158 related articles for article (PubMed ID: 28386637)
41. A system-level investigation into the cellular toxic response mechanism mediated by AhR signal transduction pathway.
Gim J; Kim HS; Kim J; Choi M; Kim JR; Chung YJ; Cho KH
Bioinformatics; 2010 Sep; 26(17):2169-75. PubMed ID: 20610613
[TBL] [Abstract][Full Text] [Related]
42. Fetal exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin transactivates aryl hydrocarbon receptor-responsive element III in the tyrosine hydroxylase immunoreactive neurons of the mouse midbrain.
Tanida T; Tasaka K; Akahoshi E; Ishihara-Sugano M; Saito M; Kawata S; Danjo M; Tokumoto J; Mantani Y; Nagahara D; Tabuchi Y; Yokoyama T; Kitagawa H; Kawata M; Hoshi N
J Appl Toxicol; 2014 Feb; 34(2):117-26. PubMed ID: 23296914
[TBL] [Abstract][Full Text] [Related]
43. Aryl hydrocarbon receptor (AHR) and AHR nuclear translocator (ARNT) expression in Baikal seal (Pusa sibirica) and association with 2,3,7,8-TCDD toxic equivalents and CYP1 expression levels.
Kim EY; Iwata H; Suda T; Tanabe S; Amano M; Miyazaki N; Petrov EA
Comp Biochem Physiol C Toxicol Pharmacol; 2005 Jul; 141(3):281-91. PubMed ID: 16111922
[TBL] [Abstract][Full Text] [Related]
44. Putative link between transcriptional regulation of IgM expression by 2,3,7,8-tetrachlorodibenzo-p-dioxin and the aryl hydrocarbon receptor/dioxin-responsive enhancer signaling pathway.
Sulentic CE; Holsapple MP; Kaminski NE
J Pharmacol Exp Ther; 2000 Nov; 295(2):705-16. PubMed ID: 11046109
[TBL] [Abstract][Full Text] [Related]
45. Ah Receptor Activation by Dioxin Disrupts Activin, BMP, and WNT Signals During the Early Differentiation of Mouse Embryonic Stem Cells and Inhibits Cardiomyocyte Functions.
Wang Q; Kurita H; Carreira V; Ko CI; Fan Y; Zhang X; Biesiada J; Medvedovic M; Puga A
Toxicol Sci; 2016 Feb; 149(2):346-57. PubMed ID: 26572662
[TBL] [Abstract][Full Text] [Related]
46. Low-dose dioxins alter gene expression related to cholesterol biosynthesis, lipogenesis, and glucose metabolism through the aryl hydrocarbon receptor-mediated pathway in mouse liver.
Sato S; Shirakawa H; Tomita S; Ohsaki Y; Haketa K; Tooi O; Santo N; Tohkin M; Furukawa Y; Gonzalez FJ; Komai M
Toxicol Appl Pharmacol; 2008 May; 229(1):10-9. PubMed ID: 18295293
[TBL] [Abstract][Full Text] [Related]
47. Responsiveness of the adult male rat reproductive tract to 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure: Ah receptor and ARNT expression, CYP1A1 induction, and Ah receptor down-regulation.
Roman BL; Pollenz RS; Peterson RE
Toxicol Appl Pharmacol; 1998 Jun; 150(2):228-39. PubMed ID: 9653054
[TBL] [Abstract][Full Text] [Related]
48. Transforming growth factor beta1 is not involved in 2,3,7,8-tetrachlorodibenzo- p-dioxin-dependent release from contact-inhibition in WB-F344 cells.
Hoelper P; Faust D; Oesch F; Dietrich C
Arch Toxicol; 2004 Nov; 78(11):643-8. PubMed ID: 15292977
[TBL] [Abstract][Full Text] [Related]
49. Disruption of cell-cell contact maximally but transiently activates AhR-mediated transcription in 10T1/2 fibroblasts.
Cho YC; Zheng W; Jefcoate CR
Toxicol Appl Pharmacol; 2004 Sep; 199(3):220-38. PubMed ID: 15364539
[TBL] [Abstract][Full Text] [Related]
50. Suppression mechanisms of flavonoids on aryl hydrocarbon receptor-mediated signal transduction.
Mukai R; Shirai Y; Saito N; Fukuda I; Nishiumi S; Yoshida K; Ashida H
Arch Biochem Biophys; 2010 Sep; 501(1):134-41. PubMed ID: 20450880
[TBL] [Abstract][Full Text] [Related]
51. Altered thyroxin and retinoid metabolic response to 2,3,7,8-tetrachlorodibenzo-p-dioxin in aryl hydrocarbon receptor-null mice.
