These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
47. DJ-1 forms complexes with mutant SOD1 and ameliorates its toxicity. Yamashita S; Mori A; Kimura E; Mita S; Maeda Y; Hirano T; Uchino M J Neurochem; 2010 May; 113(4):860-70. PubMed ID: 20202083 [TBL] [Abstract][Full Text] [Related]
48. Immunoproteasomes preserve protein homeostasis upon interferon-induced oxidative stress. Seifert U; Bialy LP; Ebstein F; Bech-Otschir D; Voigt A; Schröter F; Prozorovski T; Lange N; Steffen J; Rieger M; Kuckelkorn U; Aktas O; Kloetzel PM; Krüger E Cell; 2010 Aug; 142(4):613-24. PubMed ID: 20723761 [TBL] [Abstract][Full Text] [Related]
49. Dorfin-CHIP chimeric proteins potently ubiquitylate and degrade familial ALS-related mutant SOD1 proteins and reduce their cellular toxicity. Ishigaki S; Niwa J; Yamada S; Takahashi M; Ito T; Sone J; Doyu M; Urano F; Sobue G Neurobiol Dis; 2007 Feb; 25(2):331-41. PubMed ID: 17157513 [TBL] [Abstract][Full Text] [Related]
50. Erythrocytes as a preferential target of oxidative stress in blood. Fujii J; Homma T; Kobayashi S; Warang P; Madkaikar M; Mukherjee MB Free Radic Res; 2021 May; 55(5):562-580. PubMed ID: 33427524 [TBL] [Abstract][Full Text] [Related]
51. FAIM Is a Non-redundant Defender of Cellular Viability in the Face of Heat and Oxidative Stress and Interferes With Accumulation of Stress-Induced Protein Aggregates. Kaku H; Rothstein TL Front Mol Biosci; 2020; 7():32. PubMed ID: 32175331 [TBL] [Abstract][Full Text] [Related]
52. Managing Hyperosmotic Stress through Phase Separation. Peran I; Sabri N; Mittag T Trends Biochem Sci; 2020 Sep; 45(9):721-723. PubMed ID: 32446814 [TBL] [Abstract][Full Text] [Related]
53. [Nature of blocking of maturation of erythrocytes responsible for protein-deficiency anemia and role of hormonal balance disorders in its formation]. ASCHKENASY A J Physiol (Paris); 1955; 47(4):639-43. PubMed ID: 13307445 [No Abstract] [Full Text] [Related]
54. Distinct partitioning of ALS associated TDP-43, FUS and SOD1 mutants into cellular inclusions. Farrawell NE; Lambert-Smith IA; Warraich ST; Blair IP; Saunders DN; Hatters DM; Yerbury JJ Sci Rep; 2015 Aug; 5():13416. PubMed ID: 26293199 [TBL] [Abstract][Full Text] [Related]
55. A model for non-obligate oligomer formation in protein aggregration. Healy EF Biochem Biophys Res Commun; 2015 Sep; 465(3):523-7. PubMed ID: 26282203 [TBL] [Abstract][Full Text] [Related]
57. Destabilization of the dimer interface is a common consequence of diverse ALS-associated mutations in metal free SOD1. Broom HR; Rumfeldt JA; Vassall KA; Meiering EM Protein Sci; 2015 Dec; 24(12):2081-9. PubMed ID: 26362407 [TBL] [Abstract][Full Text] [Related]
58. Structural basis of Cu, Zn-superoxide dismutase amyloid fibril formation involves interaction of multiple peptide core regions. Ida M; Ando M; Adachi M; Tanaka A; Machida K; Hongo K; Mizobata T; Yamakawa MY; Watanabe Y; Nakashima K; Kawata Y J Biochem; 2016 Feb; 159(2):247-60. PubMed ID: 26319711 [TBL] [Abstract][Full Text] [Related]
59. Adverse effects of a SOD1-peptide immunotherapy on SOD1 G93A mouse slow model of amyotrophic lateral sclerosis. Sábado J; Casanovas A; Rodrigo H; Arqué G; Esquerda JE Neuroscience; 2015 Dec; 310():38-50. PubMed ID: 26384962 [TBL] [Abstract][Full Text] [Related]