BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

446 related articles for article (PubMed ID: 28291959)

  • 1. The Opening of ATP-Sensitive K+ Channels Protects H9c2 Cardiac Cells Against the High Glucose-Induced Injury and Inflammation by Inhibiting the ROS-TLR4-Necroptosis Pathway.
    Liang W; Chen M; Zheng D; Li J; Song M; Zhang W; Feng J; Lan J
    Cell Physiol Biochem; 2017; 41(3):1020-1034. PubMed ID: 28291959
    [TBL] [Abstract][Full Text] [Related]  

  • 2. ATP-sensitive K⁺ channels contribute to the protective effects of exogenous hydrogen sulfide against high glucose-induced injury in H9c2 cardiac cells.
    Liang W; Chen J; Mo L; Ke X; Zhang W; Zheng D; Pan W; Wu S; Feng J; Song M; Liao X
    Int J Mol Med; 2016 Mar; 37(3):763-72. PubMed ID: 26820501
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Klotho attenuates angiotensin II‑induced cardiotoxicity through suppression of necroptosis and oxidative stress.
    Yu S; Yang H; Guo X; Sun Y
    Mol Med Rep; 2021 Jan; 23(1):. PubMed ID: 33215215
    [TBL] [Abstract][Full Text] [Related]  

  • 4. A novel damage mechanism: Contribution of the interaction between necroptosis and ROS to high glucose-induced injury and inflammation in H9c2 cardiac cells.
    Liang W; Chen M; Zheng D; He J; Song M; Mo L; Feng J; Lan J
    Int J Mol Med; 2017 Jul; 40(1):201-208. PubMed ID: 28560421
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Alterations in necroptosis during ALDH2‑mediated protection against high glucose‑induced H9c2 cardiac cell injury.
    Fang T; Cao R; Wang W; Ye H; Shen L; Li Z; Hu J; Gao Q
    Mol Med Rep; 2018 Sep; 18(3):2807-2815. PubMed ID: 30015964
    [TBL] [Abstract][Full Text] [Related]  

  • 6. Naringin inhibits ROS-activated MAPK pathway in high glucose-induced injuries in H9c2 cardiac cells.
    Chen J; Guo R; Yan H; Tian L; You Q; Li S; Huang R; Wu K
    Basic Clin Pharmacol Toxicol; 2014 Apr; 114(4):293-304. PubMed ID: 24118820
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Exogenous H2S protects H9c2 cardiac cells against high glucose-induced injury and inflammation by inhibiting the activation of the NF-κB and IL-1β pathways.
    Xu W; Chen J; Lin J; Liu D; Mo L; Pan W; Feng J; Wu W; Zheng D
    Int J Mol Med; 2015 Jan; 35(1):177-86. PubMed ID: 25412187
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Exogenous hydrogen sulfide protects human umbilical vein endothelial cells against high glucose‑induced injury by inhibiting the necroptosis pathway.
    Lin J; Chen M; Liu D; Guo R; Lin K; Deng H; Zhi X; Zhang W; Feng J; Wu W
    Int J Mol Med; 2018 Mar; 41(3):1477-1486. PubMed ID: 29286079
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Necrostatin-1 Protects Against Paraquat-Induced Cardiac Contractile Dysfunction via RIP1-RIP3-MLKL-Dependent Necroptosis Pathway.
    Zhang L; Feng Q; Wang T
    Cardiovasc Toxicol; 2018 Aug; 18(4):346-355. PubMed ID: 29299822
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Diazoxide affects mitochondrial bioenergetics by the opening of mKATP channel on submicromolar scale.
    Akopova O; Kolchinskaya L; Nosar V; Mankovska I; Sagach V
    BMC Mol Cell Biol; 2020 Apr; 21(1):31. PubMed ID: 32306897
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Penehyclidine hydrochloride protects against anoxia/reoxygenation injury in cardiomyocytes through ATP-sensitive potassium channels, and the Akt/GSK-3β and Akt/mTOR signaling pathways.
    Zi C; Zhang C; Yang Y; Ma J
    Cell Biol Int; 2020 Jun; 44(6):1353-1362. PubMed ID: 32125033
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Acetylcholine leads to free radical production dependent on K(ATP) channels, G(i) proteins, phosphatidylinositol 3-kinase and tyrosine kinase.
    Oldenburg O; Qin Q; Sharma AR; Cohen MV; Downey JM; Benoit JN
    Cardiovasc Res; 2002 Aug; 55(3):544-52. PubMed ID: 12160951
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Exogenous Hydrogen Sulfide Attenuates High Glucose-Induced Cardiotoxicity by Inhibiting NLRP3 Inflammasome Activation by Suppressing TLR4/NF-κB Pathway in H9c2 Cells.
    Huang Z; Zhuang X; Xie C; Hu X; Dong X; Guo Y; Li S; Liao X
    Cell Physiol Biochem; 2016; 40(6):1578-1590. PubMed ID: 27997926
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Mitochondrial uncoupling, with low concentration FCCP, induces ROS-dependent cardioprotection independent of KATP channel activation.
    Brennan JP; Southworth R; Medina RA; Davidson SM; Duchen MR; Shattock MJ
    Cardiovasc Res; 2006 Nov; 72(2):313-21. PubMed ID: 16950237
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Cytosolic calcium mediates RIP1/RIP3 complex-dependent necroptosis through JNK activation and mitochondrial ROS production in human colon cancer cells.
    Sun W; Wu X; Gao H; Yu J; Zhao W; Lu JJ; Wang J; Du G; Chen X
    Free Radic Biol Med; 2017 Jul; 108():433-444. PubMed ID: 28414098
    [TBL] [Abstract][Full Text] [Related]  

  • 16. Exogenous hydrogen sulfide protects H9c2 cardiac cells against high glucose-induced injury by inhibiting the activities of the p38 MAPK and ERK1/2 pathways.
    Xu W; Wu W; Chen J; Guo R; Lin J; Liao X; Feng J
    Int J Mol Med; 2013 Oct; 32(4):917-25. PubMed ID: 23912965
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Accelerated recovery of mitochondrial membrane potential by GSK-3β inactivation affords cardiomyocytes protection from oxidant-induced necrosis.
    Sunaga D; Tanno M; Kuno A; Ishikawa S; Ogasawara M; Yano T; Miki T; Miura T
    PLoS One; 2014; 9(11):e112529. PubMed ID: 25390651
    [TBL] [Abstract][Full Text] [Related]  

  • 18. [Regulation of the mitochondrial ATP-sensitive potassium channel in rat uterus cells by ROS].
    Badziuk OB; Mazur IuIu; Kosterin SO
    Ukr Biokhim Zh (1999); 2011; 83(3):48-57. PubMed ID: 21888054
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Effects of ATP-sensitive potassium channel activators diazoxide and BMS-191095 on membrane potential and reactive oxygen species production in isolated piglet mitochondria.
    Busija DW; Katakam P; Rajapakse NC; Kis B; Grover G; Domoki F; Bari F
    Brain Res Bull; 2005 Jul; 66(2):85-90. PubMed ID: 15982523
    [TBL] [Abstract][Full Text] [Related]  

  • 20. Protection against ischemia-induced ventricular arrhythmias and myocardial dysfunction conferred by preconditioning in the rat heart: involvement of mitochondrial K(ATP) channels and reactive oxygen species.
    Matejíková J; Kucharská J; Pintérová M; Pancza D; Ravingerová T
    Physiol Res; 2009; 58(1):9-19. PubMed ID: 18198991
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 23.