332 related articles for article (PubMed ID: 28125666)
1. Mononuclear Phagocytes Are Dispensable for Cardiac Remodeling in Established Pressure-Overload Heart Failure.
Patel B; Ismahil MA; Hamid T; Bansal SS; Prabhu SD
PLoS One; 2017; 12(1):e0170781. PubMed ID: 28125666
[TBL] [Abstract][Full Text] [Related]
2. Remodeling of the mononuclear phagocyte network underlies chronic inflammation and disease progression in heart failure: critical importance of the cardiosplenic axis.
Ismahil MA; Hamid T; Bansal SS; Patel B; Kingery JR; Prabhu SD
Circ Res; 2014 Jan; 114(2):266-82. PubMed ID: 24186967
[TBL] [Abstract][Full Text] [Related]
3. Cytosolic DNA sensor cGAS plays an essential pathogenetic role in pressure overload-induced heart failure.
Hu D; Cui YX; Wu MY; Li L; Su LN; Lian Z; Chen H
Am J Physiol Heart Circ Physiol; 2020 Jun; 318(6):H1525-H1537. PubMed ID: 32383996
[TBL] [Abstract][Full Text] [Related]
4. CD4+ T cells promote the transition from hypertrophy to heart failure during chronic pressure overload.
Laroumanie F; Douin-Echinard V; Pozzo J; Lairez O; Tortosa F; Vinel C; Delage C; Calise D; Dutaur M; Parini A; Pizzinat N
Circulation; 2014 May; 129(21):2111-24. PubMed ID: 24657994
[TBL] [Abstract][Full Text] [Related]
5. Experimental model of congestive heart failure induced by transverse aortic constriction in BALB/c mice.
Tannu S; Allocco J; Yarde M; Wong P; Ma X
J Pharmacol Toxicol Methods; 2020; 106():106935. PubMed ID: 33096237
[TBL] [Abstract][Full Text] [Related]
6. Long Non-Coding RNA Malat-1 Is Dispensable during Pressure Overload-Induced Cardiac Remodeling and Failure in Mice.
Peters T; Hermans-Beijnsberger S; Beqqali A; Bitsch N; Nakagawa S; Prasanth KV; de Windt LJ; van Oort RJ; Heymans S; Schroen B
PLoS One; 2016; 11(2):e0150236. PubMed ID: 26919721
[TBL] [Abstract][Full Text] [Related]
7. THE CARDIOSPLENIC AXIS IS ESSENTIAL FOR THE PATHOGENESIS OF ISCHEMIC HEART FAILURE.
Prabhu SD
Trans Am Clin Climatol Assoc; 2018; 129():202-214. PubMed ID: 30166715
[TBL] [Abstract][Full Text] [Related]
8. C-reactive protein overexpression exacerbates pressure overload-induced cardiac remodeling through enhanced inflammatory response.
Nagai T; Anzai T; Kaneko H; Mano Y; Anzai A; Maekawa Y; Takahashi T; Meguro T; Yoshikawa T; Fukuda K
Hypertension; 2011 Feb; 57(2):208-15. PubMed ID: 21220701
[TBL] [Abstract][Full Text] [Related]
9. Activated T Lymphocytes are Essential Drivers of Pathological Remodeling in Ischemic Heart Failure.
Bansal SS; Ismahil MA; Goel M; Patel B; Hamid T; Rokosh G; Prabhu SD
Circ Heart Fail; 2017 Mar; 10(3):e003688. PubMed ID: 28242779
[TBL] [Abstract][Full Text] [Related]
10. Leukocyte iNOS is required for inflammation and pathological remodeling in ischemic heart failure.
Kingery JR; Hamid T; Lewis RK; Ismahil MA; Bansal SS; Rokosh G; Townes TM; Ildstad ST; Jones SP; Prabhu SD
Basic Res Cardiol; 2017 Mar; 112(2):19. PubMed ID: 28238121
[TBL] [Abstract][Full Text] [Related]
11. Variable phenotype in murine transverse aortic constriction.
Mohammed SF; Storlie JR; Oehler EA; Bowen LA; Korinek J; Lam CS; Simari RD; Burnett JC; Redfield MM
Cardiovasc Pathol; 2012; 21(3):188-98. PubMed ID: 21764606
[TBL] [Abstract][Full Text] [Related]
12. The GABA
Bu J; Huang S; Wang J; Xia T; Liu H; You Y; Wang Z; Liu K
Front Immunol; 2021; 12():670153. PubMed ID: 34135897
[TBL] [Abstract][Full Text] [Related]
13. A time-series minimally invasive transverse aortic constriction mouse model for pressure overload-induced cardiac remodeling and heart failure.
Wang X; Zhu X; Shi L; Wang J; Xu Q; Yu B; Qu A
Front Cardiovasc Med; 2023; 10():1110032. PubMed ID: 36891245
[TBL] [Abstract][Full Text] [Related]
14. Ly6C(low) and not Ly6C(high) macrophages accumulate first in the heart in a model of murine pressure-overload.
Weisheit C; Zhang Y; Faron A; Köpke O; Weisheit G; Steinsträsser A; Frede S; Meyer R; Boehm O; Hoeft A; Kurts C; Baumgarten G
PLoS One; 2014; 9(11):e112710. PubMed ID: 25415601
[TBL] [Abstract][Full Text] [Related]
15. Genetic ablation of TRPV1 exacerbates pressure overload-induced cardiac hypertrophy.
Zhong B; Rubinstein J; Ma S; Wang DH
Biomed Pharmacother; 2018 Mar; 99():261-270. PubMed ID: 29334670
[TBL] [Abstract][Full Text] [Related]
16. Cardioprotective Role of Myeloid-Derived Suppressor Cells in Heart Failure.
Zhou L; Miao K; Yin B; Li H; Fan J; Zhu Y; Ba H; Zhang Z; Chen F; Wang J; Zhao C; Li Z; Wang DW
Circulation; 2018 Jul; 138(2):181-197. PubMed ID: 29437117
[TBL] [Abstract][Full Text] [Related]
17. CD8
Gröschel C; Sasse A; Monecke S; Röhrborn C; Elsner L; Didié M; Reupke V; Bunt G; Lichtman AH; Toischer K; Zimmermann WH; Hasenfuß G; Dressel R
Front Immunol; 2018; 9():2665. PubMed ID: 30498501
[TBL] [Abstract][Full Text] [Related]
18. Diet-induced obesity promotes altered remodeling and exacerbated cardiac hypertrophy following pressure overload.
Holzem KM; Marmerstein JT; Madden EJ; Efimov IR
Physiol Rep; 2015 Aug; 3(8):. PubMed ID: 26290533
[TBL] [Abstract][Full Text] [Related]
19. Syndecan-4 deficiency accelerates the transition from compensated hypertrophy to heart failure following pressure overload.
Li G; Xie J; Chen J; Li R; Wu H; Zhang X; Chen Q; Gu R; Xu B
Cardiovasc Pathol; 2017; 28():74-79. PubMed ID: 28395201
[TBL] [Abstract][Full Text] [Related]
20. Connective tissue growth factor inhibition attenuates left ventricular remodeling and dysfunction in pressure overload-induced heart failure.
Szabó Z; Magga J; Alakoski T; Ulvila J; Piuhola J; Vainio L; Kivirikko KI; Vuolteenaho O; Ruskoaho H; Lipson KE; Signore P; Kerkelä R
Hypertension; 2014 Jun; 63(6):1235-40. PubMed ID: 24688123
[TBL] [Abstract][Full Text] [Related]
[Next] [New Search]