BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

269 related articles for article (PubMed ID: 25987743)

  • 1. Siglec-G Deficiency Leads to Autoimmunity in Aging C57BL/6 Mice.
    Müller J; Lunz B; Schwab I; Acs A; Nimmerjahn F; Daniel C; Nitschke L
    J Immunol; 2015 Jul; 195(1):51-60. PubMed ID: 25987743
    [TBL] [Abstract][Full Text] [Related]  

  • 2. CD22 x Siglec-G double-deficient mice have massively increased B1 cell numbers and develop systemic autoimmunity.
    Jellusova J; Wellmann U; Amann K; Winkler TH; Nitschke L
    J Immunol; 2010 Apr; 184(7):3618-27. PubMed ID: 20200274
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Siglec-G deficiency leads to more severe collagen-induced arthritis and earlier onset of lupus-like symptoms in MRL/lpr mice.
    Bökers S; Urbat A; Daniel C; Amann K; Smith KG; Espéli M; Nitschke L
    J Immunol; 2014 Apr; 192(7):2994-3002. PubMed ID: 24600033
    [TBL] [Abstract][Full Text] [Related]  

  • 4. CD22 and Siglec-G regulate inhibition of B-cell signaling by sialic acid ligand binding and control B-cell tolerance.
    Nitschke L
    Glycobiology; 2014 Sep; 24(9):807-17. PubMed ID: 25002414
    [TBL] [Abstract][Full Text] [Related]  

  • 5. The ligand-binding domain of Siglec-G is crucial for its selective inhibitory function on B1 cells.
    Hutzler S; Özgör L; Naito-Matsui Y; Kläsener K; Winkler TH; Reth M; Nitschke L
    J Immunol; 2014 Jun; 192(11):5406-14. PubMed ID: 24790146
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Sialic Acid Ligand Binding of CD22 and Siglec-G Determines Distinct B Cell Functions but Is Dispensable for B Cell Tolerance Induction.
    Özgör L; Meyer SJ; Korn M; Terörde K; Nitschke L
    J Immunol; 2018 Oct; 201(7):2107-2116. PubMed ID: 30143587
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Copresentation of antigen and ligands of Siglec-G induces B cell tolerance independent of CD22.
    Pfrengle F; Macauley MS; Kawasaki N; Paulson JC
    J Immunol; 2013 Aug; 191(4):1724-31. PubMed ID: 23836061
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Siglec-G is a B-1 cell inhibitory receptor and also controls B cell tolerance.
    Nitschke L
    Ann N Y Acad Sci; 2015 Dec; 1362():117-21. PubMed ID: 26194636
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Siglec-G is a B1 cell-inhibitory receptor that controls expansion and calcium signaling of the B1 cell population.
    Hoffmann A; Kerr S; Jellusova J; Zhang J; Weisel F; Wellmann U; Winkler TH; Kneitz B; Crocker PR; Nitschke L
    Nat Immunol; 2007 Jul; 8(7):695-704. PubMed ID: 17572677
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Distinct and synergistic roles of FcγRIIB deficiency and 129 strain-derived SLAM family proteins in the development of spontaneous germinal centers and autoimmunity.
    Soni C; Domeier PP; Wong EB; Shwetank ; Khan TN; Elias MJ; Schell SL; Lukacher AE; Cooper TK; Rahman ZS
    J Autoimmun; 2015 Sep; 63():31-46. PubMed ID: 26162758
    [TBL] [Abstract][Full Text] [Related]  

  • 11. FcγRIIB regulates T-cell autoreactivity, ANCA production, and neutrophil activation to suppress anti-myeloperoxidase glomerulonephritis.
    Ooi JD; Gan PY; Chen T; Eggenhuizen PJ; Chang J; Alikhan MA; Odobasic D; Holdsworth SR; Kitching AR
    Kidney Int; 2014 Dec; 86(6):1140-9. PubMed ID: 24869670
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Unexpected development of autoimmunity in BAFF-R-mutant MRL-lpr mice.
    Ju ZL; Shi GY; Zuo JX; Zhang JW;
    Immunology; 2007 Feb; 120(2):281-9. PubMed ID: 17073941
    [TBL] [Abstract][Full Text] [Related]  

  • 13. FcgammaRIIB deficiency with Fas mutation is sufficient for the development of systemic autoimmune disease.
    Yajima K; Nakamura A; Sugahara A; Takai T
    Eur J Immunol; 2003 Apr; 33(4):1020-9. PubMed ID: 12672068
    [TBL] [Abstract][Full Text] [Related]  

  • 14. A balance between B cell receptor and inhibitory receptor signaling controls plasma cell differentiation by maintaining optimal Ets1 levels.
    Luo W; Mayeux J; Gutierrez T; Russell L; Getahun A; Müller J; Tedder T; Parnes J; Rickert R; Nitschke L; Cambier J; Satterthwaite AB; Garrett-Sinha LA
    J Immunol; 2014 Jul; 193(2):909-920. PubMed ID: 24929000
    [TBL] [Abstract][Full Text] [Related]  

  • 15. CD22 and Siglec-G: B-cell inhibitory receptors with distinct functions.
    Nitschke L
    Immunol Rev; 2009 Jul; 230(1):128-43. PubMed ID: 19594633
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Antigen targeting to plasmacytoid dendritic cells via Siglec-H inhibits Th cell-dependent autoimmunity.
    Loschko J; Heink S; Hackl D; Dudziak D; Reindl W; Korn T; Krug AB
    J Immunol; 2011 Dec; 187(12):6346-56. PubMed ID: 22079988
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Immune complex-mediated co-ligation of the BCR with FcγRIIB results in homeostatic apoptosis of B cells involving Fas signalling that is defective in the MRL/Lpr model of systemic lupus erythematosus.
    Paunovic V; Carter NA; Thalhamer T; Blair D; Gordon B; Lacey E; Michie AM; Harnett MM
    J Autoimmun; 2012 Dec; 39(4):332-46. PubMed ID: 22647731
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Murine Red Blood Cells Lack Ligands for B Cell Siglecs, Allowing Strong Activation by Erythrocyte Surface Antigens.
    Spiller F; Nycholat CM; Kikuchi C; Paulson JC; Macauley MS
    J Immunol; 2018 Feb; 200(3):949-956. PubMed ID: 29288201
    [TBL] [Abstract][Full Text] [Related]  

  • 19. B Cell Siglecs-News on Signaling and Its Interplay With Ligand Binding.
    Meyer SJ; Linder AT; Brandl C; Nitschke L
    Front Immunol; 2018; 9():2820. PubMed ID: 30559744
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Decoration of T-independent antigen with ligands for CD22 and Siglec-G can suppress immunity and induce B cell tolerance in vivo.
    Duong BH; Tian H; Ota T; Completo G; Han S; Vela JL; Ota M; Kubitz M; Bovin N; Paulson JC; Nemazee D
    J Exp Med; 2010 Jan; 207(1):173-87. PubMed ID: 20038598
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 14.