BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

137 related articles for article (PubMed ID: 28069867)

  • 1. The carboxyl-terminal region of Dok-7 plays a key, but not essential, role in activation of muscle-specific receptor kinase MuSK and neuromuscular synapse formation.
    Ueta R; Tezuka T; Izawa Y; Miyoshi S; Nagatoishi S; Tsumoto K; Yamanashi Y
    J Biochem; 2017 Mar; 161(3):269-277. PubMed ID: 28069867
    [TBL] [Abstract][Full Text] [Related]  

  • 2. The MuSK activator agrin has a separate role essential for postnatal maintenance of neuromuscular synapses.
    Tezuka T; Inoue A; Hoshi T; Weatherbee SD; Burgess RW; Ueta R; Yamanashi Y
    Proc Natl Acad Sci U S A; 2014 Nov; 111(46):16556-61. PubMed ID: 25368159
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Dok-7/MuSK signaling and a congenital myasthenic syndrome.
    Yamanashi Y; Higuch O; Beeson D
    Acta Myol; 2008 Jul; 27(1):25-9. PubMed ID: 19108574
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Mutations causing DOK7 congenital myasthenia ablate functional motifs in Dok-7.
    Hamuro J; Higuchi O; Okada K; Ueno M; Iemura S; Natsume T; Spearman H; Beeson D; Yamanashi Y
    J Biol Chem; 2008 Feb; 283(9):5518-24. PubMed ID: 18165682
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Dok-7 activates the muscle receptor kinase MuSK and shapes synapse formation.
    Inoue A; Setoguchi K; Matsubara Y; Okada K; Sato N; Iwakura Y; Higuchi O; Yamanashi Y
    Sci Signal; 2009 Feb; 2(59):ra7. PubMed ID: 19244212
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Postnatal knockdown of dok-7 gene expression in mice causes structural defects in neuromuscular synapses and myasthenic pathology.
    Eguchi T; Tezuka T; Miyoshi S; Yamanashi Y
    Genes Cells; 2016 Jun; 21(6):670-6. PubMed ID: 27091576
    [TBL] [Abstract][Full Text] [Related]  

  • 7. [Molecular mechanisms underlying the formation and maintenance of neuromuscular junctions and a possible therapeutic approach.].
    Eguchi T; Tezuka T; Miyoshi S; Yamanashi Y
    Clin Calcium; 2017; 27(3):413-419. PubMed ID: 28232656
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Overexpression of Dok-7 in skeletal muscle enhances neuromuscular transmission with structural alterations of neuromuscular junctions: Implications in robustness of neuromuscular transmission.
    Eguchi T; Tezuka T; Fukudome T; Watanabe Y; Sagara H; Yamanashi Y
    Biochem Biophys Res Commun; 2020 Feb; 523(1):214-219. PubMed ID: 31848047
    [TBL] [Abstract][Full Text] [Related]  

  • 9. The muscle protein Dok-7 is essential for neuromuscular synaptogenesis.
    Okada K; Inoue A; Okada M; Murata Y; Kakuta S; Jigami T; Kubo S; Shiraishi H; Eguchi K; Motomura M; Akiyama T; Iwakura Y; Higuchi O; Yamanashi Y
    Science; 2006 Jun; 312(5781):1802-5. PubMed ID: 16794080
    [TBL] [Abstract][Full Text] [Related]  

  • 10. The cytoplasmic adaptor protein Dok7 activates the receptor tyrosine kinase MuSK via dimerization.
    Bergamin E; Hallock PT; Burden SJ; Hubbard SR
    Mol Cell; 2010 Jul; 39(1):100-9. PubMed ID: 20603078
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Dok-7 regulates neuromuscular synapse formation by recruiting Crk and Crk-L.
    Hallock PT; Xu CF; Park TJ; Neubert TA; Curran T; Burden SJ
    Genes Dev; 2010 Nov; 24(21):2451-61. PubMed ID: 21041412
    [TBL] [Abstract][Full Text] [Related]  

  • 12. The transcription factor Sp1 plays a crucial role in dok-7 gene expression.
    Hamuro J; Hishida Y; Higuchi O; Yamanashi Y
    Biochem Biophys Res Commun; 2011 May; 408(2):293-9. PubMed ID: 21504746
    [TBL] [Abstract][Full Text] [Related]  

  • 13. [Molecular mechanisms underlying the formation of neuromuscular junction].
    Higuchi O; Yamanashi Y
    Brain Nerve; 2011 Jul; 63(7):649-55. PubMed ID: 21747134
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Structure and activation of MuSK, a receptor tyrosine kinase central to neuromuscular junction formation.
    Hubbard SR; Gnanasambandan K
    Biochim Biophys Acta; 2013 Oct; 1834(10):2166-9. PubMed ID: 23467009
    [TBL] [Abstract][Full Text] [Related]  

  • 15. [Homeostasis and Disorder of Musculoskeletal System.Molecular signaling and its pathogenic alterations in neuromuscular junction formation.].
    Ueta R; Yamanashi Y
    Clin Calcium; 2018; 28(3):360-366. PubMed ID: 29512527
    [TBL] [Abstract][Full Text] [Related]  

  • 16. MuSk function during health and disease.
    Herbst R
    Neurosci Lett; 2020 Jan; 716():134676. PubMed ID: 31811897
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Dok-7 promotes slow muscle integrity as well as neuromuscular junction formation in a zebrafish model of congenital myasthenic syndromes.
    Müller JS; Jepson CD; Laval SH; Bushby K; Straub V; Lochmüller H
    Hum Mol Genet; 2010 May; 19(9):1726-40. PubMed ID: 20147321
    [TBL] [Abstract][Full Text] [Related]  

  • 18. Specific binding of collagen Q to the neuromuscular junction is exploited to cure congenital myasthenia and to explore bases of myasthenia gravis.
    Ohno K; Ito M; Kawakami Y; Krejci E; Engel AG
    Chem Biol Interact; 2013 Mar; 203(1):335-40. PubMed ID: 22981737
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Mesdc2 plays a key role in cell-surface expression of Lrp4 and postsynaptic specialization in myotubes.
    Hoshi T; Tezuka T; Yokoyama K; Iemura S; Natsume T; Yamanashi Y
    FEBS Lett; 2013 Nov; 587(23):3749-54. PubMed ID: 24140340
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Mutations in MUSK causing congenital myasthenic syndrome impair MuSK-Dok-7 interaction.
    Maselli RA; Arredondo J; Cagney O; Ng JJ; Anderson JA; Williams C; Gerke BJ; Soliven B; Wollmann RL
    Hum Mol Genet; 2010 Jun; 19(12):2370-9. PubMed ID: 20371544
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 7.