BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

403 related articles for article (PubMed ID: 25746422)

  • 1. Phloretin ameliorates arsenic trioxide induced mitochondrial dysfunction in H9c2 cardiomyoblasts mediated via alterations in membrane permeability and ETC complexes.
    Vineetha VP; Soumya RS; Raghu KG
    Eur J Pharmacol; 2015 May; 754():162-72. PubMed ID: 25746422
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Polyphenol-rich apple (Malus domestica L.) peel extract attenuates arsenic trioxide induced cardiotoxicity in H9c2 cells via its antioxidant activity.
    Vineetha VP; Girija S; Soumya RS; Raghu KG
    Food Funct; 2014 Mar; 5(3):502-11. PubMed ID: 24441683
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Dithiothreitol abrogates the effect of arsenic trioxide on normal rat liver mitochondria and human hepatocellular carcinoma cells.
    Paul MK; Kumar R; Mukhopadhyay AK
    Toxicol Appl Pharmacol; 2008 Jan; 226(2):140-52. PubMed ID: 18022205
    [TBL] [Abstract][Full Text] [Related]  

  • 4. L-ascorbic acid and α-tocopherol attenuate arsenic trioxide-induced toxicity in H9c2 cardiomyocytes by the activation of Nrf2 and Bcl2 transcription factors.
    Vineetha RC; Binu P; Arathi P; Nair RH
    Toxicol Mech Methods; 2018 Jun; 28(5):353-360. PubMed ID: 29297235
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Arsenic trioxide toxicity in H9c2 myoblasts--damage to cell organelles and possible amelioration with Boerhavia diffusa.
    Vineetha VP; Prathapan A; Soumya RS; Raghu KG
    Cardiovasc Toxicol; 2013 Jun; 13(2):123-37. PubMed ID: 23161055
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Increased apoptotic efficacy of lonidamine plus arsenic trioxide combination in human leukemia cells. Reactive oxygen species generation and defensive protein kinase (MEK/ERK, Akt/mTOR) modulation.
    Calviño E; Estañ MC; Simón GP; Sancho P; Boyano-Adánez Mdel C; de Blas E; Bréard J; Aller P
    Biochem Pharmacol; 2011 Dec; 82(11):1619-29. PubMed ID: 21889928
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Phloretin Alleviates Arsenic Trioxide-Induced Apoptosis of H9c2 Cardiomyoblasts via Downregulation in Ca
    Vadavanath Prabhakaran V; Kozhiparambil Gopalan R
    Cardiovasc Toxicol; 2021 Aug; 21(8):642-654. PubMed ID: 34037972
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Mitochondrial disruption occurs downstream from β-adrenergic overactivation by isoproterenol in differentiated, but not undifferentiated H9c2 cardiomyoblasts: differential activation of stress and survival pathways.
    Branco AF; Sampaio SF; Wieckowski MR; Sardão VA; Oliveira PJ
    Int J Biochem Cell Biol; 2013 Nov; 45(11):2379-91. PubMed ID: 23958426
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Arsenic trioxide-induced apoptosis in H9c2 cardiomyocytes: implications in cardiotoxicity.
    Zhao X; Feng T; Chen H; Shan H; Zhang Y; Lu Y; Yang B
    Basic Clin Pharmacol Toxicol; 2008 May; 102(5):419-25. PubMed ID: 18346055
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Arsenic trioxide-induced cytotoxicity in small cell lung cancer via altered redox homeostasis and mitochondrial integrity.
    Zheng CY; Lam SK; Li YY; Ho JC
    Int J Oncol; 2015 Mar; 46(3):1067-78. PubMed ID: 25572414
    [TBL] [Abstract][Full Text] [Related]  

  • 11. ROS-mediated endoplasmic reticulum stress and mitochondrial dysfunction underlie apoptosis induced by resveratrol and arsenic trioxide in A549 cells.
    Gu S; Chen C; Jiang X; Zhang Z
    Chem Biol Interact; 2016 Feb; 245():100-9. PubMed ID: 26772155
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Ellagic acid mitigates arsenic-trioxide-induced mitochondrial dysfunction and cytotoxicity in SH-SY5Y cells.
    Firdaus F; Zafeer MF; Waseem M; Anis E; Hossain MM; Afzal M
    J Biochem Mol Toxicol; 2018 Feb; 32(2):. PubMed ID: 29314450
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Omega-3 Fatty Acid Protects Against Arsenic Trioxide-Induced Cardiotoxicity In Vitro and In Vivo.
    Varghese MV; Abhilash M; Paul MV; Alex M; Nair RH
    Cardiovasc Toxicol; 2017 Apr; 17(2):109-119. PubMed ID: 26886836
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Characterization of cytotoxicity induced by arsenic trioxide (a potent anti-APL drug) in rat cardiac myocytes.
    Raghu KG; Cherian OL
    J Trace Elem Med Biol; 2009; 23(1):61-8. PubMed ID: 19203718
    [TBL] [Abstract][Full Text] [Related]  

  • 15. JWA is required for arsenic trioxide induced apoptosis in HeLa and MCF-7 cells via reactive oxygen species and mitochondria linked signal pathway.
    Zhou J; Ye J; Zhao X; Li A; Zhou J
    Toxicol Appl Pharmacol; 2008 Jul; 230(1):33-40. PubMed ID: 18387645
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Activation of the ubiquitin-proteasome system against arsenic trioxide cardiotoxicity involves ubiquitin ligase Parkin for mitochondrial homeostasis.
    Watanabe M; Funakoshi T; Unuma K; Aki T; Uemura K
    Toxicology; 2014 Aug; 322():43-50. PubMed ID: 24801902
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Effect and mechanism of Sorbus pohuashanensis (Hante) Hedl. flavonoids protect against arsenic trioxide-induced cardiotoxicity.
    Yu X; Wang Z; Shu Z; Li Z; Ning Y; Yun K; Bai H; Liu R; Liu W
    Biomed Pharmacother; 2017 Apr; 88():1-10. PubMed ID: 28092839
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Epoxyeicosatrienoic acids attenuate reactive oxygen species level, mitochondrial dysfunction, caspase activation, and apoptosis in carcinoma cells treated with arsenic trioxide.
    Liu L; Chen C; Gong W; Li Y; Edin ML; Zeldin DC; Wang DW
    J Pharmacol Exp Ther; 2011 Nov; 339(2):451-63. PubMed ID: 21846841
    [TBL] [Abstract][Full Text] [Related]  

  • 19. High glucose-induced Ca2+ overload and oxidative stress contribute to apoptosis of cardiac cells through mitochondrial dependent and independent pathways.
    Kumar S; Kain V; Sitasawad SL
    Biochim Biophys Acta; 2012 Jul; 1820(7):907-20. PubMed ID: 22402252
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Genistein selectively potentiates arsenic trioxide-induced apoptosis in human leukemia cells via reactive oxygen species generation and activation of reactive oxygen species-inducible protein kinases (p38-MAPK, AMPK).
    Sánchez Y; Amrán D; Fernández C; de Blas E; Aller P
    Int J Cancer; 2008 Sep; 123(5):1205-14. PubMed ID: 18546268
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 21.