BIOMARKERS

Molecular Biopsy of Human Tumors

- a resource for Precision Medicine *

267 related articles for article (PubMed ID: 2832737)

  • 1. Premature translation termination mediates triosephosphate isomerase mRNA degradation.
    Daar IO; Maquat LE
    Mol Cell Biol; 1988 Feb; 8(2):802-13. PubMed ID: 2832737
    [TBL] [Abstract][Full Text] [Related]  

  • 2. Translation to near the distal end of the penultimate exon is required for normal levels of spliced triosephosphate isomerase mRNA.
    Cheng J; Fogel-Petrovic M; Maquat LE
    Mol Cell Biol; 1990 Oct; 10(10):5215-25. PubMed ID: 2398889
    [TBL] [Abstract][Full Text] [Related]  

  • 3. Introns are cis effectors of the nonsense-codon-mediated reduction in nuclear mRNA abundance.
    Cheng J; Belgrader P; Zhou X; Maquat LE
    Mol Cell Biol; 1994 Sep; 14(9):6317-25. PubMed ID: 8065363
    [TBL] [Abstract][Full Text] [Related]  

  • 4. Evidence to implicate translation by ribosomes in the mechanism by which nonsense codons reduce the nuclear level of human triosephosphate isomerase mRNA.
    Belgrader P; Cheng J; Maquat LE
    Proc Natl Acad Sci U S A; 1993 Jan; 90(2):482-6. PubMed ID: 8421679
    [TBL] [Abstract][Full Text] [Related]  

  • 5. Nonsense codons can reduce the abundance of nuclear mRNA without affecting the abundance of pre-mRNA or the half-life of cytoplasmic mRNA.
    Cheng J; Maquat LE
    Mol Cell Biol; 1993 Mar; 13(3):1892-902. PubMed ID: 8441420
    [TBL] [Abstract][Full Text] [Related]  

  • 6. At least one intron is required for the nonsense-mediated decay of triosephosphate isomerase mRNA: a possible link between nuclear splicing and cytoplasmic translation.
    Zhang J; Sun X; Qian Y; LaDuca JP; Maquat LE
    Mol Cell Biol; 1998 Sep; 18(9):5272-83. PubMed ID: 9710612
    [TBL] [Abstract][Full Text] [Related]  

  • 7. Evidence that the decay of nucleus-associated nonsense mRNA for human triosephosphate isomerase involves nonsense codon recognition after splicing.
    Zhang J; Maquat LE
    RNA; 1996 Mar; 2(3):235-43. PubMed ID: 8608447
    [TBL] [Abstract][Full Text] [Related]  

  • 8. Evidence that translation reinitiation abrogates nonsense-mediated mRNA decay in mammalian cells.
    Zhang J; Maquat LE
    EMBO J; 1997 Feb; 16(4):826-33. PubMed ID: 9049311
    [TBL] [Abstract][Full Text] [Related]  

  • 9. Triose phosphate isomerase deficiency is caused by altered dimerization--not catalytic inactivity--of the mutant enzymes.
    Ralser M; Heeren G; Breitenbach M; Lehrach H; Krobitsch S
    PLoS One; 2006 Dec; 1(1):e30. PubMed ID: 17183658
    [TBL] [Abstract][Full Text] [Related]  

  • 10. Mammalian nonsense codons can be cis effectors of nuclear mRNA half-life.
    Belgrader P; Cheng J; Zhou X; Stephenson LS; Maquat LE
    Mol Cell Biol; 1994 Dec; 14(12):8219-28. PubMed ID: 7969159
    [TBL] [Abstract][Full Text] [Related]  

  • 11. Human triose-phosphate isomerase deficiency: a single amino acid substitution results in a thermolabile enzyme.
    Daar IO; Artymiuk PJ; Phillips DC; Maquat LE
    Proc Natl Acad Sci U S A; 1986 Oct; 83(20):7903-7. PubMed ID: 2876430
    [TBL] [Abstract][Full Text] [Related]  

  • 12. Human triosephosphate isomerase cDNA and protein structure. Studies of triosephosphate isomerase deficiency in man.
    Maquat LE; Chilcote R; Ryan PM
    J Biol Chem; 1985 Mar; 260(6):3748-53. PubMed ID: 2579079
    [TBL] [Abstract][Full Text] [Related]  

  • 13. Characterization of the functional gene and several processed pseudogenes in the human triosephosphate isomerase gene family.
    Brown JR; Daar IO; Krug JR; Maquat LE
    Mol Cell Biol; 1985 Jul; 5(7):1694-706. PubMed ID: 4022011
    [TBL] [Abstract][Full Text] [Related]  

  • 14. Arginine CGA codons as a source of nonsense mutations: a possible role in multivariant gene expression, control of mRNA quality, and aging.
    Romanov GA; Sukhoverov VS
    Mol Genet Genomics; 2017 Oct; 292(5):1013-1026. PubMed ID: 28523359
    [TBL] [Abstract][Full Text] [Related]  

  • 15. Nonsense but not missense mutations can decrease the abundance of nuclear mRNA for the mouse major urinary protein, while both types of mutations can facilitate exon skipping.
    Belgrader P; Maquat LE
    Mol Cell Biol; 1994 Sep; 14(9):6326-36. PubMed ID: 8065364
    [TBL] [Abstract][Full Text] [Related]  

  • 16. The product of the yeast UPF1 gene is required for rapid turnover of mRNAs containing a premature translational termination codon.
    Leeds P; Peltz SW; Jacobson A; Culbertson MR
    Genes Dev; 1991 Dec; 5(12A):2303-14. PubMed ID: 1748286
    [TBL] [Abstract][Full Text] [Related]  

  • 17. Nucleotide sequence of the triosephosphate isomerase gene from Aspergillus nidulans: implications for a differential loss of introns.
    McKnight GL; O'Hara PJ; Parker ML
    Cell; 1986 Jul; 46(1):143-7. PubMed ID: 3521890
    [TBL] [Abstract][Full Text] [Related]  

  • 18. The Lactococcus lactis triosephosphate isomerase gene, tpi, is monocistronic.
    Cancilla MR; Davidson BE; Hillier AJ; Nguyen NY; Thompson J
    Microbiology (Reading); 1995 Jan; 141 ( Pt 1)():229-38. PubMed ID: 7534588
    [TBL] [Abstract][Full Text] [Related]  

  • 19. Functional mapping of the translation-dependent instability element of yeast MATalpha1 mRNA.
    Hennigan AN; Jacobson A
    Mol Cell Biol; 1996 Jul; 16(7):3833-43. PubMed ID: 8668201
    [TBL] [Abstract][Full Text] [Related]  

  • 20. An operon encoding three glycolytic enzymes in Lactobacillus delbrueckii subsp. bulgaricus: glyceraldehyde-3-phosphate dehydrogenase, phosphoglycerate kinase and triosephosphate isomerase.
    Branny P; de la Torre F; Garel JR
    Microbiology (Reading); 1998 Apr; 144 ( Pt 4)():905-914. PubMed ID: 9579064
    [TBL] [Abstract][Full Text] [Related]  

    [Next]    [New Search]
    of 14.