202 related articles for article (PubMed ID: 28039326)
1. Proximity to AGCT sequences dictates MMR-independent versus MMR-dependent mechanisms for AID-induced mutation via UNG2.
Thientosapol ES; Sharbeen G; Lau KKE; Bosnjak D; Durack T; Stevanovski I; Weninger W; Jolly CJ
Nucleic Acids Res; 2017 Apr; 45(6):3146-3157. PubMed ID: 28039326
[TBL] [Abstract][Full Text] [Related]
2. Strikingly different properties of uracil-DNA glycosylases UNG2 and SMUG1 may explain divergent roles in processing of genomic uracil.
Doseth B; Ekre C; Slupphaug G; Krokan HE; Kavli B
DNA Repair (Amst); 2012 Jun; 11(6):587-93. PubMed ID: 22483865
[TBL] [Abstract][Full Text] [Related]
3. Error-free versus mutagenic processing of genomic uracil--relevance to cancer.
Krokan HE; Sætrom P; Aas PA; Pettersen HS; Kavli B; Slupphaug G
DNA Repair (Amst); 2014 Jul; 19():38-47. PubMed ID: 24746924
[TBL] [Abstract][Full Text] [Related]
4. Uracil-DNA glycosylases SMUG1 and UNG2 coordinate the initial steps of base excision repair by distinct mechanisms.
Pettersen HS; Sundheim O; Gilljam KM; Slupphaug G; Krokan HE; Kavli B
Nucleic Acids Res; 2007; 35(12):3879-92. PubMed ID: 17537817
[TBL] [Abstract][Full Text] [Related]
5. Analysis of Ig gene hypermutation in Ung(-/-)Polh(-/-) mice suggests that UNG and A:T mutagenesis pathway target different U:G lesions.
Li S; Zhao Y; Wang JY
Mol Immunol; 2013 Mar; 53(3):214-7. PubMed ID: 22960197
[TBL] [Abstract][Full Text] [Related]
6. Pms2 and uracil-DNA glycosylases act jointly in the mismatch repair pathway to generate Ig gene mutations at A-T base pairs.
Girelli Zubani G; Zivojnovic M; De Smet A; Albagli-Curiel O; Huetz F; Weill JC; Reynaud CA; Storck S
J Exp Med; 2017 Apr; 214(4):1169-1180. PubMed ID: 28283534
[TBL] [Abstract][Full Text] [Related]
7. Repair of U/G and U/A in DNA by UNG2-associated repair complexes takes place predominantly by short-patch repair both in proliferating and growth-arrested cells.
Akbari M; Otterlei M; Peña-Diaz J; Aas PA; Kavli B; Liabakk NB; Hagen L; Imai K; Durandy A; Slupphaug G; Krokan HE
Nucleic Acids Res; 2004; 32(18):5486-98. PubMed ID: 15479784
[TBL] [Abstract][Full Text] [Related]
8. Interplay between Target Sequences and Repair Pathways Determines Distinct Outcomes of AID-Initiated Lesions.
Chen Z; Eder MD; Elos MT; Viboolsittiseri SS; Chen X; Wang JH
J Immunol; 2016 Mar; 196(5):2335-47. PubMed ID: 26810227
[TBL] [Abstract][Full Text] [Related]
9. Ectopic restriction of DNA repair reveals that UNG2 excises AID-induced uracils predominantly or exclusively during G1 phase.
Sharbeen G; Yee CW; Smith AL; Jolly CJ
J Exp Med; 2012 May; 209(5):965-74. PubMed ID: 22529268
[TBL] [Abstract][Full Text] [Related]
10. Different organization of base excision repair of uracil in DNA in nuclei and mitochondria and selective upregulation of mitochondrial uracil-DNA glycosylase after oxidative stress.
Akbari M; Otterlei M; Peña-Diaz J; Krokan HE
Neuroscience; 2007 Apr; 145(4):1201-12. PubMed ID: 17101234
[TBL] [Abstract][Full Text] [Related]
11. Rev1 is essential in generating G to C transversions downstream of the Ung2 pathway but not the Msh2+Ung2 hybrid pathway.
Krijger PH; Tsaalbi-Shtylik A; Wit N; van den Berk PC; de Wind N; Jacobs H
Eur J Immunol; 2013 Oct; 43(10):2765-70. PubMed ID: 23857323
[TBL] [Abstract][Full Text] [Related]
12. Activation-induced cytidine deaminase-dependent DNA breaks in class switch recombination occur during G1 phase of the cell cycle and depend upon mismatch repair.
Schrader CE; Guikema JE; Linehan EK; Selsing E; Stavnezer J
J Immunol; 2007 Nov; 179(9):6064-71. PubMed ID: 17947680
[TBL] [Abstract][Full Text] [Related]
13. Non-canonical uracil processing in DNA gives rise to double-strand breaks and deletions: relevance to class switch recombination.
Bregenhorn S; Kallenberger L; Artola-Borán M; Peña-Diaz J; Jiricny J
Nucleic Acids Res; 2016 Apr; 44(6):2691-705. PubMed ID: 26743004
[TBL] [Abstract][Full Text] [Related]
14. DNA polymerase beta is able to repair breaks in switch regions and plays an inhibitory role during immunoglobulin class switch recombination.
Wu X; Stavnezer J
J Exp Med; 2007 Jul; 204(7):1677-89. PubMed ID: 17591858
[TBL] [Abstract][Full Text] [Related]
15. Is Uracil-DNA Glycosylase UNG2 a New Cellular Weapon Against HIV-1?
Kara H; Chazal N; Bouaziz S
Curr HIV Res; 2019; 17(3):148-160. PubMed ID: 31433761
[TBL] [Abstract][Full Text] [Related]
16. Mismatch-mediated error prone repair at the immunoglobulin genes.
Chahwan R; Edelmann W; Scharff MD; Roa S
Biomed Pharmacother; 2011 Dec; 65(8):529-36. PubMed ID: 22100214
[TBL] [Abstract][Full Text] [Related]
17. High-fidelity correction of genomic uracil by human mismatch repair activities.
Larson ED; Bednarski DW; Maizels N
BMC Mol Biol; 2008 Oct; 9():94. PubMed ID: 18954457
[TBL] [Abstract][Full Text] [Related]
18. Interference of mismatch and base excision repair during the processing of adjacent U/G mispairs may play a key role in somatic hypermutation.
Schanz S; Castor D; Fischer F; Jiricny J
Proc Natl Acad Sci U S A; 2009 Apr; 106(14):5593-8. PubMed ID: 19307563
[TBL] [Abstract][Full Text] [Related]
19. The concerted action of Msh2 and UNG stimulates somatic hypermutation at A . T base pairs.
Frieder D; Larijani M; Collins C; Shulman M; Martin A
Mol Cell Biol; 2009 Sep; 29(18):5148-57. PubMed ID: 19596785
[TBL] [Abstract][Full Text] [Related]
20. Expression of human AID in yeast induces mutations in context similar to the context of somatic hypermutation at G-C pairs in immunoglobulin genes.
Mayorov VI; Rogozin IB; Adkison LR; Frahm C; Kunkel TA; Pavlov YI
BMC Immunol; 2005 Jun; 6():10. PubMed ID: 15949042
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]