209 related articles for article (PubMed ID: 2569164)
1. Repression of transcription mediated at a thyroid hormone response element by the v-erb-A oncogene product.
Sap J; Muñoz A; Schmitt J; Stunnenberg H; Vennström B
Nature; 1989 Jul; 340(6230):242-4. PubMed ID: 2569164
[TBL] [Abstract][Full Text] [Related]
2. Requirement for the C-terminal domain of the v-erbA oncogene protein for biological function and transcriptional repression.
Forrest D; Muñoz A; Raynoschek C; Vennström B; Beug H
Oncogene; 1990 Mar; 5(3):309-16. PubMed ID: 1969136
[TBL] [Abstract][Full Text] [Related]
3. The oncoprotein P75gag-v-erbA represses thyroid hormone induced transcription only via response elements containing palindromic half-sites.
Wahlström GM; Harbers M; Vennström B
Oncogene; 1996 Aug; 13(4):843-52. PubMed ID: 8761306
[TBL] [Abstract][Full Text] [Related]
4. A c-erb-A binding site in rat growth hormone gene mediates trans-activation by thyroid hormone.
Glass CK; Franco R; Weinberger C; Albert VR; Evans RM; Rosenfeld MG
Nature; 1987 Oct 22-28; 329(6141):738-41. PubMed ID: 3313046
[TBL] [Abstract][Full Text] [Related]
5. V-erbA requires auxiliary proteins for dominant negative activity.
Hermann T; Hoffmann B; Piedrafita FJ; Zhang XK; Pfahl M
Oncogene; 1993 Jan; 8(1):55-65. PubMed ID: 8093812
[TBL] [Abstract][Full Text] [Related]
6. Retinoic acid and thyroid hormone induce gene expression through a common responsive element.
Umesono K; Giguere V; Glass CK; Rosenfeld MG; Evans RM
Nature; 1988 Nov; 336(6196):262-5. PubMed ID: 2848197
[TBL] [Abstract][Full Text] [Related]
7. c-ErbA, but not v-ErbA, competes with a putative erythroid repressor for binding to the carbonic anhydrase II promoter.
Rascle A; Ghysdael J; Samarut J
Oncogene; 1994 Oct; 9(10):2853-67. PubMed ID: 7916146
[TBL] [Abstract][Full Text] [Related]
8. The c-erb-A protein is a high-affinity receptor for thyroid hormone.
Sap J; Muñoz A; Damm K; Goldberg Y; Ghysdael J; Leutz A; Beug H; Vennström B
Nature; 1986 Dec 18-31; 324(6098):635-40. PubMed ID: 2879242
[TBL] [Abstract][Full Text] [Related]
9. An activator protein-1 (AP-1) response element on pro alpha1(l) collagen gene is necessary for thyroid hormone-induced inhibition of promoter activity in cardiac fibroblasts.
Lee HW; Klein LE; Raser J; Eghbali-Webb M
J Mol Cell Cardiol; 1998 Nov; 30(11):2495-506. PubMed ID: 9925384
[TBL] [Abstract][Full Text] [Related]
10. Thyroid hormone receptor alpha isoforms generated by alternative splicing differentially activate myosin HC gene transcription.
Izumo S; Mahdavi V
Nature; 1988 Aug; 334(6182):539-42. PubMed ID: 2841611
[TBL] [Abstract][Full Text] [Related]
11. Functional interaction between the two zinc finger domains of the v-erb A oncoprotein.
Hall BL; Bonde BG; Judelson C; Privalsky ML
Cell Growth Differ; 1992 Apr; 3(4):207-16. PubMed ID: 1355356
[TBL] [Abstract][Full Text] [Related]
12. Mechanism of transformation by v-ErbA: substitution for steroid hormone receptor function in self renewal induction.
Bauer A; Ulrich E; Andersson M; Beug H; von Lindern M
Oncogene; 1997 Aug; 15(6):701-15. PubMed ID: 9264411
[TBL] [Abstract][Full Text] [Related]
13. The thyroid hormone receptor functions as a ligand-operated developmental switch between proliferation and differentiation of erythroid progenitors.
Bauer A; Mikulits W; Lagger G; Stengl G; Brosch G; Beug H
EMBO J; 1998 Aug; 17(15):4291-303. PubMed ID: 9687498
[TBL] [Abstract][Full Text] [Related]
14. Biological effects of the v-erbA oncogene in transformation of avian erythroid cells.
Vennström B; Beug R; Damm K; Engel D; Gehring U; Graf T; Muñoz A; Sap J; Zenke M
Horm Metab Res Suppl; 1987; 17():14-9. PubMed ID: 2895057
[No Abstract] [Full Text] [Related]
15. The v-erb A oncogene causes repression of erythrocyte-specific genes and an immature, aberrant differentiation phenotype in normal erythroid progenitors.
Schroeder C; Raynoschek C; Fuhrmann U; Damm K; Vennström B; Beug H
Oncogene; 1990 Oct; 5(10):1445-53. PubMed ID: 1979159
[TBL] [Abstract][Full Text] [Related]
16. Functional domains of the v-erbA protein necessary for oncogenesis are required for transcriptional activation in Saccharomyces cerevisiae.
Smit-McBride Z; Privalsky ML
Oncogene; 1993 Jun; 8(6):1465-75. PubMed ID: 8099219
[TBL] [Abstract][Full Text] [Related]
17. v-erbA oncogene activation entails the loss of hormone-dependent regulator activity of c-erbA.
Zenke M; Muñoz A; Sap J; Vennström B; Beug H
Cell; 1990 Jun; 61(6):1035-49. PubMed ID: 1972036
[TBL] [Abstract][Full Text] [Related]
18. Leukemic transformation by the v-ErbA oncoprotein entails constitutive binding to and repression of an erythroid enhancer in vivo.
Ciana P; Braliou GG; Demay FG; von Lindern M; Barettino D; Beug H; Stunnenberg HG
EMBO J; 1998 Dec; 17(24):7382-94. PubMed ID: 9857194
[TBL] [Abstract][Full Text] [Related]
19. Identification of a thyroid hormone response element in the mouse myogenin gene: characterization of the thyroid hormone and retinoid X receptor heterodimeric binding site.
Downes M; Griggs R; Atkins A; Olson EN; Muscat GE
Cell Growth Differ; 1993 Nov; 4(11):901-9. PubMed ID: 8297796
[TBL] [Abstract][Full Text] [Related]
20. Mapping of functional domains within the v-erb A oncogene protein: the remnants of the hormone binding domain play multiple, vital roles in protein action.
Boucher P; Privalsky ML
Oncogene; 1990 Sep; 5(9):1303-11. PubMed ID: 2170895
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
