547 related articles for article (PubMed ID: 27365530)
1. Lessons from a double-transgenic neutrophil approach to induce antiproteinase 3 antibody-mediated vasculitis in mice.
Schreiber A; Eulenberg-Gustavus C; Bergmann A; Jerke U; Kettritz R
J Leukoc Biol; 2016 Dec; 100(6):1443-1452. PubMed ID: 27365530
[TBL] [Abstract][Full Text] [Related]
2. Complement receptor Mac-1 is an adaptor for NB1 (CD177)-mediated PR3-ANCA neutrophil activation.
Jerke U; Rolle S; Dittmar G; Bayat B; Santoso S; Sporbert A; Luft F; Kettritz R
J Biol Chem; 2011 Mar; 286(9):7070-81. PubMed ID: 21193407
[TBL] [Abstract][Full Text] [Related]
3. Interaction between CD177 and platelet endothelial cell adhesion molecule-1 downregulates membrane-bound proteinase-3 (PR3) expression on neutrophils and attenuates neutrophil activation induced by PR3-ANCA.
Deng H; Hu N; Wang C; Chen M; Zhao MH
Arthritis Res Ther; 2018 Sep; 20(1):213. PubMed ID: 30236159
[TBL] [Abstract][Full Text] [Related]
4. Coexpression of CD177 and membrane proteinase 3 on neutrophils in antineutrophil cytoplasmic autoantibody-associated systemic vasculitis: anti-proteinase 3-mediated neutrophil activation is independent of the role of CD177-expressing neutrophils.
Hu N; Westra J; Huitema MG; Bijl M; Brouwer E; Stegeman CA; Heeringa P; Limburg PC; Kallenberg CG
Arthritis Rheum; 2009 May; 60(5):1548-57. PubMed ID: 19404956
[TBL] [Abstract][Full Text] [Related]
5. Competitively disrupting the neutrophil-specific receptor-autoantigen CD177:proteinase 3 membrane complex reduces anti-PR3 antibody-induced neutrophil activation.
Marino SF; Jerke U; Rolle S; Daumke O; Kettritz R
J Biol Chem; 2022 Mar; 298(3):101598. PubMed ID: 35063507
[TBL] [Abstract][Full Text] [Related]
6. Intermediate monocytes in ANCA vasculitis: increased surface expression of ANCA autoantigens and IL-1β secretion in response to anti-MPO antibodies.
O'Brien EC; Abdulahad WH; Rutgers A; Huitema MG; O'Reilly VP; Coughlan AM; Harrington M; Heeringa P; Little MA; Hickey FB
Sci Rep; 2015 Jul; 5():11888. PubMed ID: 26149790
[TBL] [Abstract][Full Text] [Related]
7. Characterization of the CD177 interaction with the ANCA antigen proteinase 3.
Jerke U; Marino SF; Daumke O; Kettritz R
Sci Rep; 2017 Feb; 7():43328. PubMed ID: 28240246
[TBL] [Abstract][Full Text] [Related]
8. Elevated neutrophil membrane expression of proteinase 3 is dependent upon CD177 expression.
Abdgawad M; Gunnarsson L; Bengtsson AA; Geborek P; Nilsson L; Segelmark M; Hellmark T
Clin Exp Immunol; 2010 Jul; 161(1):89-97. PubMed ID: 20491791
[TBL] [Abstract][Full Text] [Related]
9. IgG from myeloperoxidase-antineutrophil cytoplasmic antibody-positive patients stimulates greater activation of primed neutrophils than IgG from proteinase 3-antineutrophil cytosplasmic antibody-positive patients.
Harper L; Radford D; Plant T; Drayson M; Adu D; Savage CO
Arthritis Rheum; 2001 Apr; 44(4):921-30. PubMed ID: 11315931
[TBL] [Abstract][Full Text] [Related]
10. Increased membrane expression of proteinase 3 during neutrophil adhesion in the presence of anti proteinase 3 antibodies.
