484 related articles for article (PubMed ID: 26490168)
1. FancJ (Brip1) loss-of-function allele results in spermatogonial cell depletion during embryogenesis and altered processing of crossover sites during meiotic prophase I in mice.
Sun X; Brieño-Enríquez MA; Cornelius A; Modzelewski AJ; Maley TT; Campbell-Peterson KM; Holloway JK; Cohen PE
Chromosoma; 2016 Jun; 125(2):237-52. PubMed ID: 26490168
[TBL] [Abstract][Full Text] [Related]
2. The DNA helicase FANCJ (BRIP1) functions in Double Strand Break repair processing, but not crossover formation during Prophase I of meiosis in male mice.
Horan TS; Ascenção CFR; Mellor CA; Wang M; Smolka MB; Cohen PE
bioRxiv; 2023 Oct; ():. PubMed ID: 37873301
[TBL] [Abstract][Full Text] [Related]
3. The DNA helicase FANCJ (BRIP1) functions in double strand break repair processing, but not crossover formation during prophase I of meiosis in male mice.
Horan TS; Ascenção CFR; Mellor C; Wang M; Smolka MB; Cohen PE
PLoS Genet; 2024 Feb; 20(2):e1011175. PubMed ID: 38377115
[TBL] [Abstract][Full Text] [Related]
4. A mutation in the endonuclease domain of mouse MLH3 reveals novel roles for MutLγ during crossover formation in meiotic prophase I.
Toledo M; Sun X; Brieño-Enríquez MA; Raghavan V; Gray S; Pea J; Milano CR; Venkatesh A; Patel L; Borst PL; Alani E; Cohen PE
PLoS Genet; 2019 Jun; 15(6):e1008177. PubMed ID: 31170160
[TBL] [Abstract][Full Text] [Related]
5. Assessing the link between BACH1/FANCJ and MLH1 in DNA crosslink repair.
Cantor SB; Xie J
Environ Mol Mutagen; 2010 Jul; 51(6):500-7. PubMed ID: 20658644
[TBL] [Abstract][Full Text] [Related]
6. FANCJ helicase operates in the Fanconi Anemia DNA repair pathway and the response to replicational stress.
Wu Y; Brosh RM
Curr Mol Med; 2009 May; 9(4):470-82. PubMed ID: 19519404
[TBL] [Abstract][Full Text] [Related]
7. Fanconi anemia group J helicase and MRE11 nuclease interact to facilitate the DNA damage response.
Suhasini AN; Sommers JA; Muniandy PA; Coulombe Y; Cantor SB; Masson JY; Seidman MM; Brosh RM
Mol Cell Biol; 2013 Jun; 33(11):2212-27. PubMed ID: 23530059
[TBL] [Abstract][Full Text] [Related]
8. The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells.
Peng M; Litman R; Xie J; Sharma S; Brosh RM; Cantor SB
EMBO J; 2007 Jul; 26(13):3238-49. PubMed ID: 17581638
[TBL] [Abstract][Full Text] [Related]
9. BRCA1-mediated repression of mutagenic end-joining of DNA double-strand breaks requires complex formation with BACH1.
Dohrn L; Salles D; Siehler SY; Kaufmann J; Wiesmüller L
Biochem J; 2012 Feb; 441(3):919-26. PubMed ID: 22032289
[TBL] [Abstract][Full Text] [Related]
10. HP1 regulates the localization of FANCJ at sites of DNA double-strand breaks.
Wu W; Togashi Y; Johmura Y; Miyoshi Y; Nobuoka S; Nakanishi M; Ohta T
Cancer Sci; 2016 Oct; 107(10):1406-1415. PubMed ID: 27399284
[TBL] [Abstract][Full Text] [Related]
11. FANCJ helicase controls the balance between short- and long-tract gene conversions between sister chromatids.
Nath S; Somyajit K; Mishra A; Scully R; Nagaraju G
Nucleic Acids Res; 2017 Sep; 45(15):8886-8900. PubMed ID: 28911102
[TBL] [Abstract][Full Text] [Related]
12. Fanconi anemia and Bloom's syndrome crosstalk through FANCJ-BLM helicase interaction.
Suhasini AN; Brosh RM
Trends Genet; 2012 Jan; 28(1):7-13. PubMed ID: 22024395
[TBL] [Abstract][Full Text] [Related]
13. Mammalian BTBD12 (SLX4) protects against genomic instability during mammalian spermatogenesis.
Holloway JK; Mohan S; Balmus G; Sun X; Modzelewski A; Borst PL; Freire R; Weiss RS; Cohen PE
PLoS Genet; 2011 Jun; 7(6):e1002094. PubMed ID: 21655083
[TBL] [Abstract][Full Text] [Related]
14. FANCJ uses its motor ATPase to destabilize protein-DNA complexes, unwind triplexes, and inhibit RAD51 strand exchange.
Sommers JA; Rawtani N; Gupta R; Bugreev DV; Mazin AV; Cantor SB; Brosh RM
J Biol Chem; 2009 Mar; 284(12):7505-17. PubMed ID: 19150983
[TBL] [Abstract][Full Text] [Related]
15. FANCJ (BACH1) helicase forms DNA damage inducible foci with replication protein A and interacts physically and functionally with the single-stranded DNA-binding protein.
Gupta R; Sharma S; Sommers JA; Kenny MK; Cantor SB; Brosh RM
Blood; 2007 Oct; 110(7):2390-8. PubMed ID: 17596542
[TBL] [Abstract][Full Text] [Related]
16. Variation in genome-wide levels of meiotic recombination is established at the onset of prophase in mammalian males.
Baier B; Hunt P; Broman KW; Hassold T
PLoS Genet; 2014 Jan; 10(1):e1004125. PubMed ID: 24497841
[TBL] [Abstract][Full Text] [Related]
17. Fanconi anemia group J mutation abolishes its DNA repair function by uncoupling DNA translocation from helicase activity or disruption of protein-DNA complexes.
Wu Y; Sommers JA; Suhasini AN; Leonard T; Deakyne JS; Mazin AV; Shin-Ya K; Kitao H; Brosh RM
Blood; 2010 Nov; 116(19):3780-91. PubMed ID: 20639400
[TBL] [Abstract][Full Text] [Related]
18. Mutational analysis of the breast cancer susceptibility gene BRIP1 /BACH1/FANCJ in high-risk non-BRCA1/BRCA2 breast cancer families.
Guénard F; Labrie Y; Ouellette G; Joly Beauparlant C; Simard J; ; Durocher F
J Hum Genet; 2008; 53(7):579. PubMed ID: 18414782
[TBL] [Abstract][Full Text] [Related]
19. CtIP- and ATR-dependent FANCJ phosphorylation in response to DNA strand breaks mediated by DNA replication.
Sakasai R; Sakai A; Iimori M; Kiyonari S; Matsuoka K; Kakeji Y; Kitao H; Maehara Y
Genes Cells; 2012 Dec; 17(12):962-70. PubMed ID: 23157317
[TBL] [Abstract][Full Text] [Related]
20. Hereditary breast cancer and the BRCA1-associated FANCJ/BACH1/BRIP1.
Cantor SB; Guillemette S
Future Oncol; 2011 Feb; 7(2):253-61. PubMed ID: 21345144
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
