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2. Distinct features between MLH1-methylated and unmethylated colorectal carcinomas with the CpG island methylator phenotype: implications in the serrated neoplasia pathway. Kim JH; Bae JM; Cho NY; Kang GH Oncotarget; 2016 Mar; 7(12):14095-111. PubMed ID: 26883113 [TBL] [Abstract][Full Text] [Related]
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9. The CpG island methylator phenotype and chromosomal instability are inversely correlated in sporadic colorectal cancer. Goel A; Nagasaka T; Arnold CN; Inoue T; Hamilton C; Niedzwiecki D; Compton C; Mayer RJ; Goldberg R; Bertagnolli MM; Boland CR Gastroenterology; 2007 Jan; 132(1):127-38. PubMed ID: 17087942 [TBL] [Abstract][Full Text] [Related]
10. BRAF mutation, CpG island methylator phenotype and microsatellite instability occur more frequently and concordantly in mucinous than non-mucinous colorectal cancer. Tanaka H; Deng G; Matsuzaki K; Kakar S; Kim GE; Miura S; Sleisenger MH; Kim YS Int J Cancer; 2006 Jun; 118(11):2765-71. PubMed ID: 16381005 [TBL] [Abstract][Full Text] [Related]
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16. The role of the CpG island methylator phenotype in colorectal cancer prognosis depends on microsatellite instability screening status. Dahlin AM; Palmqvist R; Henriksson ML; Jacobsson M; Eklöf V; Rutegård J; Oberg A; Van Guelpen BR Clin Cancer Res; 2010 Mar; 16(6):1845-55. PubMed ID: 20197478 [TBL] [Abstract][Full Text] [Related]
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