175 related articles for article (PubMed ID: 27163630)
1. Ah receptor expression in cardiomyocytes protects adult female mice from heart dysfunction induced by TCDD exposure.
Kurita H; Carreira VS; Fan Y; Jiang M; Naticchioni M; Koch S; Rubinstein J; Puga A
Toxicology; 2016 Apr; 355-356():9-20. PubMed ID: 27163630
[TBL] [Abstract][Full Text] [Related]
2. Aryl Hydrocarbon Receptor Ablation in Cardiomyocytes Protects Male Mice From Heart Dysfunction Induced by NKX2.5 Haploinsufficiency.
Wang Q; Fan Y; Kurita H; Jiang M; Koch S; Rao MB; Rubinstein J; Puga A
Toxicol Sci; 2017 Nov; 160(1):74-82. PubMed ID: 28973413
[TBL] [Abstract][Full Text] [Related]
3. TCDD-induced expression of Ah receptor responsive genes in the pituitary and brain of cellular retinol-binding protein (CRBP-I) knockout mice.
Huang P; Ceccatelli S; Hoegberg P; Sten Shi TJ; Håkansson H; Rannug A
Toxicol Appl Pharmacol; 2003 Nov; 192(3):262-74. PubMed ID: 14575644
[TBL] [Abstract][Full Text] [Related]
4. Effects of aryl hydrocarbon receptor null mutation and in utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure on prostate and seminal vesicle development in C57BL/6 mice.
Lin TM; Ko K; Moore RW; Simanainen U; Oberley TD; Peterson RE
Toxicol Sci; 2002 Aug; 68(2):479-87. PubMed ID: 12151645
[TBL] [Abstract][Full Text] [Related]
5. Ah Receptor Activation by Dioxin Disrupts Activin, BMP, and WNT Signals During the Early Differentiation of Mouse Embryonic Stem Cells and Inhibits Cardiomyocyte Functions.
Wang Q; Kurita H; Carreira V; Ko CI; Fan Y; Zhang X; Biesiada J; Medvedovic M; Puga A
Toxicol Sci; 2016 Feb; 149(2):346-57. PubMed ID: 26572662
[TBL] [Abstract][Full Text] [Related]
6. Altered thyroxin and retinoid metabolic response to 2,3,7,8-tetrachlorodibenzo-p-dioxin in aryl hydrocarbon receptor-null mice.
Nishimura N; Yonemoto J; Miyabara Y; Fujii-Kuriyama Y; Tohyama C
Arch Toxicol; 2005 May; 79(5):260-7. PubMed ID: 15902423
[TBL] [Abstract][Full Text] [Related]
7. Role of aryl hydrocarbon receptor (AHR) in overall retinoid metabolism: Response comparisons to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure between wild-type and AHR knockout mice.
Esteban J; Sánchez-Pérez I; Hamscher G; Miettinen HM; Korkalainen M; Viluksela M; Pohjanvirta R; Håkansson H
Reprod Toxicol; 2021 Apr; 101():33-49. PubMed ID: 33607186
[TBL] [Abstract][Full Text] [Related]
8. In utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure: effects on fetal and adult cardiac gene expression and adult cardiac and renal morphology.
Aragon AC; Kopf PG; Campen MJ; Huwe JK; Walker MK
Toxicol Sci; 2008 Feb; 101(2):321-30. PubMed ID: 17975115
[TBL] [Abstract][Full Text] [Related]
9. NTP technical report on the toxicology and carcinogenesis studies of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) (CAS No. 1746-01-6) in female Harlan Sprague-Dawley rats (Gavage Studies).
National Toxicology Program
Natl Toxicol Program Tech Rep Ser; 2006 Apr; (521):4-232. PubMed ID: 16835633
[TBL] [Abstract][Full Text] [Related]
10. Expression of the aryl hydrocarbon receptor (AhR) and AhR nuclear translocator during chick cardiogenesis is consistent with 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced heart defects.
