330 related articles for article (PubMed ID: 26152239)
1. Progression of chromosomal damage induced by etoposide in G2 phase in a DNA-PKcs-deficient context.
Palmitelli M; de Campos-Nebel M; González-Cid M
Chromosome Res; 2015 Dec; 23(4):719-32. PubMed ID: 26152239
[TBL] [Abstract][Full Text] [Related]
2. DNA-PKcs-dependent NHEJ pathway supports the progression of topoisomerase II poison-induced chromosome aberrant cells.
Elguero ME; de Campos-Nebel M; González-Cid M
Environ Mol Mutagen; 2012 Oct; 53(8):608-18. PubMed ID: 22987276
[TBL] [Abstract][Full Text] [Related]
3. Alternative end-joining originates stable chromosome aberrations induced by etoposide during targeted inhibition of DNA-PKcs in ATM-deficient tumor cells.
de Campos Nebel M; Palmitelli M; Pérez Maturo J; González-Cid M
Chromosome Res; 2022 Dec; 30(4):459-476. PubMed ID: 35604590
[TBL] [Abstract][Full Text] [Related]
4. The efficiency of homologous recombination and non-homologous end joining systems in repairing double-strand breaks during cell cycle progression.
Bee L; Fabris S; Cherubini R; Mognato M; Celotti L
PLoS One; 2013; 8(7):e69061. PubMed ID: 23874869
[TBL] [Abstract][Full Text] [Related]
5. Non-homologous end joining is the responsible pathway for the repair of fludarabine-induced DNA double strand breaks in mammalian cells.
de Campos-Nebel M; Larripa I; González-Cid M
Mutat Res; 2008 Nov; 646(1-2):8-16. PubMed ID: 18812179
[TBL] [Abstract][Full Text] [Related]
6. Topoisomerase II-mediated DNA damage is differently repaired during the cell cycle by non-homologous end joining and homologous recombination.
de Campos-Nebel M; Larripa I; González-Cid M
PLoS One; 2010 Sep; 5(9):. PubMed ID: 20824055
[TBL] [Abstract][Full Text] [Related]
7. Chloroethylnitrosourea-induced cell death and genotoxicity: cell cycle dependence and the role of DNA double-strand breaks, HR and NHEJ.
Nikolova T; Hennekes F; Bhatti A; Kaina B
Cell Cycle; 2012 Jul; 11(14):2606-19. PubMed ID: 22751442
[TBL] [Abstract][Full Text] [Related]
8. DNA end resection is needed for the repair of complex lesions in G1-phase human cells.
Averbeck NB; Ringel O; Herrlitz M; Jakob B; Durante M; Taucher-Scholz G
Cell Cycle; 2014; 13(16):2509-16. PubMed ID: 25486192
[TBL] [Abstract][Full Text] [Related]
9. Mechanisms of DNA double strand break repair and chromosome aberration formation.
Iliakis G; Wang H; Perrault AR; Boecker W; Rosidi B; Windhofer F; Wu W; Guan J; Terzoudi G; Pantelias G
Cytogenet Genome Res; 2004; 104(1-4):14-20. PubMed ID: 15162010
[TBL] [Abstract][Full Text] [Related]
10. Wortmannin potentiates the combined effect of etoposide and cisplatin in human glioma cells.
Pastwa E; Poplawski T; Lewandowska U; Somiari SB; Blasiak J; Somiari RI
Int J Biochem Cell Biol; 2014 Aug; 53():423-31. PubMed ID: 24953561
[TBL] [Abstract][Full Text] [Related]
11. Inhibition of ERK activation enhances the repair of double-stranded breaks via non-homologous end joining by increasing DNA-PKcs activation.
Wei F; Yan J; Tang D; Lin X; He L; Xie Y; Tao L; Wang S
Biochim Biophys Acta; 2013 Jan; 1833(1):90-100. PubMed ID: 23098854
[TBL] [Abstract][Full Text] [Related]
12. Analysis of chromatid-break-repair detects a homologous recombination to non-homologous end-joining switch with increasing load of DNA double-strand breaks.
Murmann-Konda T; Soni A; Stuschke M; Iliakis G
Mutat Res Genet Toxicol Environ Mutagen; 2021 Jul; 867():503372. PubMed ID: 34266628
[TBL] [Abstract][Full Text] [Related]
13. p21 promotes error-free replication-coupled DNA double-strand break repair.
Mauro M; Rego MA; Boisvert RA; Esashi F; Cavallo F; Jasin M; Howlett NG
Nucleic Acids Res; 2012 Sep; 40(17):8348-60. PubMed ID: 22735704
[TBL] [Abstract][Full Text] [Related]
14. Inhibiting DNA-PKcs in a non-homologous end-joining pathway in response to DNA double-strand breaks.
Dong J; Zhang T; Ren Y; Wang Z; Ling CC; He F; Li GC; Wang C; Wen B
Oncotarget; 2017 Apr; 8(14):22662-22673. PubMed ID: 28186989
[TBL] [Abstract][Full Text] [Related]
15. DNA-PKcs plays a dominant role in the regulation of H2AX phosphorylation in response to DNA damage and cell cycle progression.
An J; Huang YC; Xu QZ; Zhou LJ; Shang ZF; Huang B; Wang Y; Liu XD; Wu DC; Zhou PK
BMC Mol Biol; 2010 Mar; 11():18. PubMed ID: 20205745
[TBL] [Abstract][Full Text] [Related]
16. DNA repair kinetics in SCID mice Sertoli cells and DNA-PKcs-deficient mouse embryonic fibroblasts.
Ahmed EA; Vélaz E; Rosemann M; Gilbertz KP; Scherthan H
Chromosoma; 2017 Mar; 126(2):287-298. PubMed ID: 27136939
[TBL] [Abstract][Full Text] [Related]
17. Beta human papillomavirus 8 E6 allows colocalization of non-homologous end joining and homologous recombination repair factors.
Hu C; Bugbee T; Dacus D; Palinski R; Wallace N
PLoS Pathog; 2022 Feb; 18(2):e1010275. PubMed ID: 35148356
[TBL] [Abstract][Full Text] [Related]
18. Tyrosyl-DNA-phosphodiesterase I (TDP1) participates in the removal and repair of stabilized-Top2α cleavage complexes in human cells.
Borda MA; Palmitelli M; Verón G; González-Cid M; de Campos Nebel M
Mutat Res; 2015 Nov; 781():37-48. PubMed ID: 26421495
[TBL] [Abstract][Full Text] [Related]
19. Homologous recombination protects mammalian cells from replication-associated DNA double-strand breaks arising in response to methyl methanesulfonate.
Nikolova T; Ensminger M; Löbrich M; Kaina B
DNA Repair (Amst); 2010 Oct; 9(10):1050-63. PubMed ID: 20708982
[TBL] [Abstract][Full Text] [Related]
20. Human polynucleotide kinase participates in repair of DNA double-strand breaks by nonhomologous end joining but not homologous recombination.
Karimi-Busheri F; Rasouli-Nia A; Allalunis-Turner J; Weinfeld M
Cancer Res; 2007 Jul; 67(14):6619-25. PubMed ID: 17638872
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]