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2. Effects of substrate specificity on initiating the base excision repair of N-methylpurines by variant human 3-methyladenine DNA glycosylases. Connor EE; Wilson JJ; Wyatt MD Chem Res Toxicol; 2005 Jan; 18(1):87-94. PubMed ID: 15651853 [TBL] [Abstract][Full Text] [Related]
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4. Human base excision repair creates a bias toward -1 frameshift mutations. Lyons DM; O'Brien PJ J Biol Chem; 2010 Aug; 285(33):25203-12. PubMed ID: 20547483 [TBL] [Abstract][Full Text] [Related]
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8. Aag-initiated base excision repair promotes ischemia reperfusion injury in liver, brain, and kidney. Ebrahimkhani MR; Daneshmand A; Mazumder A; Allocca M; Calvo JA; Abolhassani N; Jhun I; Muthupalani S; Ayata C; Samson LD Proc Natl Acad Sci U S A; 2014 Nov; 111(45):E4878-86. PubMed ID: 25349415 [TBL] [Abstract][Full Text] [Related]
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11. Relative rates of insertion and deletion mutations in dinucleotide repeats of various lengths in mismatch repair proficient mouse and mismatch repair deficient human cells. Yamada NA; Smith GA; Castro A; Roques CN; Boyer JC; Farber RA Mutat Res; 2002 Feb; 499(2):213-25. PubMed ID: 11827714 [TBL] [Abstract][Full Text] [Related]
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20. Slow base excision by human alkyladenine DNA glycosylase limits the rate of formation of AP sites and AP endonuclease 1 does not stimulate base excision. Maher RL; Vallur AC; Feller JA; Bloom LB DNA Repair (Amst); 2007 Jan; 6(1):71-81. PubMed ID: 17018265 [TBL] [Abstract][Full Text] [Related] [Next] [New Search]