490 related articles for article (PubMed ID: 26762397)
1. Adult nephron-specific MR-deficient mice develop a severe renal PHA-1 phenotype.
Canonica J; Sergi C; Maillard M; Klusonova P; Odermatt A; Koesters R; Loffing-Cueni D; Loffing J; Rossier B; Frateschi S; Hummler E
Pflugers Arch; 2016 May; 468(5):895-908. PubMed ID: 26762397
[TBL] [Abstract][Full Text] [Related]
2. Severe Salt-Losing Syndrome and Hyperkalemia Induced by Adult Nephron-Specific Knockout of the Epithelial Sodium Channel α-Subunit.
Perrier R; Boscardin E; Malsure S; Sergi C; Maillard MP; Loffing J; Loffing-Cueni D; Sørensen MV; Koesters R; Rossier BC; Frateschi S; Hummler E
J Am Soc Nephrol; 2016 Aug; 27(8):2309-18. PubMed ID: 26701978
[TBL] [Abstract][Full Text] [Related]
3. Severe hyperkalemia is rescued by low-potassium diet in renal βENaC-deficient mice.
Boscardin E; Perrier R; Sergi C; Maillard M; Loffing J; Loffing-Cueni D; Koesters R; Rossier BC; Hummler E
Pflugers Arch; 2017 Oct; 469(10):1387-1399. PubMed ID: 28567665
[TBL] [Abstract][Full Text] [Related]
4. Plasma Potassium Determines NCC Abundance in Adult Kidney-Specific
Boscardin E; Perrier R; Sergi C; Maillard MP; Loffing J; Loffing-Cueni D; Koesters R; Rossier BC; Hummler E
J Am Soc Nephrol; 2018 Mar; 29(3):977-990. PubMed ID: 29371419
[TBL] [Abstract][Full Text] [Related]
5. [Regulation of kidney on potassium balance and its clinical significance].
Xie QH; Hao CM
Sheng Li Xue Bao; 2023 Apr; 75(2):216-230. PubMed ID: 37089096
[TBL] [Abstract][Full Text] [Related]
6. The mineralocorticoid receptor (MR) regulates ENaC but not NCC in mice with random MR deletion.
Czogalla J; Vohra T; Penton D; Kirschmann M; Craigie E; Loffing J
Pflugers Arch; 2016 May; 468(5):849-58. PubMed ID: 26898302
[TBL] [Abstract][Full Text] [Related]
7. Inducible kidney-specific Sgk1 knockout mice show a salt-losing phenotype.
Faresse N; Lagnaz D; Debonneville A; Ismailji A; Maillard M; Fejes-Toth G; Náray-Fejes-Tóth A; Staub O
Am J Physiol Renal Physiol; 2012 Apr; 302(8):F977-85. PubMed ID: 22301619
[TBL] [Abstract][Full Text] [Related]
8. Mineralocorticoid receptor antagonizes Dot1a-Af9 complex to increase αENaC transcription.
Zhang X; Zhou Q; Chen L; Berger S; Wu H; Xiao Z; Pearce D; Zhou X; Zhang W
Am J Physiol Renal Physiol; 2013 Nov; 305(10):F1436-44. PubMed ID: 24026182
[TBL] [Abstract][Full Text] [Related]
9. Glucocorticoid-induced leucine zipper protein regulates sodium and potassium balance in the distal nephron.
Rashmi P; Colussi G; Ng M; Wu X; Kidwai A; Pearce D
Kidney Int; 2017 May; 91(5):1159-1177. PubMed ID: 28094030
[TBL] [Abstract][Full Text] [Related]
10. Critical role of the mineralocorticoid receptor in aldosterone-dependent and aldosterone-independent regulation of ENaC in the distal nephron.
Nesterov V; Bertog M; Canonica J; Hummler E; Coleman R; Welling PA; Korbmacher C
Am J Physiol Renal Physiol; 2021 Sep; 321(3):F257-F268. PubMed ID: 34251271
[TBL] [Abstract][Full Text] [Related]
11. Renin-aldosterone response, urinary Na/K ratio and growth in pseudohypoaldosteronism patients with mutations in epithelial sodium channel (ENaC) subunit genes.
Hanukoglu A; Edelheit O; Shriki Y; Gizewska M; Dascal N; Hanukoglu I
J Steroid Biochem Mol Biol; 2008 Sep; 111(3-5):268-74. PubMed ID: 18634878
[TBL] [Abstract][Full Text] [Related]
12. Pseudohypoaldosteronism.
Riepe FG
Endocr Dev; 2013; 24():86-95. PubMed ID: 23392097
[TBL] [Abstract][Full Text] [Related]
13. Reducing αENaC expression in the kidney connecting tubule induces pseudohypoaldosteronism type 1 symptoms during K+ loading.
Poulsen SB; Praetorius J; Damkier HH; Miller L; Nelson RD; Hummler E; Christensen BM
Am J Physiol Renal Physiol; 2016 Feb; 310(4):F300-10. PubMed ID: 26582762
[TBL] [Abstract][Full Text] [Related]
14. [Mineralocorticoid resistance: pseudohypoaldosteronism type 1].
Fernandes-Rosa FL; Antonini SR
Arq Bras Endocrinol Metabol; 2007 Apr; 51(3):373-81. PubMed ID: 17546235
[TBL] [Abstract][Full Text] [Related]
15. The epithelial sodium channel and the control of sodium balance.
Schild L
Biochim Biophys Acta; 2010 Dec; 1802(12):1159-65. PubMed ID: 20600867
[TBL] [Abstract][Full Text] [Related]
16. Deletion of renal Nedd4-2 abolishes the effect of high sodium intake (HS) on Kir4.1, ENaC, and NCC and causes hypokalemia during high HS.
Zhang DD; Duan XP; Xiao Y; Wu P; Gao ZX; Wang WH; Lin DH
Am J Physiol Renal Physiol; 2021 May; 320(5):F883-F896. PubMed ID: 33818128
[TBL] [Abstract][Full Text] [Related]
17. Syndromes of impaired ion handling in the distal nephron: pseudohypoaldosteronism and familial hyperkalemic hypertension.
Kostakis ID; Cholidou KG; Perrea D
Hormones (Athens); 2012; 11(1):31-53. PubMed ID: 22450343
[TBL] [Abstract][Full Text] [Related]
18. 30 YEARS OF THE MINERALOCORTICOID RECEPTOR: Mineralocorticoid receptor mutations.
Zennaro MC; Fernandes-Rosa F
J Endocrinol; 2017 Jul; 234(1):T93-T106. PubMed ID: 28348114
[TBL] [Abstract][Full Text] [Related]
19. In Liddle Syndrome, Epithelial Sodium Channel Is Hyperactive Mainly in the Early Part of the Aldosterone-Sensitive Distal Nephron.
Nesterov V; Krueger B; Bertog M; Dahlmann A; Palmisano R; Korbmacher C
Hypertension; 2016 Jun; 67(6):1256-62. PubMed ID: 27170740
[TBL] [Abstract][Full Text] [Related]
20. A mouse model for the renal salt-wasting syndrome pseudohypoaldosteronism.
Hummler E; Barker P; Talbot C; Wang Q; Verdumo C; Grubb B; Gatzy J; Burnier M; Horisberger JD; Beermann F; Boucher R; Rossier BC
Proc Natl Acad Sci U S A; 1997 Oct; 94(21):11710-5. PubMed ID: 9326675
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
