BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

500 related articles for article (PubMed ID: 25906181)

  • 1. ATG16L1: A multifunctional susceptibility factor in Crohn disease.
    Salem M; Ammitzboell M; Nys K; Seidelin JB; Nielsen OH
    Autophagy; 2015 Apr; 11(4):585-94. PubMed ID: 25906181
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Classification of genetic profiles of Crohn's disease: a focus on the ATG16L1 gene.
    Grant SF; Baldassano RN; Hakonarson H
    Expert Rev Mol Diagn; 2008 Mar; 8(2):199-207. PubMed ID: 18366306
    [TBL] [Abstract][Full Text] [Related]  

  • 3. New insights into the interplay between autophagy, gut microbiota and inflammatory responses in IBD.
    Larabi A; Barnich N; Nguyen HTT
    Autophagy; 2020 Jan; 16(1):38-51. PubMed ID: 31286804
    [TBL] [Abstract][Full Text] [Related]  

  • 4. The Crohn Disease-associated ATG16L1
    Gao P; Liu H; Huang H; Sun Y; Jia B; Hou B; Zhou X; Strober W; Zhang F
    Autophagy; 2022 Nov; 18(11):2561-2575. PubMed ID: 35220902
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Genetic association and functional role of Crohn disease risk alleles involved in microbial sensing, autophagy, and endoplasmic reticulum (ER) stress.
    Hoefkens E; Nys K; John JM; Van Steen K; Arijs I; Van der Goten J; Van Assche G; Agostinis P; Rutgeerts P; Vermeire S; Cleynen I
    Autophagy; 2013 Dec; 9(12):2046-55. PubMed ID: 24247223
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Genomic ATG16L1 risk allele-restricted Paneth cell ER stress in quiescent Crohn's disease.
    Deuring JJ; Fuhler GM; Konstantinov SR; Peppelenbosch MP; Kuipers EJ; de Haar C; van der Woude CJ
    Gut; 2014 Jul; 63(7):1081-91. PubMed ID: 23964099
    [TBL] [Abstract][Full Text] [Related]  

  • 7. A nonsynonymous SNP in ATG16L1 predisposes to ileal Crohn's disease and is independent of CARD15 and IBD5.
    Prescott NJ; Fisher SA; Franke A; Hampe J; Onnie CM; Soars D; Bagnall R; Mirza MM; Sanderson J; Forbes A; Mansfield JC; Lewis CM; Schreiber S; Mathew CG
    Gastroenterology; 2007 May; 132(5):1665-71. PubMed ID: 17484864
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Unconventional autophagy mediated by the WD40 domain of ATG16L1 is derailed by the T300A Crohn disease risk polymorphism.
    Serramito-Gómez I; Boada-Romero E; Pimentel-Muiños FX
    Autophagy; 2016 Nov; 12(11):2254-2255. PubMed ID: 27541200
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Crohn's disease: NOD2, autophagy and ER stress converge.
    Fritz T; Niederreiter L; Adolph T; Blumberg RS; Kaser A
    Gut; 2011 Nov; 60(11):1580-8. PubMed ID: 21252204
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Impaired autophagy of an intracellular pathogen induced by a Crohn's disease associated ATG16L1 variant.
    Kuballa P; Huett A; Rioux JD; Daly MJ; Xavier RJ
    PLoS One; 2008; 3(10):e3391. PubMed ID: 18852889
    [TBL] [Abstract][Full Text] [Related]  

  • 11. ATG16L1 and IL23R are associated with inflammatory bowel diseases but not with celiac disease in the Netherlands.
    Weersma RK; Zhernakova A; Nolte IM; Lefebvre C; Rioux JD; Mulder F; van Dullemen HM; Kleibeuker JH; Wijmenga C; Dijkstra G
    Am J Gastroenterol; 2008 Mar; 103(3):621-7. PubMed ID: 18047540
    [TBL] [Abstract][Full Text] [Related]  

  • 12. A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1.
    Hampe J; Franke A; Rosenstiel P; Till A; Teuber M; Huse K; Albrecht M; Mayr G; De La Vega FM; Briggs J; Günther S; Prescott NJ; Onnie CM; Häsler R; Sipos B; Fölsch UR; Lengauer T; Platzer M; Mathew CG; Krawczak M; Schreiber S
    Nat Genet; 2007 Feb; 39(2):207-11. PubMed ID: 17200669
    [TBL] [Abstract][Full Text] [Related]  

  • 13. ATG16L1 WD40 domain-dependent IL10R (interleukin 10 receptor) signaling is insensitive to the T300A Crohn disease risk polymorphism.
    Serramito-Gómez I; Terraza-Silvestre E; Fernández-Cabrera Á; Villamuera R; Pimentel-Muiños FX
    Autophagy; 2022 Dec; 18(12):3023-3030. PubMed ID: 35311452
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Is there a role for Crohn's disease-associated autophagy genes ATG16L1 and IRGM in formation of granulomas?
    Wolfkamp SC; Te Velde AA; Weersma RK; Ponsioen CY; Stokkers PC
    Eur J Gastroenterol Hepatol; 2010 Aug; 22(8):933-7. PubMed ID: 20395867
    [TBL] [Abstract][Full Text] [Related]  

  • 15. ATG16L1 and NOD2 interact in an autophagy-dependent antibacterial pathway implicated in Crohn's disease pathogenesis.
    Homer CR; Richmond AL; Rebert NA; Achkar JP; McDonald C
    Gastroenterology; 2010 Nov; 139(5):1630-41, 1641.e1-2. PubMed ID: 20637199
    [TBL] [Abstract][Full Text] [Related]  

  • 16. ATG16L1 rs2241880/T300A increases susceptibility to perianal Crohn's disease: An updated meta-analysis on inflammatory bowel disease risk and clinical outcomes.
    Simovic I; Hilmi I; Ng RT; Chew KS; Wong SY; Lee WS; Riordan S; Castaño-Rodríguez N
    United European Gastroenterol J; 2024 Feb; 12(1):103-121. PubMed ID: 37837511
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Autophagy 16-like 1 rs2241880 G allele is associated with Crohn's disease in German children.
    Lacher M; Schroepf S; Ballauff A; Lohse P; von Schweinitz D; Kappler R; Koletzko S
    Acta Paediatr; 2009 Nov; 98(11):1835-40. PubMed ID: 19659808
    [TBL] [Abstract][Full Text] [Related]  

  • 18. The T300A Crohn's disease risk polymorphism impairs function of the WD40 domain of ATG16L1.
    Boada-Romero E; Serramito-Gómez I; Sacristán MP; Boone DL; Xavier RJ; Pimentel-Muiños FX
    Nat Commun; 2016 Jun; 7():11821. PubMed ID: 27273576
    [TBL] [Abstract][Full Text] [Related]  

  • 19. A Crohn's disease variant in Atg16l1 enhances its degradation by caspase 3.
    Murthy A; Li Y; Peng I; Reichelt M; Katakam AK; Noubade R; Roose-Girma M; DeVoss J; Diehl L; Graham RR; van Lookeren Campagne M
    Nature; 2014 Feb; 506(7489):456-62. PubMed ID: 24553140
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Crohn's disease-associated ATG16L1 polymorphism modulates pro-inflammatory cytokine responses selectively upon activation of NOD2.
    Plantinga TS; Crisan TO; Oosting M; van de Veerdonk FL; de Jong DJ; Philpott DJ; van der Meer JW; Girardin SE; Joosten LA; Netea MG
    Gut; 2011 Sep; 60(9):1229-35. PubMed ID: 21406388
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 25.