216 related articles for article (PubMed ID: 27564129)
1. TBP loading by AF4 through SL1 is the major rate-limiting step in MLL fusion-dependent transcription.
Okuda H; Takahashi S; Takaori-Kondo A; Yokoyama A
Cell Cycle; 2016 Oct; 15(20):2712-22. PubMed ID: 27564129
[TBL] [Abstract][Full Text] [Related]
2. AF4 uses the SL1 components of RNAP1 machinery to initiate MLL fusion- and AEP-dependent transcription.
Okuda H; Kanai A; Ito S; Matsui H; Yokoyama A
Nat Commun; 2015 Nov; 6():8869. PubMed ID: 26593443
[TBL] [Abstract][Full Text] [Related]
3. Transcriptional activation by MLL fusion proteins in leukemogenesis.
Yokoyama A
Exp Hematol; 2017 Feb; 46():21-30. PubMed ID: 27865805
[TBL] [Abstract][Full Text] [Related]
4. RNA Polymerase II-Dependent Transcription Initiated by Selectivity Factor 1: A Central Mechanism Used by MLL Fusion Proteins in Leukemic Transformation.
Yokoyama A
Front Genet; 2018; 9():722. PubMed ID: 30693017
[TBL] [Abstract][Full Text] [Related]
5. A higher-order complex containing AF4 and ENL family proteins with P-TEFb facilitates oncogenic and physiologic MLL-dependent transcription.
Yokoyama A; Lin M; Naresh A; Kitabayashi I; Cleary ML
Cancer Cell; 2010 Feb; 17(2):198-212. PubMed ID: 20153263
[TBL] [Abstract][Full Text] [Related]
6. The PAF complex synergizes with MLL fusion proteins at HOX loci to promote leukemogenesis.
Muntean AG; Tan J; Sitwala K; Huang Y; Bronstein J; Connelly JA; Basrur V; Elenitoba-Johnson KS; Hess JL
Cancer Cell; 2010 Jun; 17(6):609-21. PubMed ID: 20541477
[TBL] [Abstract][Full Text] [Related]
7. Cooperative gene activation by AF4 and DOT1L drives MLL-rearranged leukemia.
Okuda H; Stanojevic B; Kanai A; Kawamura T; Takahashi S; Matsui H; Takaori-Kondo A; Yokoyama A
J Clin Invest; 2017 May; 127(5):1918-1931. PubMed ID: 28394257
[TBL] [Abstract][Full Text] [Related]
8. Crosstalk between 14-3-3θ and AF4 enhances MLL-AF4 activity and promotes leukemia cell proliferation.
Fioretti T; Cevenini A; Zanobio M; Raia M; Sarnataro D; Salvatore F; Esposito G
Cell Oncol (Dordr); 2019 Dec; 42(6):829-845. PubMed ID: 31493143
[TBL] [Abstract][Full Text] [Related]
9. The full transforming capacity of MLL-Af4 is interlinked with lymphoid lineage commitment.
Lin S; Luo RT; Shrestha M; Thirman MJ; Mulloy JC
Blood; 2017 Aug; 130(7):903-907. PubMed ID: 28637661
[TBL] [Abstract][Full Text] [Related]
10. AF4 and AF4-MLL mediate transcriptional elongation of 5-lipoxygenase mRNA by 1, 25-dihydroxyvitamin D3.
Ahmad K; Scholz B; Capelo R; Schweighöfer I; Kahnt AS; Marschalek R; Steinhilber D
Oncotarget; 2015 Sep; 6(28):25784-800. PubMed ID: 26329759
[TBL] [Abstract][Full Text] [Related]
11. Leukemogenesis via aberrant self-renewal by the MLL/AEP-mediated transcriptional activation system.
Yokoyama A
Cancer Sci; 2021 Oct; 112(10):3935-3944. PubMed ID: 34251718
[TBL] [Abstract][Full Text] [Related]
12. Function of leukemogenic mixed lineage leukemia 1 (MLL) fusion proteins through distinct partner protein complexes.
Biswas D; Milne TA; Basrur V; Kim J; Elenitoba-Johnson KS; Allis CD; Roeder RG
Proc Natl Acad Sci U S A; 2011 Sep; 108(38):15751-6. PubMed ID: 21896721
[TBL] [Abstract][Full Text] [Related]
13. The AF4.MLL fusion protein is capable of inducing ALL in mice without requirement of MLL.AF4.
Bursen A; Schwabe K; Rüster B; Henschler R; Ruthardt M; Dingermann T; Marschalek R
Blood; 2010 Apr; 115(17):3570-9. PubMed ID: 20194896
[TBL] [Abstract][Full Text] [Related]
14. HBO1-MLL interaction promotes AF4/ENL/P-TEFb-mediated leukemogenesis.
Takahashi S; Kanai A; Okuda H; Miyamoto R; Komata Y; Kawamura T; Matsui H; Inaba T; Takaori-Kondo A; Yokoyama A
Elife; 2021 Aug; 10():. PubMed ID: 34431785
[TBL] [Abstract][Full Text] [Related]
15. The heterodimerization domains of MLL-FYRN and FYRC--are potential target structures in t(4;11) leukemia.
Pless B; Oehm C; Knauer S; Stauber RH; Dingermann T; Marschalek R
Leukemia; 2011 Apr; 25(4):663-70. PubMed ID: 21233834
[TBL] [Abstract][Full Text] [Related]
16. Nuclear FGFR2 Interacts with the MLL-AF4 Oncogenic Chimera and Positively Regulates
Fioretti T; Cevenini A; Zanobio M; Raia M; Sarnataro D; Cattaneo F; Ammendola R; Esposito G
Int J Mol Sci; 2021 Apr; 22(9):. PubMed ID: 33924850
[TBL] [Abstract][Full Text] [Related]
17. Activation of Ras-dependent Elk-1 activity by MLL-AF4 family fusion oncoproteins.
Ng MH; Ng RK; Kong CT; Jin DY; Chan LC
Exp Hematol; 2010 Jun; 38(6):481-8. PubMed ID: 20362031
[TBL] [Abstract][Full Text] [Related]
18. The mixed lineage leukemia (MLL) fusion-associated gene AF4 promotes CD133 transcription.
Mak AB; Nixon AM; Moffat J
Cancer Res; 2012 Apr; 72(8):1929-34. PubMed ID: 22337994
[TBL] [Abstract][Full Text] [Related]
19. circRNA circAF4 functions as an oncogene to regulate MLL-AF4 fusion protein expression and inhibit MLL leukemia progression.
Huang W; Fang K; Chen TQ; Zeng ZC; Sun YM; Han C; Sun LY; Chen ZH; Yang QQ; Pan Q; Luo XQ; Wang WT; Chen YQ
J Hematol Oncol; 2019 Oct; 12(1):103. PubMed ID: 31623653
[TBL] [Abstract][Full Text] [Related]
20. The leukemogenic AF4-MLL fusion protein causes P-TEFb kinase activation and altered epigenetic signatures.
Benedikt A; Baltruschat S; Scholz B; Bursen A; Arrey TN; Meyer B; Varagnolo L; Müller AM; Karas M; Dingermann T; Marschalek R
Leukemia; 2011 Jan; 25(1):135-44. PubMed ID: 21030982
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