Nishimura N; Yonemoto J; Miyabara Y; Fujii-Kuriyama Y; Tohyama C
Arch Toxicol; 2005 May; 79(5):260-7. PubMed ID: 15902423
[TBL] [Abstract][Full Text] [Related]
52. Persistent, low-dose 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure: effect on aryl hydrocarbon receptor expression in a dioxin-resistance model.
Franc MA; Pohjanvirta R; Tuomisto J; Okey AB
Toxicol Appl Pharmacol; 2001 Aug; 175(1):43-53. PubMed ID: 11509025
[TBL] [Abstract][Full Text] [Related]
53. Aryl hydrocarbon receptor activation by 2,3,7,8-tetrachlorodibenzo-p-dioxin impairs human B lymphopoiesis.
Li J; Phadnis-Moghe AS; Crawford RB; Kaminski NE
Toxicology; 2017 Mar; 378():17-24. PubMed ID: 28049042
[TBL] [Abstract][Full Text] [Related]
54. Differences in acute toxicity syndromes of 2,3,7,8-tetrachlorodibenzo-p-dioxin and 1,2,3,4,7,8-hexachlorodibenzo-p-dioxin in rats.
Niittynen M; Simanainen U; Syrjälä P; Pohjanvirta R; Viluksela M; Tuomisto J; Tuomisto JT
Toxicology; 2007 Jun; 235(1-2):39-51. PubMed ID: 17448584
[TBL] [Abstract][Full Text] [Related]
55. Molecular and functional characterization of a novel aryl hydrocarbon receptor isoform, AHR1β, in the chicken (Gallus gallus).
Lee JS; Iwabuchi K; Nomaru K; Nagahama N; Kim EY; Iwata H
Toxicol Sci; 2013 Dec; 136(2):450-66. PubMed ID: 23997109
[TBL] [Abstract][Full Text] [Related]
56. Ah receptor: dioxin-mediated toxic responses as hints to deregulated physiologic functions.
Bock KW; Köhle C
Biochem Pharmacol; 2006 Aug; 72(4):393-404. PubMed ID: 16545780
[TBL] [Abstract][Full Text] [Related]
57. The aryl-hydrocarbon receptor, but not the aryl-hydrocarbon receptor nuclear translocator protein, is rapidly depleted in hepatic and nonhepatic culture cells exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin.
Pollenz RS
Mol Pharmacol; 1996 Mar; 49(3):391-8. PubMed ID: 8643077
[TBL] [Abstract][Full Text] [Related]
58. 2,3,7,8-Tetrachlorodibenzo-p-dioxin modulates functional differentiation of mouse bone marrow-derived dendritic cells Downregulation of RelB by 2,3,7,8-tetrachlorodibenzo-p-dioxin.
Lee JA; Hwang JA; Sung HN; Jeon CH; Gill BC; Youn HJ; Park JH
Toxicol Lett; 2007 Aug; 173(1):31-40. PubMed ID: 17681673
[TBL] [Abstract][Full Text] [Related]
59. Role of the aryl hydrocarbon receptor in the development of control and 2,3,7,8-tetrachlorodibenzo-p-dioxin-exposed male mice.
Lin TM; Ko K; Moore RW; Buchanan DL; Cooke PS; Peterson RE
J Toxicol Environ Health A; 2001 Oct; 64(4):327-42. PubMed ID: 11693491
[TBL] [Abstract][Full Text] [Related]
60. 2,3,7,8-Tetrachlorodibenzo-p-dioxin activation of the aryl hydrocarbon receptor/aryl hydrocarbon receptor nuclear translocator pathway causes developmental toxicity through a CYP1A-independent mechanism in zebrafish.
Carney SA; Peterson RE; Heideman W
Mol Pharmacol; 2004 Sep; 66(3):512-21. PubMed ID: 15322242
[TBL] [Abstract][Full Text] [Related]
[Previous] [Next] [New Search]
