281 related articles for article (PubMed ID: 26212128)
1. Mitochondrial impairment and oxidative stress compromise autophagosomal degradation of α-synuclein in oligodendroglial cells.
Pukaß K; Goldbaum O; Richter-Landsberg C
J Neurochem; 2015 Oct; 135(1):194-205. PubMed ID: 26212128
[TBL] [Abstract][Full Text] [Related]
2. Oxidative stress promotes uptake, accumulation, and oligomerization of extracellular α-synuclein in oligodendrocytes.
Pukass K; Richter-Landsberg C
J Mol Neurosci; 2014 Mar; 52(3):339-52. PubMed ID: 24217795
[TBL] [Abstract][Full Text] [Related]
3. Autophagy mediates the clearance of oligodendroglial SNCA/alpha-synuclein and TPPP/p25A in multiple system atrophy models.
Mavroeidi P; Arvanitaki F; Vetsi M; Becker S; Vlachakis D; Jensen PH; Stefanis L; Xilouri M
Autophagy; 2022 Sep; 18(9):2104-2133. PubMed ID: 35000546
[TBL] [Abstract][Full Text] [Related]
4. Alpha-synuclein transfers from neurons to oligodendrocytes.
Reyes JF; Rey NL; Bousset L; Melki R; Brundin P; Angot E
Glia; 2014 Mar; 62(3):387-98. PubMed ID: 24382629
[TBL] [Abstract][Full Text] [Related]
5. Increased neuronal α-synuclein pathology associates with its accumulation in oligodendrocytes in mice modeling α-synucleinopathies.
Kisos H; Pukaß K; Ben-Hur T; Richter-Landsberg C; Sharon R
PLoS One; 2012; 7(10):e46817. PubMed ID: 23077527
[TBL] [Abstract][Full Text] [Related]
6. Inhibition of UCH-L1 in oligodendroglial cells results in microtubule stabilization and prevents α-synuclein aggregate formation by activating the autophagic pathway: implications for multiple system atrophy.
Pukaß K; Richter-Landsberg C
Front Cell Neurosci; 2015; 9():163. PubMed ID: 25999815
[TBL] [Abstract][Full Text] [Related]
7. Pathological Endogenous α-Synuclein Accumulation in Oligodendrocyte Precursor Cells Potentially Induces Inclusions in Multiple System Atrophy.
Kaji S; Maki T; Kinoshita H; Uemura N; Ayaki T; Kawamoto Y; Furuta T; Urushitani M; Hasegawa M; Kinoshita Y; Ono Y; Mao X; Quach TH; Iwai K; Dawson VL; Dawson TM; Takahashi R
Stem Cell Reports; 2018 Feb; 10(2):356-365. PubMed ID: 29337114
[TBL] [Abstract][Full Text] [Related]
8. Endogenous oligodendroglial alpha-synuclein and TPPP/p25α orchestrate alpha-synuclein pathology in experimental multiple system atrophy models.
Mavroeidi P; Arvanitaki F; Karakitsou AK; Vetsi M; Kloukina I; Zweckstetter M; Giller K; Becker S; Sorrentino ZA; Giasson BI; Jensen PH; Stefanis L; Xilouri M
Acta Neuropathol; 2019 Sep; 138(3):415-441. PubMed ID: 31011860
[TBL] [Abstract][Full Text] [Related]
9. Involvement of macroautophagy in multiple system atrophy and protein aggregate formation in oligodendrocytes.
Schwarz L; Goldbaum O; Bergmann M; Probst-Cousin S; Richter-Landsberg C
J Mol Neurosci; 2012 Jun; 47(2):256-66. PubMed ID: 22411133
[TBL] [Abstract][Full Text] [Related]
10. Inhibition of HDAC6 modifies tau inclusion body formation and impairs autophagic clearance.
Leyk J; Goldbaum O; Noack M; Richter-Landsberg C
J Mol Neurosci; 2015 Apr; 55(4):1031-46. PubMed ID: 25434725
[TBL] [Abstract][Full Text] [Related]
11. Prodegenerative IκBα expression in oligodendroglial α-synuclein models of multiple system atrophy.
Kragh CL; Gysbers AM; Rockenstein E; Murphy K; Halliday GM; Masliah E; Jensen PH
Neurobiol Dis; 2014 Mar; 63():171-83. PubMed ID: 24361600
[TBL] [Abstract][Full Text] [Related]
12. Overexpression of α-Synuclein by Oligodendrocytes in Transgenic Mice Does Not Recapitulate the Fibrillar Aggregation Seen in Multiple System Atrophy.
