BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

176 related articles for article (PubMed ID: 26446785)

  • 1. A Failure to Communicate: MYOSIN RESIDUES INVOLVED IN HYPERTROPHIC CARDIOMYOPATHY AFFECT INTER-DOMAIN INTERACTION.
    Kronert WA; Melkani GC; Melkani A; Bernstein SI
    J Biol Chem; 2015 Dec; 290(49):29270-80. PubMed ID: 26446785
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Mutating the converter-relay interface of Drosophila myosin perturbs ATPase activity, actin motility, myofibril stability and flight ability.
    Kronert WA; Melkani GC; Melkani A; Bernstein SI
    J Mol Biol; 2010 May; 398(5):625-32. PubMed ID: 20362584
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Alternative relay domains of Drosophila melanogaster myosin differentially affect ATPase activity, in vitro motility, myofibril structure and muscle function.
    Kronert WA; Dambacher CM; Knowles AF; Swank DM; Bernstein SI
    J Mol Biol; 2008 Jun; 379(3):443-56. PubMed ID: 18462751
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Mapping interactions between myosin relay and converter domains that power muscle function.
    Kronert WA; Melkani GC; Melkani A; Bernstein SI
    J Biol Chem; 2014 May; 289(18):12779-90. PubMed ID: 24627474
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Alternative relay and converter domains tune native muscle myosin isoform function in Drosophila.
    Kronert WA; Melkani GC; Melkani A; Bernstein SI
    J Mol Biol; 2012 Mar; 416(4):543-57. PubMed ID: 22226837
    [TBL] [Abstract][Full Text] [Related]  

  • 6. A Restrictive Cardiomyopathy Mutation in an Invariant Proline at the Myosin Head/Rod Junction Enhances Head Flexibility and Function, Yielding Muscle Defects in Drosophila.
    Achal M; Trujillo AS; Melkani GC; Farman GP; Ocorr K; Viswanathan MC; Kaushik G; Newhard CS; Glasheen BM; Melkani A; Suggs JA; Moore JR; Swank DM; Bodmer R; Cammarato A; Bernstein SI
    J Mol Biol; 2016 Jun; 428(11):2446-2461. PubMed ID: 27107639
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Reductions in ATPase activity, actin sliding velocity, and myofibril stability yield muscle dysfunction in Drosophila models of myosin-based Freeman-Sheldon syndrome.
    Rao DS; Kronert WA; Guo Y; Hsu KH; Sarsoza F; Bernstein SI
    Mol Biol Cell; 2019 Jan; 30(1):30-41. PubMed ID: 30379605
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Myosin Transducer Inter-Strand Communication Is Critical for Normal ATPase Activity and Myofibril Structure.
    Kronert WA; Hsu KH; Madan A; Sarsoza F; Cammarato A; Bernstein SI
    Biology (Basel); 2022 Jul; 11(8):. PubMed ID: 36009764
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Drosophila myosin mutants model the disparate severity of type 1 and type 2B distal arthrogryposis and indicate an enhanced actin affinity mechanism.
    Guo Y; Kronert WA; Hsu KH; Huang A; Sarsoza F; Bell KM; Suggs JA; Swank DM; Bernstein SI
    Skelet Muscle; 2020 Aug; 10(1):24. PubMed ID: 32799913
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Myosin heavy chain mutations that cause Freeman-Sheldon syndrome lead to muscle structural and functional defects in Drosophila.
    Das S; Kumar P; Verma A; Maiti TK; Mathew SJ
    Dev Biol; 2019 May; 449(2):90-98. PubMed ID: 30826400
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Kinetic analysis of Drosophila muscle myosin isoforms suggests a novel mode of mechanochemical coupling.
    Miller BM; Nyitrai M; Bernstein SI; Geeves MA
    J Biol Chem; 2003 Dec; 278(50):50293-300. PubMed ID: 14506231
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Point mutations in human beta cardiac myosin heavy chain have differential effects on sarcomeric structure and assembly: an ATP binding site change disrupts both thick and thin filaments, whereas hypertrophic cardiomyopathy mutations display normal assembly.
    Becker KD; Gottshall KR; Hickey R; Perriard JC; Chien KR
    J Cell Biol; 1997 Apr; 137(1):131-40. PubMed ID: 9105042
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Determining structure/function relationships for sarcomeric myosin heavy chain by genetic and transgenic manipulation of Drosophila.
    Swank DM; Wells L; Kronert WA; Morrill GE; Bernstein SI
    Microsc Res Tech; 2000 Sep; 50(6):430-42. PubMed ID: 10998634
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Myosin dilated cardiomyopathy mutation S532P disrupts actomyosin interactions, leading to altered muscle kinetics, reduced locomotion, and cardiac dilation in
    Trujillo AS; Hsu KH; Puthawala J; Viswanathan MC; Loya A; Irving TC; Cammarato A; Swank DM; Bernstein SI
    Mol Biol Cell; 2021 Aug; 32(18):1690-1706. PubMed ID: 34081531
    [TBL] [Abstract][Full Text] [Related]  

  • 15. A
    Suggs JA; Melkani GC; Glasheen BM; Detor MM; Melkani A; Marsan NP; Swank DM; Bernstein SI
    Dis Model Mech; 2017 Jun; 10(6):761-771. PubMed ID: 28258125
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Alternative versions of the myosin relay domain differentially respond to load to influence Drosophila muscle kinetics.
    Yang C; Ramanath S; Kronert WA; Bernstein SI; Maughan DW; Swank DM
    Biophys J; 2008 Dec; 95(11):5228-37. PubMed ID: 18805920
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Expression of the inclusion body myopathy 3 mutation in Drosophila depresses myosin function and stability and recapitulates muscle inclusions and weakness.
    Wang Y; Melkani GC; Suggs JA; Melkani A; Kronert WA; Cammarato A; Bernstein SI
    Mol Biol Cell; 2012 Jun; 23(11):2057-65. PubMed ID: 22496423
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Functional analysis of myosin mutations that cause familial hypertrophic cardiomyopathy.
    Roopnarine O; Leinwand LA
    Biophys J; 1998 Dec; 75(6):3023-30. PubMed ID: 9826622
    [TBL] [Abstract][Full Text] [Related]  

  • 19. The Relay/Converter Interface Influences Hydrolysis of ATP by Skeletal Muscle Myosin II.
    Bloemink MJ; Melkani GC; Bernstein SI; Geeves MA
    J Biol Chem; 2016 Jan; 291(4):1763-1773. PubMed ID: 26586917
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Aging-affiliated post-translational modifications of skeletal muscle myosin affect biochemical properties, myofibril structure, muscle function, and proteostasis.
    Neal CL; Kronert WA; Camillo JRT; Suggs JA; Huxford T; Bernstein SI
    Aging Cell; 2024 Jun; 23(6):e14134. PubMed ID: 38506610
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 9.