1585 related articles for article (PubMed ID: 27340545)
1. Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
Beqollari D; Romberg CF; Dobrowolny G; Martini M; Voss AA; Musarò A; Bannister RA
Skelet Muscle; 2016; 6():24. PubMed ID: 27340545
[TBL] [Abstract][Full Text] [Related]
2. Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.
Li QX; Mok SS; Laughton KM; McLean CA; Volitakis I; Cherny RA; Cheung NS; White AR; Masters CL
Aging Cell; 2006 Apr; 5(2):153-65. PubMed ID: 16626394
[TBL] [Abstract][Full Text] [Related]
3. The effect of peripheral nerve injury on disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
Sharp PS; Dick JR; Greensmith L
Neuroscience; 2005; 130(4):897-910. PubMed ID: 15652988
[TBL] [Abstract][Full Text] [Related]
4. Intact single muscle fibres from SOD1
Cheng AJ; Allodi I; Chaillou T; Schlittler M; Ivarsson N; Lanner JT; Thams S; Hedlund E; Andersson DC
J Physiol; 2019 Jun; 597(12):3133-3146. PubMed ID: 31074054
[TBL] [Abstract][Full Text] [Related]
5. Perturbations in intracellular Ca2+ handling in skeletal muscle in the G93A*SOD1 mouse model of amyotrophic lateral sclerosis.
Chin ER; Chen D; Bobyk KD; Mázala DA
Am J Physiol Cell Physiol; 2014 Dec; 307(11):C1031-8. PubMed ID: 25252949
[TBL] [Abstract][Full Text] [Related]
6. Defective mitochondrial dynamics is an early event in skeletal muscle of an amyotrophic lateral sclerosis mouse model.
Luo G; Yi J; Ma C; Xiao Y; Yi F; Yu T; Zhou J
PLoS One; 2013; 8(12):e82112. PubMed ID: 24324755
[TBL] [Abstract][Full Text] [Related]
7. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
Milanese M; Giribaldi F; Melone M; Bonifacino T; Musante I; Carminati E; Rossi PI; Vergani L; Voci A; Conti F; Puliti A; Bonanno G
Neurobiol Dis; 2014 Apr; 64():48-59. PubMed ID: 24361555
[TBL] [Abstract][Full Text] [Related]
8. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
Jaarsma D; Haasdijk ED; Grashorn JA; Hawkins R; van Duijn W; Verspaget HW; London J; Holstege JC
Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
[TBL] [Abstract][Full Text] [Related]
9. Functional over-load saves motor units in the SOD1-G93A transgenic mouse model of amyotrophic lateral sclerosis.
Gordon T; Tyreman N; Li S; Putman CT; Hegedus J
Neurobiol Dis; 2010 Feb; 37(2):412-22. PubMed ID: 19879358
[TBL] [Abstract][Full Text] [Related]
10. Amyloid precursor protein (APP) contributes to pathology in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
Bryson JB; Hobbs C; Parsons MJ; Bosch KD; Pandraud A; Walsh FS; Doherty P; Greensmith L
Hum Mol Genet; 2012 Sep; 21(17):3871-82. PubMed ID: 22678056
[TBL] [Abstract][Full Text] [Related]
11. Different human copper-zinc superoxide dismutase mutants, SOD1G93A and SOD1H46R, exert distinct harmful effects on gross phenotype in mice.
Pan L; Yoshii Y; Otomo A; Ogawa H; Iwasaki Y; Shang HF; Hadano S
PLoS One; 2012; 7(3):e33409. PubMed ID: 22438926
[TBL] [Abstract][Full Text] [Related]
12. Assessment of metal concentrations in the SOD1
Enge TG; Ecroyd H; Jolley DF; Yerbury JJ; Kalmar B; Dosseto A
Mol Cell Neurosci; 2018 Apr; 88():319-329. PubMed ID: 29524628
[TBL] [Abstract][Full Text] [Related]
13. Macrophage-mediated inflammation and glial response in the skeletal muscle of a rat model of familial amyotrophic lateral sclerosis (ALS).
Van Dyke JM; Smit-Oistad IM; Macrander C; Krakora D; Meyer MG; Suzuki M
Exp Neurol; 2016 Mar; 277():275-282. PubMed ID: 26775178
[TBL] [Abstract][Full Text] [Related]
14. Time course of preferential motor unit loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis.
Hegedus J; Putman CT; Gordon T
Neurobiol Dis; 2007 Nov; 28(2):154-64. PubMed ID: 17766128
[TBL] [Abstract][Full Text] [Related]
15. Glycoprotein nonmetastatic melanoma protein B ameliorates skeletal muscle lesions in a SOD1G93A mouse model of amyotrophic lateral sclerosis.
Nagahara Y; Shimazawa M; Tanaka H; Ono Y; Noda Y; Ohuchi K; Tsuruma K; Katsuno M; Sobue G; Hara H
J Neurosci Res; 2015 Oct; 93(10):1552-66. PubMed ID: 26140698
[TBL] [Abstract][Full Text] [Related]
16. Trehalose decreases mutant SOD1 expression and alleviates motor deficiency in early but not end-stage amyotrophic lateral sclerosis in a SOD1-G93A mouse model.
Li Y; Guo Y; Wang X; Yu X; Duan W; Hong K; Wang J; Han H; Li C
Neuroscience; 2015 Jul; 298():12-25. PubMed ID: 25841320
[TBL] [Abstract][Full Text] [Related]
17. Early changes of neuromuscular transmission in the SOD1(G93A) mice model of ALS start long before motor symptoms onset.
Rocha MC; Pousinha PA; Correia AM; Sebastião AM; Ribeiro JA
PLoS One; 2013; 8(9):e73846. PubMed ID: 24040091
[TBL] [Abstract][Full Text] [Related]
18. Stabilization of mutant Cu/Zn superoxide dismutase (SOD1) protein by coexpressed wild SOD1 protein accelerates the disease progression in familial amyotrophic lateral sclerosis mice.
Fukada K; Nagano S; Satoh M; Tohyama C; Nakanishi T; Shimizu A; Yanagihara T; Sakoda S
Eur J Neurosci; 2001 Dec; 14(12):2032-6. PubMed ID: 11860498
[TBL] [Abstract][Full Text] [Related]
19. Novel behavioural characteristics of the superoxide dismutase 1 G93A (SOD1
Kreilaus F; Guerra S; Masanetz R; Menne V; Yerbury J; Karl T
Genes Brain Behav; 2020 Feb; 19(2):e12604. PubMed ID: 31412164
[TBL] [Abstract][Full Text] [Related]
20. Equivalent L-type channel (Ca
Beqollari D; Kohrt WM; Bannister RA
Biochem Biophys Res Commun; 2020 Feb; 522(4):996-1002. PubMed ID: 31812241
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]