These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

299 related articles for article (PubMed ID: 25883218)

  • 1. Deriving a cardiac ageing signature to reveal MMP-9-dependent inflammatory signalling in senescence.
    Ma Y; Chiao YA; Clark R; Flynn ER; Yabluchanskiy A; Ghasemi O; Zouein F; Lindsey ML; Jin YF
    Cardiovasc Res; 2015 Jun; 106(3):421-31. PubMed ID: 25883218
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Matrix metalloproteinase-9 deletion attenuates myocardial fibrosis and diastolic dysfunction in ageing mice.
    Chiao YA; Ramirez TA; Zamilpa R; Okoronkwo SM; Dai Q; Zhang J; Jin YF; Lindsey ML
    Cardiovasc Res; 2012 Dec; 96(3):444-55. PubMed ID: 22918978
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Cardiomyocyte-specific Bmal1 deletion in mice triggers diastolic dysfunction, extracellular matrix response, and impaired resolution of inflammation.
    Ingle KA; Kain V; Goel M; Prabhu SD; Young ME; Halade GV
    Am J Physiol Heart Circ Physiol; 2015 Dec; 309(11):H1827-36. PubMed ID: 26432841
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Protein tyrosine phosphatase 1B inactivation limits aging-associated heart failure in mice.
    Besnier M; Coquerel D; Favre J; Dumesnil A; Guerrot D; Remy-Jouet I; Mulder P; Djerada Z; Tamion F; Richard V; Ouvrard-Pascaud A
    Am J Physiol Heart Circ Physiol; 2018 Jun; 314(6):H1279-H1288. PubMed ID: 29569957
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.
    Meschiari CA; Jung M; Iyer RP; Yabluchanskiy A; Toba H; Garrett MR; Lindsey ML
    Am J Physiol Heart Circ Physiol; 2018 Feb; 314(2):H224-H235. PubMed ID: 29030341
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Cardiac aging is initiated by matrix metalloproteinase-9-mediated endothelial dysfunction.
    Yabluchanskiy A; Ma Y; Chiao YA; Lopez EF; Voorhees AP; Toba H; Hall ME; Han HC; Lindsey ML; Jin YF
    Am J Physiol Heart Circ Physiol; 2014 May; 306(10):H1398-407. PubMed ID: 24658018
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Myocardial Infarction Superimposed on Aging: MMP-9 Deletion Promotes M2 Macrophage Polarization.
    Yabluchanskiy A; Ma Y; DeLeon-Pennell KY; Altara R; Halade GV; Voorhees AP; Nguyen NT; Jin YF; Winniford MD; Hall ME; Han HC; Lindsey ML
    J Gerontol A Biol Sci Med Sci; 2016 Apr; 71(4):475-83. PubMed ID: 25878031
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Matrix metalloproteinase-28 deletion exacerbates cardiac dysfunction and rupture after myocardial infarction in mice by inhibiting M2 macrophage activation.
    Ma Y; Halade GV; Zhang J; Ramirez TA; Levin D; Voorhees A; Jin YF; Han HC; Manicone AM; Lindsey ML
    Circ Res; 2013 Feb; 112(4):675-88. PubMed ID: 23261783
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Local production of tenascin-C acts as a trigger for monocyte/macrophage recruitment that provokes cardiac dysfunction.
    Abbadi D; Laroumanie F; Bizou M; Pozzo J; Daviaud D; Delage C; Calise D; Gaits-Iacovoni F; Dutaur M; Tortosa F; Renaud-Gabardos E; Douin-Echinard V; Prats AC; Roncalli J; Parini A; Pizzinat N
    Cardiovasc Res; 2018 Jan; 114(1):123-137. PubMed ID: 29136112
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Transgenic overexpression of macrophage matrix metalloproteinase-9 exacerbates age-related cardiac hypertrophy, vessel rarefaction, inflammation, and fibrosis.
    Toba H; Cannon PL; Yabluchanskiy A; Iyer RP; D'Armiento J; Lindsey ML
    Am J Physiol Heart Circ Physiol; 2017 Mar; 312(3):H375-H383. PubMed ID: 28011588
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Interferon-γ ablation exacerbates myocardial hypertrophy in diastolic heart failure.
    Garcia AG; Wilson RM; Heo J; Murthy NR; Baid S; Ouchi N; Sam F
    Am J Physiol Heart Circ Physiol; 2012 Sep; 303(5):H587-96. PubMed ID: 22730392
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Cardiomyocyte dimethylarginine dimethylaminohydrolase-1 (DDAH1) plays an important role in attenuating ventricular hypertrophy and dysfunction.
    Xu X; Zhang P; Kwak D; Fassett J; Yue W; Atzler D; Hu X; Liu X; Wang H; Lu Z; Guo H; Schwedhelm E; Böger RH; Chen P; Chen Y
    Basic Res Cardiol; 2017 Aug; 112(5):55. PubMed ID: 28819685
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Dissecting the role of myeloid and mesenchymal fibroblasts in age-dependent cardiac fibrosis.
    Trial J; Heredia CP; Taffet GE; Entman ML; Cieslik KA
    Basic Res Cardiol; 2017 Jul; 112(4):34. PubMed ID: 28478479
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Tenascin-C promotes chronic pressure overload-induced cardiac dysfunction, hypertrophy and myocardial fibrosis.
    Podesser BK; Kreibich M; Dzilic E; Santer D; Förster L; Trojanek S; Abraham D; Krššák M; Klein KU; Tretter EV; Kaun C; Wojta J; Kapeller B; Gonçalves IF; Trescher K; Kiss A
    J Hypertens; 2018 Apr; 36(4):847-856. PubMed ID: 29283973
    [TBL] [Abstract][Full Text] [Related]  