Brachemi S; Mambole A; Fakhouri F; Mouthon L; Guillevin L; Lesavre P; Halbwachs-Mecarelli L
J Am Soc Nephrol; 2007 Aug; 18(8):2330-9. PubMed ID: 17634439
[TBL] [Abstract][Full Text] [Related]
11. Neutrophil priming and apoptosis in anti-neutrophil cytoplasmic autoantibody-associated vasculitis.
Harper L; Cockwell P; Adu D; Savage CO
Kidney Int; 2001 May; 59(5):1729-38. PubMed ID: 11318943
[TBL] [Abstract][Full Text] [Related]
12. Neutrophil surface presentation of the anti-neutrophil cytoplasmic antibody-antigen proteinase 3 depends on N-terminal processing.
von Vietinghoff S; Eulenberg C; Wellner M; Luft FC; Kettritz R
Clin Exp Immunol; 2008 Jun; 152(3):508-16. PubMed ID: 18462208
[TBL] [Abstract][Full Text] [Related]
13. Antineutrophil cytoplasm autoantibodies from patients with systemic vasculitis activate neutrophils through distinct signaling cascades: comparison with conventional Fcgamma receptor ligation.
Ben-Smith A; Dove SK; Martin A; Wakelam MJ; Savage CO
Blood; 2001 Sep; 98(5):1448-55. PubMed ID: 11520794
[TBL] [Abstract][Full Text] [Related]
14. Clinical significance of positive anti-neutrophil cytoplasmic antibodies without evidence of anti-neutrophil cytoplasmic antibodies-associated vasculitis.
Bornstein G; Ben-Zvi I; Furie N; Grossman C
Int J Rheum Dis; 2019 May; 22(5):940-945. PubMed ID: 30729688
[TBL] [Abstract][Full Text] [Related]
15. Pathogenesis of PR3-ANCA associated vasculitis.
Kallenberg CG
J Autoimmun; 2008; 30(1-2):29-36. PubMed ID: 18162369
[TBL] [Abstract][Full Text] [Related]
16. Distinction between MPO-ANCA and PR3-ANCA-associated glomerulonephritis in Chinese patients: a retrospective single-center study.
Hong Y; Shi P; Liu X; Yang L; Li K; Xu F; Liang S; Liu Z; Zhang H; Chen Y; Hu W
Clin Rheumatol; 2019 Jun; 38(6):1665-1673. PubMed ID: 30737591
[TBL] [Abstract][Full Text] [Related]
17. Targeting Cathepsin C in PR3-ANCA Vasculitis.
Jerke U; Eulenberg-Gustavus C; Rousselle A; Nicklin P; Kreideweiss S; Grundl MA; Eickholz P; Nickles K; Schreiber A; Korkmaz B; Kettritz R
J Am Soc Nephrol; 2022 May; 33(5):936-947. PubMed ID: 35292437
[TBL] [Abstract][Full Text] [Related]
18. Alternative complement pathway in the pathogenesis of disease mediated by anti-neutrophil cytoplasmic autoantibodies.
Xiao H; Schreiber A; Heeringa P; Falk RJ; Jennette JC
Am J Pathol; 2007 Jan; 170(1):52-64. PubMed ID: 17200182
[TBL] [Abstract][Full Text] [Related]
19. Pathogenesis of ANCA-associated vasculitides.
Kallenberg CG
Ann Rheum Dis; 2011 Mar; 70 Suppl 1():i59-63. PubMed ID: 21339221
[TBL] [Abstract][Full Text] [Related]
20. Bone marrow-derived cells are sufficient and necessary targets to mediate glomerulonephritis and vasculitis induced by anti-myeloperoxidase antibodies.
Schreiber A; Xiao H; Falk RJ; Jennette JC
J Am Soc Nephrol; 2006 Dec; 17(12):3355-64. PubMed ID: 17108314
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]