180 related articles for article (PubMed ID: 28025109)
1. DEET potentiates the development and persistence of anticholinesterase dependent chronic pain signs in a rat model of Gulf War Illness pain.
Flunker LK; Nutter TJ; Johnson RD; Cooper BY
Toxicol Appl Pharmacol; 2017 Feb; 316():48-62. PubMed ID: 28025109
[TBL] [Abstract][Full Text] [Related]
2. Behavioral, cellular and molecular maladaptations covary with exposure to pyridostigmine bromide in a rat model of gulf war illness pain.
Cooper BY; Flunker LD; Johnson RD; Nutter TJ
Toxicol Appl Pharmacol; 2018 Aug; 352():119-131. PubMed ID: 29803855
[TBL] [Abstract][Full Text] [Related]
3. A delayed chronic pain like condition with decreased Kv channel activity in a rat model of Gulf War Illness pain syndrome.
Nutter TJ; Johnson RD; Cooper BY
Neurotoxicology; 2015 Dec; 51():67-79. PubMed ID: 26409647
[TBL] [Abstract][Full Text] [Related]
4. Neurotoxicity resulting from coexposure to pyridostigmine bromide, deet, and permethrin: implications of Gulf War chemical exposures.
Abou-Donia MB; Wilmarth KR; Jensen KF; Oehme FW; Kurt TL
J Toxicol Environ Health; 1996 May; 48(1):35-56. PubMed ID: 8637057
[TBL] [Abstract][Full Text] [Related]
5. Corticosterone and pyridostigmine/DEET exposure attenuate peripheral cytokine expression: Supporting a dominant role for neuroinflammation in a mouse model of Gulf War Illness.
Michalovicz LT; Locker AR; Kelly KA; Miller JV; Barnes Z; Fletcher MA; Miller DB; Klimas NG; Morris M; Lasley SM; O'Callaghan JP
Neurotoxicology; 2019 Jan; 70():26-32. PubMed ID: 30339781
[TBL] [Abstract][Full Text] [Related]
6. Pyridostigmine bromide modulates topical irritant-induced cytokine release from human epidermal keratinocytes and isolated perfused porcine skin.
Monteiro-Riviere NA; Baynes RE; Riviere JE
Toxicology; 2003 Feb; 183(1-3):15-28. PubMed ID: 12504339
[TBL] [Abstract][Full Text] [Related]
7. Mood and memory deficits in a model of Gulf War illness are linked with reduced neurogenesis, partial neuron loss, and mild inflammation in the hippocampus.
Parihar VK; Hattiangady B; Shuai B; Shetty AK
Neuropsychopharmacology; 2013 Nov; 38(12):2348-62. PubMed ID: 23807240
[TBL] [Abstract][Full Text] [Related]
8. Testicular germ-cell apoptosis in stressed rats following combined exposure to pyridostigmine bromide, N,N-diethyl m-toluamide (DEET), and permethrin.
Abou-Donia MB; Suliman HB; Khan WA; Abdel-Rahman AA
J Toxicol Environ Health A; 2003 Jan; 66(1):57-73. PubMed ID: 12587291
[TBL] [Abstract][Full Text] [Related]
9. Persistent Na+ and K+ channel dysfunctions after chronic exposure to insecticides and pyridostigmine bromide.
Nutter TJ; Jiang N; Cooper BY
Neurotoxicology; 2013 Dec; 39():72-83. PubMed ID: 23994030
[TBL] [Abstract][Full Text] [Related]
10. Corticosterone primes the neuroinflammatory response to DFP in mice: potential animal model of Gulf War Illness.
O'Callaghan JP; Kelly KA; Locker AR; Miller DB; Lasley SM
J Neurochem; 2015 Jun; 133(5):708-21. PubMed ID: 25753028
[TBL] [Abstract][Full Text] [Related]
11. Stress and combined exposure to low doses of pyridostigmine bromide, DEET, and permethrin produce neurochemical and neuropathological alterations in cerebral cortex, hippocampus, and cerebellum.
Abdel-Rahman A; Abou-Donia S; El-Masry E; Shetty A; Abou-Donia M
J Toxicol Environ Health A; 2004 Jan; 67(2):163-92. PubMed ID: 14675905
[TBL] [Abstract][Full Text] [Related]
12. Exposure to Gulf War Illness chemicals induces functional muscarinic receptor maladaptations in muscle nociceptors.
Cooper BY; Johnson RD; Nutter TJ
Neurotoxicology; 2016 May; 54():99-110. PubMed ID: 27058124
[TBL] [Abstract][Full Text] [Related]
13. Cardiorespiratory effects following acute exposure to pyridostigmine bromide and/or N,N-diethyl-m-toluamide (DEET) in rats.
Chaney LA; Rockhold RW; Hume AS
Int J Toxicol; 2002; 21(4):287-300. PubMed ID: 12171630
[TBL] [Abstract][Full Text] [Related]
14. Persistent modification of Nav1.9 following chronic exposure to insecticides and pyridostigmine bromide.
Nutter TJ; Cooper BY
Toxicol Appl Pharmacol; 2014 Jun; 277(3):298-309. PubMed ID: 24732443
[TBL] [Abstract][Full Text] [Related]
15. Co-exposure to pyridostigmine bromide, DEET, and/or permethrin causes sensorimotor deficit and alterations in brain acetylcholinesterase activity.
Abou-Donia MB; Dechkovskaia AM; Goldstein LB; Abdel-Rahman A; Bullman SL; Khan WA
Pharmacol Biochem Behav; 2004 Feb; 77(2):253-62. PubMed ID: 14751452
[TBL] [Abstract][Full Text] [Related]
16. Locomotor and sensorimotor performance deficit in rats following exposure to pyridostigmine bromide, DEET, and permethrin. Alone and in combination.
Schoenig GP
Toxicol Sci; 2002 Aug; 68(2):516-7; author reply 517-9. PubMed ID: 12151649
[No Abstract] [Full Text] [Related]
17. Exposure to Gulf War Illness-related agents leads to the development of chronic pain and fatigue.
Nguyen H; Sahbaie P; Goba L; Sul J; Suzaki A; Clark JD; Huang TT
Life Sci; 2021 Oct; 283():119867. PubMed ID: 34358550
[TBL] [Abstract][Full Text] [Related]
18. Induction of urinary excretion of 3-nitrotyrosine, a marker of oxidative stress, following administration of pyridostigmine bromide, DEET (N,N-diethyl-m-toluamide) and permethrin, alone and in combination in rats.
Abu-Qare AW; Suliman HB; Abou-Donia MB
Toxicol Lett; 2001 Apr; 121(2):127-34. PubMed ID: 11325563
[TBL] [Abstract][Full Text] [Related]
19. Increased neurotoxicity following concurrent exposure to pyridostigmine bromide, DEET, and chlorpyrifos.
Abou-Donia MB; Wilmarth KR; Abdel-Rahman AA; Jensen KF; Oehme FW; Kurt TL
Fundam Appl Toxicol; 1996 Dec; 34(2):201-22. PubMed ID: 8954750
[TBL] [Abstract][Full Text] [Related]
20. Evaluation of immunotoxicity induced by single or concurrent exposure to N,N-diethyl-m-toluamide (DEET), pyridostigmine bromide (PYR), and JP-8 jet fuel.
Peden-Adam MM; Eudaly J; Eudaly E; Dudley A; Zeigler J; Lee A; Robbs J; Gilkeson G; Keil DE
Toxicol Ind Health; 2001 Jun; 17(5-10):192-209. PubMed ID: 12539864
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]
