213 related articles for article (PubMed ID: 27881679)
1. The Shigella Virulence Factor IcsA Relieves N-WASP Autoinhibition by Displacing the Verprolin Homology/Cofilin/Acidic (VCA) Domain.
Mauricio RP; Jeffries CM; Svergun DI; Deane JE
J Biol Chem; 2017 Jan; 292(1):134-145. PubMed ID: 27881679
[TBL] [Abstract][Full Text] [Related]
2. Activation of the CDC42 effector N-WASP by the Shigella flexneri IcsA protein promotes actin nucleation by Arp2/3 complex and bacterial actin-based motility.
Egile C; Loisel TP; Laurent V; Li R; Pantaloni D; Sansonetti PJ; Carlier MF
J Cell Biol; 1999 Sep; 146(6):1319-32. PubMed ID: 10491394
[TBL] [Abstract][Full Text] [Related]
3. Identification of Shigella flexneri IcsA residues affecting interaction with N-WASP, and evidence for IcsA-IcsA co-operative interaction.
Teh MY; Morona R
PLoS One; 2013; 8(2):e55152. PubMed ID: 23405119
[TBL] [Abstract][Full Text] [Related]
4. Absence of O antigen suppresses Shigella flexneri IcsA autochaperone region mutations.
Teh MY; Tran ENH; Morona R
Microbiology (Reading); 2012 Nov; 158(Pt 11):2835-2850. PubMed ID: 22936034
[TBL] [Abstract][Full Text] [Related]
5. Mutagenesis of the Shigella flexneri autotransporter IcsA reveals novel functional regions involved in IcsA biogenesis and recruitment of host neural Wiscott-Aldrich syndrome protein.
May KL; Morona R
J Bacteriol; 2008 Jul; 190(13):4666-76. PubMed ID: 18456802
[TBL] [Abstract][Full Text] [Related]
6. Bacterial actin assembly requires toca-1 to relieve N-wasp autoinhibition.
Leung Y; Ally S; Goldberg MB
Cell Host Microbe; 2008 Jan; 3(1):39-47. PubMed ID: 18191793
[TBL] [Abstract][Full Text] [Related]
7. Identification of another actin-related protein (Arp) 2/3 complex binding site in neural Wiskott-Aldrich syndrome protein (N-WASP) that complements actin polymerization induced by the Arp2/3 complex activating (VCA) domain of N-WASP.
Suetsugu S; Miki H; Takenawa T
J Biol Chem; 2001 Aug; 276(35):33175-80. PubMed ID: 11432863
[TBL] [Abstract][Full Text] [Related]
8. Neural Wiskott-Aldrich syndrome protein is implicated in the actin-based motility of Shigella flexneri.
Suzuki T; Miki H; Takenawa T; Sasakawa C
EMBO J; 1998 May; 17(10):2767-76. PubMed ID: 9582270
[TBL] [Abstract][Full Text] [Related]
9. Neural Wiskott-Aldrich syndrome protein (N-WASP) is the specific ligand for Shigella VirG among the WASP family and determines the host cell type allowing actin-based spreading.
Suzuki T; Mimuro H; Suetsugu S; Miki H; Takenawa T; Sasakawa C
Cell Microbiol; 2002 Apr; 4(4):223-33. PubMed ID: 11952639
[TBL] [Abstract][Full Text] [Related]
10. Cysteine-Dependent Conformational Heterogeneity of Shigella flexneri Autotransporter IcsA and Implications of Its Function.
Qin J; Hong Y; Morona R; Totsika M
Microbiol Spectr; 2022 Dec; 10(6):e0341022. PubMed ID: 36374106
[No Abstract] [Full Text] [Related]
11. Functional analysis of a rickettsial OmpA homology domain of Shigella flexneri icsA.
Charles M; Magdalena J; Theriot JA; Goldberg MB
J Bacteriol; 1999 Feb; 181(3):869-78. PubMed ID: 9922250
[TBL] [Abstract][Full Text] [Related]
12. A small conserved motif supports polarity augmentation of Shigella flexneri IcsA.
Doyle MT; Grabowicz M; Morona R
Microbiology (Reading); 2015 Nov; 161(11):2087-97. PubMed ID: 26315462
[TBL] [Abstract][Full Text] [Related]
13. Genetic modulation of Shigella flexneri 2a lipopolysaccharide O antigen modal chain length reveals that it has been optimized for virulence.
Morona R; Daniels C; Van Den Bosch L
Microbiology (Reading); 2003 Apr; 149(Pt 4):925-939. PubMed ID: 12686635
[TBL] [Abstract][Full Text] [Related]
14. Effects of ectopically expressed neuronal Wiskott-Aldrich syndrome protein domains on Rickettsia rickettsii actin-based motility.
Harlander RS; Way M; Ren Q; Howe D; Grieshaber SS; Heinzen RA
Infect Immun; 2003 Mar; 71(3):1551-6. PubMed ID: 12595475
[TBL] [Abstract][Full Text] [Related]
15. The WH1 and EVH1 domains of WASP and Ena/VASP family members bind distinct sequence motifs.
Zettl M; Way M
Curr Biol; 2002 Sep; 12(18):1617-22. PubMed ID: 12372256
[TBL] [Abstract][Full Text] [Related]
16. Disruption of IcsP, the major Shigella protease that cleaves IcsA, accelerates actin-based motility.
Shere KD; Sallustio S; Manessis A; D'Aversa TG; Goldberg MB
Mol Microbiol; 1997 Aug; 25(3):451-62. PubMed ID: 9302008
[TBL] [Abstract][Full Text] [Related]
17. Polar localization of PhoN2, a periplasmic virulence-associated factor of Shigella flexneri, is required for proper IcsA exposition at the old bacterial pole.
Scribano D; Petrucca A; Pompili M; Ambrosi C; Bruni E; Zagaglia C; Prosseda G; Nencioni L; Casalino M; Polticelli F; Nicoletti M
PLoS One; 2014; 9(2):e90230. PubMed ID: 24587292
[TBL] [Abstract][Full Text] [Related]
18. Structural mechanism of WASP activation by the enterohaemorrhagic E. coli effector EspF(U).
Cheng HC; Skehan BM; Campellone KG; Leong JM; Rosen MK
Nature; 2008 Aug; 454(7207):1009-13. PubMed ID: 18650809
[TBL] [Abstract][Full Text] [Related]
19. The unipolar Shigella surface protein IcsA is targeted directly to the bacterial old pole: IcsP cleavage of IcsA occurs over the entire bacterial surface.
Steinhauer J; Agha R; Pham T; Varga AW; Goldberg MB
Mol Microbiol; 1999 Apr; 32(2):367-77. PubMed ID: 10231492
[TBL] [Abstract][Full Text] [Related]
20. Contribution of the periplasmic chaperone Skp to efficient presentation of the autotransporter IcsA on the surface of Shigella flexneri.
Wagner JK; Heindl JE; Gray AN; Jain S; Goldberg MB
J Bacteriol; 2009 Feb; 191(3):815-21. PubMed ID: 19047350
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]