  • 15. NADPH oxidase-4 promotes eccentric cardiac hypertrophy in response to volume overload.
    Schnelle M; Sawyer I; Anilkumar N; Mohamed BA; Richards DA; Toischer K; Zhang M; Catibog N; Sawyer G; Mongue-Din H; Schröder K; Hasenfuss G; Shah AM
    Cardiovasc Res; 2021 Jan; 117(1):178-187. PubMed ID: 31821410
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution.
    Iyer RP; de Castro Brás LE; Patterson NL; Bhowmick M; Flynn ER; Asher M; Cannon PL; Deleon-Pennell KY; Fields GB; Lindsey ML
    J Mol Cell Cardiol; 2016 Nov; 100():109-117. PubMed ID: 27746126
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Secreted protein acidic and rich in cysteine facilitates age-related cardiac inflammation and macrophage M1 polarization.
    Toba H; de Castro Brás LE; Baicu CF; Zile MR; Lindsey ML; Bradshaw AD
    Am J Physiol Cell Physiol; 2015 Jun; 308(12):C972-82. PubMed ID: 25877699
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Neuraminidase-1 promotes heart failure after ischemia/reperfusion injury by affecting cardiomyocytes and invading monocytes/macrophages.
    Heimerl M; Sieve I; Ricke-Hoch M; Erschow S; Battmer K; Scherr M; Hilfiker-Kleiner D
    Basic Res Cardiol; 2020 Sep; 115(6):62. PubMed ID: 32975669
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Tumor necrosis factor-alpha mediates cardiac remodeling and ventricular dysfunction after pressure overload state.
    Sun M; Chen M; Dawood F; Zurawska U; Li JY; Parker T; Kassiri Z; Kirshenbaum LA; Arnold M; Khokha R; Liu PP
    Circulation; 2007 Mar; 115(11):1398-407. PubMed ID: 17353445
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Deletion of obscurin immunoglobulin domains Ig58/59 leads to age-dependent cardiac remodeling and arrhythmia.
    Grogan A; Coleman A; Joca H; Granzier H; Russel MW; Ward CW; Kontrogianni-Konstantopoulos A
    Basic Res Cardiol; 2020 Sep; 115(6):60. PubMed ID: 32910221
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 15.