Laferrière F; He X; Zinghirino F; Doudnikoff E; Faggiani E; Meissner WG; Bezard E; De Giorgi F; Ichas F
Cells; 2020 Oct; 9(11):. PubMed ID: 33138150
[TBL] [Abstract][Full Text] [Related]
13. SNCA and TPPP transcripts increase in oligodendroglial cytoplasmic inclusions in multiple system atrophy.
Kon T; Forrest SL; Lee S; Li J; Chasiotis H; Nassir N; Uddin MJ; Lang AE; Kovacs GG
Neurobiol Dis; 2024 Aug; 198():106551. PubMed ID: 38839023
[TBL] [Abstract][Full Text] [Related]
14. Oxidative stress in transgenic mice with oligodendroglial alpha-synuclein overexpression replicates the characteristic neuropathology of multiple system atrophy.
Stefanova N; Reindl M; Neumann M; Haass C; Poewe W; Kahle PJ; Wenning GK
Am J Pathol; 2005 Mar; 166(3):869-76. PubMed ID: 15743798
[TBL] [Abstract][Full Text] [Related]
15. Limited effects of dysfunctional macroautophagy on the accumulation of extracellularly derived α-synuclein in oligodendroglia: implications for MSA pathogenesis.
Fellner L; Buchinger E; Brueck D; Irschick R; Wenning GK; Stefanova N
BMC Neurosci; 2018 May; 19(1):32. PubMed ID: 29783943
[TBL] [Abstract][Full Text] [Related]
16. Oligodendroglial α-synucleinopathy-driven neuroinflammation in multiple system atrophy.
Hoffmann A; Ettle B; Battis K; Reiprich S; Schlachetzki JCM; Masliah E; Wegner M; Kuhlmann T; Riemenschneider MJ; Winkler J
Brain Pathol; 2019 May; 29(3):380-396. PubMed ID: 30444295
[TBL] [Abstract][Full Text] [Related]
17. Membrane lipid modification by docosahexaenoic acid (DHA) promotes the formation of α-synuclein inclusion bodies immunopositive for SUMO-1 in oligodendroglial cells after oxidative stress.
Riedel M; Goldbaum O; Wille M; Richter-Landsberg C
J Mol Neurosci; 2011 Mar; 43(3):290-302. PubMed ID: 20725866
[TBL] [Abstract][Full Text] [Related]
18. Alpha-synuclein overexpression in oligodendrocytic cells results in impaired adhesion to fibronectin and cell death.
Tsuboi K; Grzesiak JJ; Bouvet M; Hashimoto M; Masliah E; Shults CW
Mol Cell Neurosci; 2005 Jun; 29(2):259-68. PubMed ID: 15911350
[TBL] [Abstract][Full Text] [Related]
19. Identification of a highly neurotoxic α-synuclein species inducing mitochondrial damage and mitophagy in Parkinson's disease.
Grassi D; Howard S; Zhou M; Diaz-Perez N; Urban NT; Guerrero-Given D; Kamasawa N; Volpicelli-Daley LA; LoGrasso P; Lasmézas CI
Proc Natl Acad Sci U S A; 2018 Mar; 115(11):E2634-E2643. PubMed ID: 29487216
[TBL] [Abstract][Full Text] [Related]
20. Neuropathology of multiple system atrophy: new thoughts about pathogenesis.
Jellinger KA
Mov Disord; 2014 Dec; 29(14):1720-41. PubMed ID: 25297524
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]