These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
630 related articles for article (PubMed ID: 27494840)
1. Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1. Bakr A; Köcher S; Volquardsen J; Petersen C; Borgmann K; Dikomey E; Rothkamm K; Mansour WY Oncotarget; 2016 Sep; 7(36):57679-57693. PubMed ID: 27494840 [TBL] [Abstract][Full Text] [Related]
2. A cell cycle-dependent regulatory circuit composed of 53BP1-RIF1 and BRCA1-CtIP controls DNA repair pathway choice. Escribano-Díaz C; Orthwein A; Fradet-Turcotte A; Xing M; Young JT; Tkáč J; Cook MA; Rosebrock AP; Munro M; Canny MD; Xu D; Durocher D Mol Cell; 2013 Mar; 49(5):872-83. PubMed ID: 23333306 [TBL] [Abstract][Full Text] [Related]
3. BRCA1 Directs the Repair Pathway to Homologous Recombination by Promoting 53BP1 Dephosphorylation. Isono M; Niimi A; Oike T; Hagiwara Y; Sato H; Sekine R; Yoshida Y; Isobe SY; Obuse C; Nishi R; Petricci E; Nakada S; Nakano T; Shibata A Cell Rep; 2017 Jan; 18(2):520-532. PubMed ID: 28076794 [TBL] [Abstract][Full Text] [Related]
4. 53BP1-RIF1-shieldin counteracts DSB resection through CST- and Polα-dependent fill-in. Mirman Z; Lottersberger F; Takai H; Kibe T; Gong Y; Takai K; Bianchi A; Zimmermann M; Durocher D; de Lange T Nature; 2018 Aug; 560(7716):112-116. PubMed ID: 30022158 [TBL] [Abstract][Full Text] [Related]
5. RIF1 is essential for 53BP1-dependent nonhomologous end joining and suppression of DNA double-strand break resection. Chapman JR; Barral P; Vannier JB; Borel V; Steger M; Tomas-Loba A; Sartori AA; Adams IR; Batista FD; Boulton SJ Mol Cell; 2013 Mar; 49(5):858-71. PubMed ID: 23333305 [TBL] [Abstract][Full Text] [Related]
6. H4K20me2 distinguishes pre-replicative from post-replicative chromatin to appropriately direct DNA repair pathway choice by 53BP1-RIF1-MAD2L2. Simonetta M; de Krijger I; Serrat J; Moatti N; Fortunato D; Hoekman L; Bleijerveld OB; Altelaar AFM; Jacobs JJL Cell Cycle; 2018; 17(1):124-136. PubMed ID: 29160738 [TBL] [Abstract][Full Text] [Related]
7. Regulation of repair pathway choice at two-ended DNA double-strand breaks. Shibata A Mutat Res; 2017 Oct; 803-805():51-55. PubMed ID: 28781144 [TBL] [Abstract][Full Text] [Related]
8. 53BP1 regulates DSB repair using Rif1 to control 5' end resection. Zimmermann M; Lottersberger F; Buonomo SB; Sfeir A; de Lange T Science; 2013 Feb; 339(6120):700-4. PubMed ID: 23306437 [TBL] [Abstract][Full Text] [Related]
9. Histone chaperone ASF1 acts with RIF1 to promote DNA end joining in BRCA1-deficient cells. Tang M; Chen Z; Wang C; Feng X; Lee N; Huang M; Zhang H; Li S; Xiong Y; Chen J J Biol Chem; 2022 Jun; 298(6):101979. PubMed ID: 35472331 [TBL] [Abstract][Full Text] [Related]
10. DNA end resection is needed for the repair of complex lesions in G1-phase human cells. Averbeck NB; Ringel O; Herrlitz M; Jakob B; Durante M; Taucher-Scholz G Cell Cycle; 2014; 13(16):2509-16. PubMed ID: 25486192 [TBL] [Abstract][Full Text] [Related]
11. DNA Double-Strand Break Resection Occurs during Non-homologous End Joining in G1 but Is Distinct from Resection during Homologous Recombination. Biehs R; Steinlage M; Barton O; Juhász S; Künzel J; Spies J; Shibata A; Jeggo PA; Löbrich M Mol Cell; 2017 Feb; 65(4):671-684.e5. PubMed ID: 28132842 [TBL] [Abstract][Full Text] [Related]
12. Age-associated deficient recruitment of 53BP1 in G1 cells directs DNA double-strand break repair to BRCA1/CtIP-mediated DNA-end resection. Anglada T; Genescà A; Martín M Aging (Albany NY); 2020 Dec; 12(24):24872-24893. PubMed ID: 33361520 [TBL] [Abstract][Full Text] [Related]
13. RIF1 acts in DNA repair through phosphopeptide recognition of 53BP1. Setiaputra D; Escribano-Díaz C; Reinert JK; Sadana P; Zong D; Callen E; Sifri C; Seebacher J; Nussenzweig A; Thomä NH; Durocher D Mol Cell; 2022 Apr; 82(7):1359-1371.e9. PubMed ID: 35216668 [TBL] [Abstract][Full Text] [Related]
15. A role for BLM in double-strand break repair pathway choice: prevention of CtIP/Mre11-mediated alternative nonhomologous end-joining. Grabarz A; Guirouilh-Barbat J; Barascu A; Pennarun G; Genet D; Rass E; Germann SM; Bertrand P; Hickson ID; Lopez BS Cell Rep; 2013 Oct; 5(1):21-8. PubMed ID: 24095737 [TBL] [Abstract][Full Text] [Related]
16. Regulation of DNA repair in the absence of classical non-homologous end joining. Kang YJ; Yan CT DNA Repair (Amst); 2018 Aug; 68():34-40. PubMed ID: 29929045 [TBL] [Abstract][Full Text] [Related]
18. Roles for 53BP1 in the repair of radiation-induced DNA double strand breaks. Shibata A; Jeggo PA DNA Repair (Amst); 2020 Sep; 93():102915. PubMed ID: 33087281 [TBL] [Abstract][Full Text] [Related]
19. BRCA2 antagonizes classical and alternative nonhomologous end-joining to prevent gross genomic instability. Han J; Ruan C; Huen MSY; Wang J; Xie A; Fu C; Liu T; Huang J Nat Commun; 2017 Nov; 8(1):1470. PubMed ID: 29133916 [TBL] [Abstract][Full Text] [Related]
20. PARP2 controls double-strand break repair pathway choice by limiting 53BP1 accumulation at DNA damage sites and promoting end-resection. Fouquin A; Guirouilh-Barbat J; Lopez B; Hall J; Amor-Guéret M; Pennaneach V Nucleic Acids Res; 2017 Dec; 45(21):12325-12339. PubMed ID: 29036662 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]