acta Gen Subj; 2023 May; 1867(5):130325. PubMed ID: 36791828 [TBL] [Abstract][Full Text] [Related]
29. Metal-free superoxide dismutase forms soluble oligomers under physiological conditions: a possible general mechanism for familial ALS. Banci L; Bertini I; Durazo A; Girotto S; Gralla EB; Martinelli M; Valentine JS; Vieru M; Whitelegge JP Proc Natl Acad Sci U S A; 2007 Jul; 104(27):11263-7. PubMed ID: 17592131 [TBL] [Abstract][Full Text] [Related]
30. E6-AP association promotes SOD1 aggresomes degradation and suppresses toxicity. Mishra A; Maheshwari M; Chhangani D; Fujimori-Tonou N; Endo F; Joshi AP; Jana NR; Yamanaka K Neurobiol Aging; 2013 Apr; 34(4):1310.e11-23. PubMed ID: 23040663 [TBL] [Abstract][Full Text] [Related]