162 related articles for article (PubMed ID: 27672733)
1. A haploid HSV-1 genome platform for vector development: testing of the tetracycline-responsive switch shows interference by infected cell protein 0.
Khalique H; López Marco J; Lim F
J Gene Med; 2016 Oct; 18(10):302-311. PubMed ID: 27672733
[TBL] [Abstract][Full Text] [Related]
2. ICP0, ICP4, or VP16 expressed from adenovirus vectors induces reactivation of latent herpes simplex virus type 1 in primary cultures of latently infected trigeminal ganglion cells.
Halford WP; Kemp CD; Isler JA; Davido DJ; Schaffer PA
J Virol; 2001 Jul; 75(13):6143-53. PubMed ID: 11390616
[TBL] [Abstract][Full Text] [Related]
3. Cellular Transcriptional Coactivator RanBP10 and Herpes Simplex Virus 1 ICP0 Interact and Synergistically Promote Viral Gene Expression and Replication.
Sato Y; Kato A; Maruzuru Y; Oyama M; Kozuka-Hata H; Arii J; Kawaguchi Y
J Virol; 2016 Jan; 90(6):3173-86. PubMed ID: 26739050
[TBL] [Abstract][Full Text] [Related]
4. The Glucocorticoid Receptor (GR) Stimulates Herpes Simplex Virus 1 Productive Infection, in Part Because the Infected Cell Protein 0 (ICP0) Promoter Is Cooperatively Transactivated by the GR and Krüppel-Like Transcription Factor 15.
Ostler JB; Harrison KS; Schroeder K; Thunuguntla P; Jones C
J Virol; 2019 Mar; 93(6):. PubMed ID: 30602606
[TBL] [Abstract][Full Text] [Related]
5. HSV-1 infected cell proteins influence tetracycline-regulated transgene expression.
Herrlinger U; Pechan PA; Jacobs AH; Woiciechowski C; Rainov NG; Fraefel C; Paulus W; Reeves SA
J Gene Med; 2000; 2(5):379-89. PubMed ID: 11045432
[TBL] [Abstract][Full Text] [Related]
6. Equine herpesvirus 1 gene 12 can substitute for vmw65 in the growth of herpes simplex virus (HSV) type 1, allowing the generation of optimized cell lines for the propagation of HSV vectors with multiple immediate-early gene defects.
Thomas SK; Lilley CE; Latchman DS; Coffin RS
J Virol; 1999 Sep; 73(9):7399-409. PubMed ID: 10438830
[TBL] [Abstract][Full Text] [Related]
7. Tetracycline-regulated gene expression in replication-incompetent herpes simplex virus vectors.
Schmeisser F; Donohue M; Weir JP
Hum Gene Ther; 2002 Dec; 13(18):2113-24. PubMed ID: 12542843
[TBL] [Abstract][Full Text] [Related]
8. Evaluation of the T-REx transcription switch for conditional expression and regulation of HSV-1 vectors.
Knopf CW; Zavidij O; Rezuchova I; Rajcáni J
Virus Genes; 2008 Feb; 36(1):55-66. PubMed ID: 18193347
[TBL] [Abstract][Full Text] [Related]
9. Evidence that the herpes simplex virus type 1 ICP0 protein does not initiate reactivation from latency in vivo.
Thompson RL; Sawtell NM
J Virol; 2006 Nov; 80(22):10919-30. PubMed ID: 16943285
[TBL] [Abstract][Full Text] [Related]
10. Highly efficient regulation of gene expression by tetracycline in a replication-defective herpes simplex viral vector.
Yao F; Theopold C; Hoeller D; Bleiziffer O; Lu Z
Mol Ther; 2006 Jun; 13(6):1133-41. PubMed ID: 16574491
[TBL] [Abstract][Full Text] [Related]
11. Replication-Competent Controlled Herpes Simplex Virus.
Bloom DC; Feller J; McAnany P; Vilaboa N; Voellmy R
J Virol; 2015 Oct; 89(20):10668-79. PubMed ID: 26269179
[TBL] [Abstract][Full Text] [Related]
12. Multiple immediate-early gene-deficient herpes simplex virus vectors allowing efficient gene delivery to neurons in culture and widespread gene delivery to the central nervous system in vivo.
Lilley CE; Groutsi F; Han Z; Palmer JA; Anderson PN; Latchman DS; Coffin RS
J Virol; 2001 May; 75(9):4343-56. PubMed ID: 11287583
[TBL] [Abstract][Full Text] [Related]
13. Herpes simplex virus ICP0 promotes both histone removal and acetylation on viral DNA during lytic infection.
Cliffe AR; Knipe DM
J Virol; 2008 Dec; 82(24):12030-8. PubMed ID: 18842720
[TBL] [Abstract][Full Text] [Related]
14. Engineering cell lines for production of replication defective HSV-1 gene therapy vectors.
Grant KG; Krisky DM; Ataai MM; Glorioso JC
Biotechnol Bioeng; 2009 Mar; 102(4):1087-97. PubMed ID: 18828174
[TBL] [Abstract][Full Text] [Related]
15. Reactivation of expression from quiescent herpes simplex virus type 1 genomes in the absence of immediate-early protein ICP0.
Preston CM
J Virol; 2007 Nov; 81(21):11781-9. PubMed ID: 17715242
[TBL] [Abstract][Full Text] [Related]
16. Increased neurovirulence and reactivation of the herpes simplex virus type 1 latency-associated transcript (LAT)-negative mutant dLAT2903 with a disrupted LAT miR-H2.
Jiang X; Brown D; Osorio N; Hsiang C; BenMohamed L; Wechsler SL
J Neurovirol; 2016 Feb; 22(1):38-49. PubMed ID: 26069184
[TBL] [Abstract][Full Text] [Related]
17. Efficient activation of viral genomes by levels of herpes simplex virus ICP0 insufficient to affect cellular gene expression or cell survival.
Hobbs WE; Brough DE; Kovesdi I; DeLuca NA
J Virol; 2001 Apr; 75(7):3391-403. PubMed ID: 11238865
[TBL] [Abstract][Full Text] [Related]
18. Neurovirulent factor ICP34.5 uniquely expressed in the herpes simplex virus type 1 Delta gamma 1 34.5 mutant 1716.
Holman HA; MacLean AR
J Neurovirol; 2008 Jan; 14(1):28-40. PubMed ID: 18300073
[TBL] [Abstract][Full Text] [Related]
19. The immediate-early protein, ICP0, is essential for the resistance of herpes simplex virus to interferon-alpha/beta.
Härle P; Sainz B; Carr DJ; Halford WP
Virology; 2002 Feb; 293(2):295-304. PubMed ID: 11886249
[TBL] [Abstract][Full Text] [Related]
20. CTCF-dependent chromatin boundary element between the latency-associated transcript and ICP0 promoters in the herpes simplex virus type 1 genome.
Chen Q; Lin L; Smith S; Huang J; Berger SL; Zhou J
J Virol; 2007 May; 81(10):5192-201. PubMed ID: 17267480
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
