BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

577 related articles for article (PubMed ID: 27190276)

  • 1. Intermittent hypoxia training protects cerebrovascular function in Alzheimer's disease.
    Manukhina EB; Downey HF; Shi X; Mallet RT
    Exp Biol Med (Maywood); 2016 Jun; 241(12):1351-63. PubMed ID: 27190276
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Cerebral hypoperfusion and glucose hypometabolism: Key pathophysiological modulators promote neurodegeneration, cognitive impairment, and Alzheimer's disease.
    Daulatzai MA
    J Neurosci Res; 2017 Apr; 95(4):943-972. PubMed ID: 27350397
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Alzheimer's disease in late-life dementia: a minor toxic consequence of devastating cerebrovascular dysfunction.
    Henry-Feugeas MC
    Med Hypotheses; 2008; 70(4):866-75. PubMed ID: 17825499
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Intermittent hypoxia training effectively protects against cognitive decline caused by acute hypoxia exposure.
    Zhang G; Yang G; Zhou Y; Cao Z; Yin M; Ma L; Fan M; Zhao YQ; Zhu L
    Pflugers Arch; 2024 Feb; 476(2):197-210. PubMed ID: 37994929
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Intermittent Hypoxia Training Prevents Deficient Learning-Memory Behavior in Mice Modeling Alzheimer's Disease: A Pilot Study.
    Ryou MG; Chen X; Cai M; Wang H; Jung ME; Metzger DB; Mallet RT; Shi X
    Front Aging Neurosci; 2021; 13():674688. PubMed ID: 34276338
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Targeting thrombin: an inflammatory neurotoxin in Alzheimer's disease.
    Grammas P; Martinez JM
    J Alzheimers Dis; 2014; 42 Suppl 4():S537-44. PubMed ID: 25079808
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Capillary dysfunction is associated with symptom severity and neurodegeneration in Alzheimer's disease.
    Nielsen RB; Egefjord L; Angleys H; Mouridsen K; Gejl M; Møller A; Brock B; Brændgaard H; Gottrup H; Rungby J; Eskildsen SF; Østergaard L
    Alzheimers Dement; 2017 Oct; 13(10):1143-1153. PubMed ID: 28343848
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Alzheimer's disease, cerebrovascular dysfunction and the benefits of exercise: from vessels to neurons.
    Lange-Asschenfeldt C; Kojda G
    Exp Gerontol; 2008 Jun; 43(6):499-504. PubMed ID: 18474414
    [TBL] [Abstract][Full Text] [Related]  

  • 9. The effects of impaired cerebral circulation on Alzheimer's disease pathology: evidence from animal studies.
    Villarreal AE; Barron R; Rao KS; Britton GB
    J Alzheimers Dis; 2014; 42(3):707-22. PubMed ID: 24927711
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Alzheimer disease and cerebrovascular pathology: an update.
    Jellinger KA
    J Neural Transm (Vienna); 2002 May; 109(5-6):813-36. PubMed ID: 12111471
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Vascular risk factors: a ticking time bomb to Alzheimer's disease.
    de la Torre JC
    Am J Alzheimers Dis Other Demen; 2013 Sep; 28(6):551-9. PubMed ID: 23813612
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Aberrant expression of nitric oxide synthase III in Alzheimer's disease: relevance to cerebral vasculopathy and neurodegeneration.
    de la Monte SM; Lu BX; Sohn YK; Etienne D; Kraft J; Ganju N; Wands JR
    Neurobiol Aging; 2000; 21(2):309-19. PubMed ID: 10867216
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Oxidative stress and cerebrovascular dysfunction in mouse models of Alzheimer's disease.
    Hamel E; Nicolakakis N; Aboulkassim T; Ongali B; Tong XK
    Exp Physiol; 2008 Jan; 93(1):116-20. PubMed ID: 17911359
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Cerebral hemodynamics and vascular risk factors: setting the stage for Alzheimer's disease.
    de la Torre JC
    J Alzheimers Dis; 2012; 32(3):553-67. PubMed ID: 22842871
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Vascular factors in Alzheimer's disease.
    Kalaria RN
    Int Psychogeriatr; 2003; 15 Suppl 1():47-52. PubMed ID: 16191216
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Treatment of intermittent hypoxia increases phosphorylated tau in the hippocampus via biological processes common to aging.
    Yagishita S; Suzuki S; Yoshikawa K; Iida K; Hirata A; Suzuki M; Takashima A; Maruyama K; Hirasawa A; Awaji T
    Mol Brain; 2017 Jan; 10(1):2. PubMed ID: 28057021
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Alzheimer's disease is a vasocognopathy: a new term to describe its nature.
    de la Torre JC
    Neurol Res; 2004 Jul; 26(5):517-24. PubMed ID: 15265269
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Is Vasomotion in Cerebral Arteries Impaired in Alzheimer's Disease?
    Di Marco LY; Farkas E; Martin C; Venneri A; Frangi AF
    J Alzheimers Dis; 2015; 46(1):35-53. PubMed ID: 25720414
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Metabolic Syndrome and the Cellular Phase of Alzheimer's Disease.
    Pugazhenthi S
    Prog Mol Biol Transl Sci; 2017; 146():243-258. PubMed ID: 28253987
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Evidence that Alzheimer's disease is a microvascular disorder: the role of constitutive nitric oxide.
    de la Torre JC; Stefano GB
    Brain Res Brain Res Rev; 2000 Dec; 34(3):119-36. PubMed ID: 11113503
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 29.