Walker MK; Pollenz RS; Smith SM
Toxicol Appl Pharmacol; 1997 Apr; 143(2):407-19. PubMed ID: 9144457
[TBL] [Abstract][Full Text] [Related]
11. Aryl hydrocarbon receptors in urogenital sinus mesenchyme mediate the inhibition of prostatic epithelial bud formation by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
Ko K; Moore RW; Peterson RE
Toxicol Appl Pharmacol; 2004 Apr; 196(1):149-55. PubMed ID: 15050416
[TBL] [Abstract][Full Text] [Related]
12. Role of the aryl hydrocarbon receptor in the development of control and 2,3,7,8-tetrachlorodibenzo-p-dioxin-exposed male mice.
Lin TM; Ko K; Moore RW; Buchanan DL; Cooke PS; Peterson RE
J Toxicol Environ Health A; 2001 Oct; 64(4):327-42. PubMed ID: 11693491
[TBL] [Abstract][Full Text] [Related]
13. Aryl hydrocarbon receptor activation in genital tubercle, palate, and other embryonic tissues in 2,3,7, 8-tetrachlorodibenzo-p-dioxin-responsive lacZ mice.
Willey JJ; Stripp BR; Baggs RB; Gasiewicz TA
Toxicol Appl Pharmacol; 1998 Jul; 151(1):33-44. PubMed ID: 9705885
[TBL] [Abstract][Full Text] [Related]
14. The aryl hydrocarbon receptor is indispensable for dioxin-induced defects in sexually-dimorphic behaviors due to the reduction in fetal steroidogenesis of the pituitary-gonadal axis in rats.
Hattori Y; Takeda T; Nakamura A; Nishida K; Shioji Y; Fukumitsu H; Yamada H; Ishii Y
Biochem Pharmacol; 2018 Aug; 154():213-221. PubMed ID: 29753751
[TBL] [Abstract][Full Text] [Related]
15. Hypertension, cardiac hypertrophy, and impaired vascular relaxation induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin are associated with increased superoxide.
Kopf PG; Huwe JK; Walker MK
Cardiovasc Toxicol; 2008 Dec; 8(4):181-93. PubMed ID: 18850075
[TBL] [Abstract][Full Text] [Related]
16. Aryl hydrocarbon receptor-dependent induction of loss of mitochondrial membrane potential in epididydimal spermatozoa by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD).
Fisher MT; Nagarkatti M; Nagarkatti PS
Toxicol Lett; 2005 Jun; 157(2):99-107. PubMed ID: 15836997
[TBL] [Abstract][Full Text] [Related]
17. The effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on molar and mandible traits in congenic mice: a test of the role of the Ahr locus.
Keller JM; Huang JC; Huet-Hudson Y; Leamy LJ
Toxicology; 2007 Dec; 242(1-3):52-62. PubMed ID: 17949883
[TBL] [Abstract][Full Text] [Related]
18. 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) induces plasminogen activator inhibitor-1 through an aryl hydrocarbon receptor-mediated pathway in mouse hepatoma cell lines.
Son DS; Rozman KK
Arch Toxicol; 2002 Jul; 76(7):404-13. PubMed ID: 12111005
[TBL] [Abstract][Full Text] [Related]
19. Putative link between transcriptional regulation of IgM expression by 2,3,7,8-tetrachlorodibenzo-p-dioxin and the aryl hydrocarbon receptor/dioxin-responsive enhancer signaling pathway.
Sulentic CE; Holsapple MP; Kaminski NE
J Pharmacol Exp Ther; 2000 Nov; 295(2):705-16. PubMed ID: 11046109
[TBL] [Abstract][Full Text] [Related]
20. Editor's Highlight: Ah Receptor Activation Potentiates Neutrophil Chemoattractant (C-X-C Motif) Ligand 5 Expression in Keratinocytes and Skin.
Smith KJ; Boyer JA; Muku GE; Murray IA; Gowda K; Desai D; Amin SG; Glick AB; Perdew GH
Toxicol Sci; 2017 Nov; 160(1):83-94. PubMed ID: 28973351
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
